96 research outputs found
The rare top quark decays in the topcolor-assisted technicolor model
We consider the rare top quark decays in the framework of topcolor-assisted
technicolor (TC2) model. We find that the contributions of top-pions and
top-Higgs predicted by the TC2 model can enhance the SM branching ratios by as
much as 6-9 orders of magnitude. i.e., in the most case, the orders of
magnitude of branching ratios are , , . With the reasonable values of the
parameters in TC2 model, such rare top quark decays may be testable in the
future experiments. So, rare top quark decays provide us a unique way to test
TC2 model.Comment: 14 pages, 4 figure
Glueball spectrum based on a rigorous three-dimensional relativistic equation for two-gluon bound states I: Derivation of the relativistic equation
A rigorous three-dimensional relativistic equation satisfied by two-gluon
bound states is derived from the QCD with massive gluons. With the gluon fields
and the quark fields being expanded in terms of the gluon multipole fields and
the spherical Dirac spinors respectively, the equation is well established in
the angular momentum representation and hence is much convenient for solving
the problem of two-gluon glueball spectra. In particular, the interaction
kernel in the equation is exactly derived and given a closed expression which
includes all the interactions taking place in the two-gluon glueballs. The
kernel contains only a few types of Green's functions and commutators.
Therefore, it is not only easily calculated by the perturbation method, but
also provides a suitable basis for nonperturbative investigations
Glueball spectrum based on a rigorous three-dimensional relativistic equation for two-gluon bound states II: calculation of the glueball spectrum
In the preceding paper, a rigorous three-dimensional relativistic equation
for two-gluon bound states was derived from the QCD with massive gluons and
represented in the angular momentum representation. In order to apply this
equation to calculate the glueball spectrum, in this paper, the equation is
recast in an equivalent three-dimensional relativistic equation satisfied by
the two-gluon positive energy state amplitude. The interaction Hamiltonian in
the equation is exactly derived and expressed as a perturbative series. The
first term in the series describes the one-gluon exchange interaction which
includes fully the retardation effect in it. This term plus the linear
confining potential are chosen to be the interaction Hamiltonian and employed
in the practical calculation. With the integrals containing three and four
spherical Bessel functions in the QCD vertices being analytically calculated,
the interaction Hamiltonian is given an explicit expression in the angular
momentum representation. Numerically solving the relativistic equation with
taking the contributions arising from the retardation effect and the
longitudinal mode of gluon fields into account, a set of masses for the
and glueball states are
obtained and are in fairly good agreement with the predictions given by the
lattice simulatio
The Pan-STARRS1 Medium-deep Survey: Star Formation Quenching in Group and Cluster Environments
We make use of a catalog of 1600 Pan-STARRS1 groups produced by the probability friends-of-friends algorithm to explore how the galaxy properties, i.e., the specific star formation rate (SSFR) and quiescent fraction, depend on stellar mass and group-centric radius. The work is the extension of Lin et al. In this work, powered by a stacking technique plus a background subtraction for contamination removal, a finer correction and more precise results are obtained than in our previous work. We find that while the quiescent fraction increases with decreasing group-centric radius, the median SSFRs of star-forming galaxies in groups at fixed stellar mass drop slightly from the field toward the group center. This suggests that the main quenching process in groups is likely a fast mechanism. On the other hand, a reduction in SSFRs by ~0.2 dex is seen inside clusters as opposed to the field galaxies. If the reduction is attributed to the slow quenching effect, the slow quenching process acts dominantly in clusters. In addition, we also examine the densityâcolor relation, where the density is defined by using a sixth-nearest-neighbor approach. Comparing the quiescent fractions contributed from the density and radial effect, we find that the density effect dominates the massive group or cluster galaxies, and the radial effect becomes more effective in less massive galaxies. The results support mergers and/or starvation as the main quenching mechanisms in the group environment, while harassment and/or starvation dominate in clusters
CP violation in in the model III 2HDM
We have calculated the Wilson coefficients (i=1,2) in the
renormalization scheme in the model III 2HDM. Using the obtained
Wilson coefficients, we have analyzed the CP violation in decays (q=d,s) in the model. The CP asymmetry, , depends on the
parameters of models and in can be as large as 40% and
35% for and respectively. It can reach 4% for decays.
