33 research outputs found
Iron Behaving Badly: Inappropriate Iron Chelation as a Major Contributor to the Aetiology of Vascular and Other Progressive Inflammatory and Degenerative Diseases
- Author
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- The Long-Term Intervention with Pravastatin in Ischaemic Disease (LIPID) Study Group
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- ZZ Chong
- Publication venue
- Publication date
- 09/08/2008
- Field of study
The production of peroxide and superoxide is an inevitable consequence of
aerobic metabolism, and while these particular "reactive oxygen species" (ROSs)
can exhibit a number of biological effects, they are not of themselves
excessively reactive and thus they are not especially damaging at physiological
concentrations. However, their reactions with poorly liganded iron species can
lead to the catalytic production of the very reactive and dangerous hydroxyl
radical, which is exceptionally damaging, and a major cause of chronic
inflammation. We review the considerable and wide-ranging evidence for the
involvement of this combination of (su)peroxide and poorly liganded iron in a
large number of physiological and indeed pathological processes and
inflammatory disorders, especially those involving the progressive degradation
of cellular and organismal performance. These diseases share a great many
similarities and thus might be considered to have a common cause (i.e.
iron-catalysed free radical and especially hydroxyl radical generation). The
studies reviewed include those focused on a series of cardiovascular, metabolic
and neurological diseases, where iron can be found at the sites of plaques and
lesions, as well as studies showing the significance of iron to aging and
longevity. The effective chelation of iron by natural or synthetic ligands is
thus of major physiological (and potentially therapeutic) importance. As
systems properties, we need to recognise that physiological observables have
multiple molecular causes, and studying them in isolation leads to inconsistent
patterns of apparent causality when it is the simultaneous combination of
multiple factors that is responsible. This explains, for instance, the
decidedly mixed effects of antioxidants that have been observed, etc...Comment: 159 pages, including 9 Figs and 2184 reference
GABAergic regulation of cerebellar NG2 cell development is altered in perinatal white matter injury.
- Author
- A Messerschmidt
- AR Calver
- B Bi
- B Jablonska
- B Larroque
- BA Barres
- Brian McEllin
- C Limperopoulos
- C Nosarti
- CA Ghiani
- CA Schneider
- Christian A Hübner
- CR Pierson
- Dandan Sun
- DE Bergles
- DM Fagel
- DR Foran
- G López-Bendito
- H Ring
- HC Kinney
- HT Chao
- J Glykys
- J Scafidi
- J Scafidi
- J Song
- JD Clements
- Jeffrey L Dupree
- JJ LoTurco
- JJ Volpe
- JM Bekkers
- JM Bekkers
- JM Mangin
- JM Mangin
- Jorge Edwards
- Joseph Scafidi
- LK Pacey
- Lloyd Harvey
- LM De Biase
- M Allin
- M Andäng
- M Anjari
- M Flagella
- M Häusser
- M Komitova
- M Kukley
- M Raymond
- M Simat
- M Vélez-Fort
- Mark Farrant
- Marzieh Zonouzi
- MV Nguyen
- MW Antoine
- P Wulff
- Peijun Li
- PT Tsai
- R Káradóttir
- RL Buckner
- RN Fernando
