64 research outputs found

    A Causal-Comparative Study of Student Teachers\u27 Skill in Classroom and Behavior Management

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    Because many new teachers leave the field within the first five years of their first teaching job, a looming teacher shortage calls for further examination. An often-cited reason is new teachersā€™ perception of unpreparedness for handling classroom and behavior management tasks; thus, teacher preparation programs are under national pressure to design programs that produce well-prepared, effective teachers. The purpose of this study was to examine whether there was a difference in the classroom management performance of student teachers who are earning only a general education teacher license and those who are earning dual licensure in general and special education. The result was a longitudinal study using existing evaluative data. The archival data used was synthesized from cooperating teacher evaluations of all student teachers at Northwestern College during five semesters between the fall of 2009 and the spring of 2013. This quantitative study determined that there is a statistically significant relationship between preparation route and evaluation scores

    Prolegomena to a hybrid Classical/Rydberg simulator for hadronization (QuPYTH)

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    We present the classically simulated integration of a hadronization model, based on neutral Rydberg atoms, with the PYTHIA event generator. The real-time evolution of an interpreted charge-anticharge, in our Rydberg model, displays features similar to string-breaking - reminiscent of the Lund string fragmentation model used in PYTHIA for hadronization. We calculate the real-time evolution classically for a Rydberg simulator with up to 26 atoms. The behavior for increasing sites suggests that the model would be approaching semi-realistic hadron multiplicities using current QuEra hardware.Comment: 6 pages, 6 figures. Minor revisions to abstract, text, and figures. Added additional acknowledgements based on first-version feedbac

    HFPK 334: An unusual Supernova Remnant in the Small Magellanic Cloud

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    We present new Australia Telescope Compact Array (ATCA) radio-continuum and XMM-Newton/Chandra X-ray Observatory (CXO) observations of the unusual supernova remnant HFPK 334 in the Small Magellanic Cloud (SMC). The remnant follows a shell type morphology in the radio-continuum and has a size of āˆ¼\sim20~pc at the SMC distance. The X-ray morphology is similar, however, we detect a prominent point source close to the center of the SNR exhibiting a spectrum with a best fit powerlaw with a photon index of Ī“=2.7Ā±0.5\Gamma = 2.7 \pm 0.5. This central point source is most likely a background object and cannot be directly associated with the remnant. The high temperature, nonequilibrium conditions in the diffuse region suggest that this gas has been recently shocked and point toward a younger SNR with an age of ā‰²1800\lesssim 1800 years. With an average radio spectral index of Ī±=āˆ’0.59Ā±0.09\alpha=-0.59\pm0.09 we find that an equipartition magnetic field for the remnant is āˆ¼\sim90~Ī¼\muG, a value typical of younger SNRs in low-density environments. Also, we report detection of scattered radio polarisation across the remnant at 20~cm, with a peak fractional polarisation level of 25Ā±\pm5\%.Comment: 19 pages, 6-figures, submitted to A

    A HIF1Ī± Regulatory Loop Links Hypoxia and Mitochondrial Signals in Pheochromocytomas

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    Pheochromocytomas are neural crestā€“derived tumors that arise from inherited or sporadic mutations in at least six independent genes. The proteins encoded by these multiple genes regulate distinct functions. We show here a functional link between tumors with VHL mutations and those with disruption of the genes encoding for succinate dehydrogenase (SDH) subunits B (SDHB) and D (SDHD). A transcription profile of reduced oxidoreductase is detected in all three of these tumor types, together with an angiogenesis/hypoxia profile typical of VHL dysfunction. The oxidoreductase defect, not previously detected in VHL-null tumors, is explained by suppression of the SDHB protein, a component of mitochondrial complex II. The decrease in SDHB is also noted in tumors with SDHD mutations. Gain-of-function and loss-of-function analyses show that the link between hypoxia signals (via VHL) and mitochondrial signals (via SDH) is mediated by HIF1Ī±. These findings explain the shared features of pheochromocytomas with VHL and SDH mutations and suggest an additional mechanism for increased HIF1Ī± activity in tumors
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