Because in SM CP violation is smaller than or equal to O() which is
unobservably small, an observation of CP asymmetry in the decays would unambiguously signal the existence of new physics.Comment: revtex4, 16 pages, 7 figure
Clinical Characteristics and Neonatal Outcomes of Pregnant Patients With COVID-19: A Systematic Review
Background and Objective: Coronavirus disease 2019 (COVID-19) characterized by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has created serious concerns about its potential adverse effects. There are limited data on clinical, radiological, and neonatal outcomes of pregnant women with COVID-19 pneumonia. This study aimed to assess clinical manifestations and neonatal outcomes of pregnant women with COVID-19. Methods: We conducted a systematic article search of PubMed, EMBASE, Scopus, Google Scholar, and Web of Science for studies that discussed pregnant patients with confirmed COVID-19 between January 1, 2020, and April 20, 2020, with no restriction on language. Articles were independently evaluated by two expert authors. We included all retrospective studies that reported the clinical features and outcomes of pregnant patients with COVID-19. Results: Forty-seven articles were assessed for eligibility; 13 articles met the inclusion criteria for the systematic review. Data is reported for 235 pregnant women with COVID-19. The age range of patients was 25Ăąïżœïżœ40 years, and the gestational age ranged from 8 to 40 weeks plus 6 days. Clinical characteristics were fever 138/235 (58.72%), cough 111/235 (47.23%), and sore throat 21/235 (8.93%). One hundred fifty six out of 235 (66.38%) pregnant women had cesarean section, and 79 (33.62%) had a vaginal delivery. All the patients showed lung abnormalities in CT scan images, and none of the patients died. Neutrophil cell count, C-reactive protein (CRP) concentration, ALT, and AST were increased but lymphocyte count and albumin levels were decreased. Amniotic fluid, neonatal throat swab, and breastmilk samples were taken to test for SARS-CoV-2 but all found negativ results. Recent published evidence showed the possibility of vertical transmission up to 30%, and neonatal death up to 2.5%. Pre-eclampsia, fetal distress, PROM, pre-mature delivery were the major complications of pregnant women with COVID-19. Conclusions: Our study findings show that the clinical, laboratory and radiological characteristics of pregnant women with COVID-19 were similar to those of the general populations. The possibility of vertical transmission cannot be ignored but C-section should not be routinely recommended anymore according to latest evidences and, in any case, decisions should be taken after proper discussion with the family. Future studies are needed to confirm or refute these findings with a larger number of sample sizes and a long-term follow-up period. Ă© Copyright Ă© 2020 Islam, Poly, Walther, Yang, Wang, Hsieh, Atique, Salmani, Alsinglawi, Lin, Jian and Jack Li
Inclusive eta' Production from the Upsilon(1S)
Using the CLEO II detector at CESR, we measure the eta' - gluon - gluon
form-factor in Y(1S) decays. This form-factor especially at large eta' energies
may provide an explanation of the large rate for B -> Xs eta'. Our data do not
support a large anomalous coupling at higher q^2 and thus the large eta' rate
remains a mystery, possibly requiring a non-Standard Model explanation.Comment: 14 pages postscript, also available through
http://w4.lns.cornell.edu/public/CLNS, submitted to PR
Serum magnesium and calcium levels in relation to ischemic stroke : Mendelian randomization study
ObjectiveTo determine whether serum magnesium and calcium concentrations are causally associated with ischemic stroke or any of its subtypes using the mendelian randomization approach.MethodsAnalyses were conducted using summary statistics data for 13 single-nucleotide polymorphisms robustly associated with serum magnesium (n = 6) or serum calcium (n = 7) concentrations. The corresponding data for ischemic stroke were obtained from the MEGASTROKE consortium (34,217 cases and 404,630 noncases).ResultsIn standard mendelian randomization analysis, the odds ratios for each 0.1 mmol/L (about 1 SD) increase in genetically predicted serum magnesium concentrations were 0.78 (95% confidence interval [CI] 0.69-0.89; p = 1.3
7 10-4) for all ischemic stroke, 0.63 (95% CI 0.50-0.80; p = 1.6
7 10-4) for cardioembolic stroke, and 0.60 (95% CI 0.44-0.82; p = 0.001) for large artery stroke; there was no association with small vessel stroke (odds ratio 0.90, 95% CI 0.67-1.20; p = 0.46). Only the association with cardioembolic stroke was robust in sensitivity analyses. There was no association of genetically predicted serum calcium concentrations with all ischemic stroke (per 0.5 mg/dL [about 1 SD] increase in serum calcium: odds ratio 1.03, 95% CI 0.88-1.21) or with any subtype.ConclusionsThis study found that genetically higher serum magnesium concentrations are associated with a reduced risk of cardioembolic stroke but found no significant association of genetically higher serum calcium concentrations with any ischemic stroke subtype
New insights into the genetic etiology of Alzheimer's disease and related dementias.
Characterization of the genetic landscape of Alzheimer's disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/'proxy' AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE Δ4 allele
New insights into the genetic etiology of Alzheimer's disease and related dementias
Characterization of the genetic landscape of Alzheimer's disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/'proxy' AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE Δ4 allele
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