- S Ge
- S Robinson
- S Wang
- SA Back
- SA Back
- SA Back
- SB Glickstein
- SC Lin
- SH Kang
- SM Maricich
- SP Fancy
- Stuart G Cull-Candy
- T Hothorn
- TA Pitier
- TJ Yuen
- TZ Baram
- VA Deshmukh
- Vittorio Gallo
- X Liu
- X Zhu
- XP Tong
- Y Chen
- Y Lee
- Y Tozuka
- Publication venue
- Publication date
- 01/01/2015
- Field of study
Diffuse white matter injury (DWMI), a leading cause of neurodevelopmental disabilities in preterm infants, is characterized by reduced oligodendrocyte formation. NG2-expressing oligodendrocyte precursor cells (NG2 cells) are exposed to various extrinsic regulatory signals, including the neurotransmitter GABA. We investigated GABAergic signaling to cerebellar white matter NG2 cells in a mouse model of DWMI (chronic neonatal hypoxia). We found that hypoxia caused a loss of GABAA receptor-mediated synaptic input to NG2 cells, extensive proliferation of these cells and delayed oligodendrocyte maturation, leading to dysmyelination. Treatment of control mice with a GABAA receptor antagonist or deletion of the chloride-accumulating transporter NKCC1 mimicked the effects of hypoxia. Conversely, blockade of GABA catabolism or GABA uptake reduced NG2 cell numbers and increased the formation of mature oligodendrocytes both in control and hypoxic mice. Our results indicate that GABAergic signaling regulates NG2 cell differentiation and proliferation in vivo, and suggest that its perturbation is a key factor in DWMI
Cell cycle regulation of proliferation versus differentiation in the central nervous system
- Author
- A Clarke
- A Lasorella
- A Lasorella
- A Tury
- A Tury
- AE Vernon
- AE Vernon
- AI Lukaszewicz
- Anna Philpott
- AR Desai
- B Artegiani
- B Durand
- B Joseph
- B Joseph
- C Bertoli
- C Hindley
- C Hindley
- C Lange
- C Lange
- CJ Sherr
- D Morgan
- D Yellajoshyula
- DV Hansen
- E Batsché
- E Hartfuss
- E Peco
- EA Caronna
- EJ Bellefroid
- EY Lee
- EY Lee
- F Calegari
- F Calegari
- F Cremisi
- F Doetsch
- FA Dhariwala
- FA Dick
- Fahad R. Ali
- G Mairet-Coello
- G Wilkinson
- H Shimojo
- H Ueno
- J Briscoe
- J Hatakeyama
- J-W Lim
- JC Dugas
- JH Lui
- KF Macleod
- KL Ferguson
- KL Ferguson
- KL Kroll
- L Bally-Cuif
- L Luo
- L Luo
- L Nguyen
- L Nguyen
- L Richard-Parpaillon
- Laura J. A. Hardwick
- M Götz
- M Lacomme
- M Pitulescu
- M Roccio
- M Sauer
- MA Dyer
- MA Dyer
- MH Farah
- MM Lipinski
- N Bertrand
- N Koyano-Nakagawa
- P Malatesta
- P Malatesta
- P Paszek
- PM Nurse
- R Dhavan
- R Roark
- RA Cosgrove
- Roberta Azzarelli
- S Carruthers
- S Lim
- S Ohnuma
- S Pauklin
- S Seo
- S Seo
- S Seo
- S Seo
- S-i Ohnuma
- S-i Ohnuma
- S-i Ohnuma
- SA Fietz
- SB Glickstein
- SC Noctor
- SK McConnell
- SL Karsten
- T Goto
- T Jacks
- T Kawauchi
- T Tarui
- TE Anthony
- TJ McGarry
- W Haubensak
- W Zhong
- X Li
- Y Dobashi
- Y Itoh
- Y Tsunekawa
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Consensus Paper: Towards a Systems-Level View of Cerebellar Function: the Interplay Between Cerebellum, Basal Ganglia, and Cortex
- Author
- A Clark
- A Conte
- A Gruart
- A Kishore
- A Kishore
- A Kishore
- A Parent
- A Ponzi
- A Rasmussen
- A Sadnicka
- A Spanne
- A Spanne
- AC Bostan
- AC Bostan
- AG Barto
