17 research outputs found

    A Relationship Between Stellar Metallicity Gradients and Galaxy Age in Dwarf Galaxies

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    We explore the origin of stellar metallicity gradients in simulated and observed dwarf galaxies. We use FIRE-2 cosmological baryonic zoom-in simulations of 26 isolated galaxies as well as existing observational data for 10 Local Group dwarf galaxies. Our simulated galaxies have stellar masses between 105.510^{5.5} and 10^{8.6} \msun. Whilst gas-phase metallicty gradients are generally weak in our simulated galaxies, we find that stellar metallicity gradients are common, with central regions tending to be more metal-rich than the outer parts. The strength of the gradient is correlated with galaxy-wide median stellar age, such that galaxies with younger stellar populations have flatter gradients. Stellar metallicty gradients are set by two competing processes: (1) the steady "puffing" of old, metal-poor stars by feedback-driven potential fluctuations, and (2) the accretion of extended, metal-rich gas at late times, which fuels late-time metal-rich star formation. If recent star formation dominates, then extended, metal-rich star formation washes out pre-existing gradients from the "puffing" process. We use published results from ten Local Group dwarf galaxies to show that a similar relationship between age and stellar metallicity-gradient strength exists among real dwarfs. This suggests that observed stellar metallicity gradients may be driven largely by the baryon/feedback cycle rather than by external environmental effects.Comment: 14 pages, 13 figures, submitted to MNRA

    Pressure balance in the multiphase ISM of cosmologically simulated disc galaxies

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    Pressure balance plays a central role in models of the interstellar medium (ISM), but whether and how pressure balance is realized in a realistic multiphase ISM is not yet well understood. We address this question by using a set of FIRE-2 cosmological zoom-in simulations of Milky Way-mass disc galaxies, in which a multiphase ISM is self-consistently shaped by gravity, cooling, and stellar feedback. We analyse how gravity determines the vertical pressure profile as well as how the total ISM pressure is partitioned between different phases and components (thermal, dispersion/turbulence, and bulk flows). We show that, on average and consistent with previous more idealized simulations, the total ISM pressure balances the weight of the overlying gas. Deviations from vertical pressure balance increase with increasing galactocentric radius and with decreasing averaging scale. The different phases are in rough total pressure equilibrium with one another, but with large deviations from thermal pressure equilibrium owing to kinetic support in the cold and warm phases, which dominate the total pressure near the mid-plane. Bulk flows (e.g. inflows and fountains) are important at a few disc scale heights, while thermal pressure from hot gas dominates at larger heights. Overall, the total mid-plane pressure is well-predicted by the weight of the disc gas and we show that it also scales linearly with the star formation rate surface density (Ï‚SFR). These results support the notion that the Kennicutt-Schmidt relation arises because Ï‚SFR and the gas surface density (Ï‚g) are connected via the ISM mid-plane pressure

    Copy number rather than epigenetic alterations are the major dictator of imprinted methylation in tumors

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    It has been postulated that imprinting aberrations are common in tumors. To understand the role of imprinting in cancer, we have characterized copy-number and methylation in over 280 cancer cell lines and confirm our observations in primary tumors. Imprinted differentially methylated regions (DMRs) regulate parent-of-origin monoallelic expression of neighboring transcripts in cis. Unlike single-copy CpG islands that may be prone to hypermethylation, imprinted DMRs can either loose or gain methylation during tumorigenesis. Here, we show that methylation profiles at imprinted DMRs often not represent genuine epigenetic changes but simply the accumulation of underlying copy-number aberrations (CNAs), which is independent of the genome methylation state inferred from cancer susceptible loci. Our results reveal that CNAs also influence allelic expression as loci with copy-number neutral loss-of-heterozygosity or amplifications may be expressed from the appropriate parental chromosomes, which is indicative of maintained imprinting, although not observed as a single expression foci by RNA FISH

    Ferromanganese nodules of the Kara Sea

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