- AJ Carrel
- AJ Gruber
- AL Benabid
- AM Borghi
- AM Graybiel
- AM Haith
- AM Lozano
- Andreea C. Bostan
- Angel Lago-Rodriguez
- Asha Kishore
- B Alstermark
- B Alstermark
- B Rosin
- BE Pfeiffer
- BH Ross
- C Hofstötter
- C Hubsch
- C Soderblom
- CH Chen
- CI De Zeeuw
- D Caligiore
- D Caligiore
- D Caligiore
- D Caligiore
- D Popa
- DA McCormick
- Daniele Caligiore
- DJ Foster
- DJ Herzfeld
- DM Wolpert
- DP Calderon
- E Azim
- E D’Angelo
- E Hoshi
- E Lesage
- EA Pelzer
- F Bengtsson
- F Bengtsson
- F Morgante
- F Papengut
- FA Middleton
- G Baldassarre
- G Hesslow
- G Hesslow
- G Jayaram
- G Jayaram
- G Koch
- G Lakoff
- G Percheron
- G Pezzulo
- G Pezzulo
- G Pezzulo
- G Pezzulo
- G Pezzulo
- G Rizzolatti
- GA Bortoff
- GE Alexander
- GI Allen
- Gianluca Baldassarre
- Giovanni Pezzulo
- H Jörntell
- Henrik Jörntell
- HH Yin
- I Gormezano
- I Herreros
- Ivan Herreros
- J Bonaiuto
- J Glascher
- J Groß
- J Houk
- J Izawa
- J Izawa
- J Troncoso
- J Volkmann
- J Wang
- JA Taylor
- James Houk
- JC Houk
- JC Houk
- JC Houk
- JC Houk
- JC Houk
- JC Houk
- JC Houk
- JD Schmahmann
- JD Schmahmann
- JE Hoover
- JE Schlerf
- JF Marsden
- JF Medina
- JF Medina
- JL Emken
- JM Galea
- JM Galea
- JM Galea
- JM Hurtado
- JNJ Reynolds
- Joseph M. Galea
- JR Wolpaw
- JT Green
- JW Mink
- K Doya
- K Doya
- K Kultas-Ilinsky
- K Seepanomwan
- Kenji Doya
- L Meteyard
- LB Smith
- LF Koziol
- LF Koziol
- LW Barsalou
- LW Barsalou
- M Glickstein
- M Hallett
- M Hamada
- M Iacoboni
- M Ito
- M Ito
- M Jahanshahi
- M Jeannerod
- M Kawato
- M Manto
- M Santello
- M Wexler
- MA Arbib
- MD Mauk
- Michiel Dirkx
- MJ Frank
- MJ Frank
- MM Merzenich
- N Ichinohe
- NB Albert
- ND Daw
- NE Berthier
- NH Bhanpuri
- NM White
- O Hikosaka
- O Hikosaka
- O Oscarsson
- P Cisek
- P Cisek
- P Dayan
- P Dean
- P Geborek
- P Geborek
- P Heman
- P Krack
- P Plaha
- P Pope
- P Redgrave
- P Thoma
- Paul F. M. J. Verschure
- Peter L. Strick
- PFMJ Verschure
- PFMJ Verschure
- PFMJ Verschure
- PL Strick
- PS Goldman-Rakic
- R Hardwick
- R Proville
- R Shadmehr
- R. Chris Miall
- RA Scheidt
- RC Helmich
- RC Helmich
- RC Miall
- RC Miall
- RF Thompson
- Riccardo Zucca
- Rick C. Helmich
- RM Kelly
- RM Kelly
- RN Holdefer
- RP Dum
- S Brandi
- S Ohmae
- S Sami
- SA Heiney
- SE Hua
- SH Scott
- SS Shrestha
- T Popa
- T Wu
- Traian Popa
- V Boulenger
- V Bracha
- VB Penhune
- VB Penhune
- Y Shinoda
- Y Ugawa
- Y-Z Huang
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Spatial arrangement of cerebro-pontine terminals
- Author
- Publication venue
- 'Wiley'
- Publication date
- Field of study
Sensorimotor processing for balance in spinocerebellar ataxia type 6
- Author
- Publication venue
- 'Wiley'
- Publication date
- Field of study
Incremental Approach to Derivative Polarography
- Author
- Publication venue
- 'American Chemical Society (ACS)'
- Publication date
- Field of study
A neuroscience perspective on transference
- Author
- Publication venue
- 'Elsevier BV'
- Publication date
- Field of study
Secretory studies in whole stomachs
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Secretory studies in whole stomachs
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study