Does Time-Symmetry Imply Retrocausality? How the Quantum World Says "Maybe"

It has often been suggested that retrocausality offers a solution to some of the puzzles of quantum mechanics: e.g., that it allows a Lorentz-invariant explanation of Bell correlations, and other manifestations of quantum nonlocality, without action-at-a-distance. Some writers have argued that time-symmetry counts in favour of such a view, in the sense that retrocausality would be a natural consequence of a truly time-symmetric theory of the quantum world. Critics object that there is complete time-symmetry in classical physics, and yet no apparent retrocausality. Why should the quantum world be any different? This note throws some new light on these matters. I call attention to a respect in which quantum mechanics is different, under some assumptions about quantum ontology. Under these assumptions, the combination of time-symmetry without retrocausality is unavailable in quantum mechanics, for reasons intimately connected with the differences between classical and quantum physics (especially the role of discreteness in the latter). Not all interpretations of quantum mechanics share these assumptions, however, and in those that do not, time-symmetry does not entail retrocausality.Comment: 22 pages, 6 figures; significant revision

Participatory Security: Citizen Security, Participation, and the Inequities of Citizenship in Urban Peru

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/90078/1/j.1470-9856.2011.00656.x.pd

A múltipla Cecília

ABSTRACT: We are members of a group which researches Cecília Meireles poetry. Our aim in this paper is to offer a broad comprehension of the writer's world view, looking for relations between the poetic and the other dimensions. We are going to present three different portraits: 1) Cecilia as a Literature scholar; 2) Cecilia as a folklorist; 3) Cecilia as a journalist writing about education

Simultaneous Dual Frequency Observations of Giant Pulses from the Crab Pulsar

Simultaneous measurements of giant pulses from the Crab pulsar were taken at two widely spaced frequencies using the real-time detection of a giant pulse at 1.4 GHz at the Very Large Array to trigger the observation of that same pulse at 0.6 GHz at a 25-m telescope in Green Bank, WV. Interstellar dispersion of the signals provided the necessary time to communicate the trigger across the country via the Internet. About 70% of the pulses are seen at both 1.4 GHz and 0.6 GHz, implying an emission mechanism bandwidth of at least 0.8 GHz at 1 GHz for pulse structure on time scales of one to ten microseconds. The arrival times at both frequencies display a jitter of 100 microseconds within the window defined by the average main pulse profile and are tightly correlated. This tight correlation places limits on both the emission mechanism and on frequency dependent propagation within the magnetosphere. At 1.4 GHz the giant pulses are resolved into several, closely spaced components. Simultaneous observations at 1.4 GHz and 4.9 GHz show that the component splitting is frequency independent. We conclude that the multiplicity of components is intrinsic to the emission from the pulsar, and reject the hypothesis that this is the result of multiple imaging as the signal propagates through the perturbed thermal plasma in the surrounding nebula. At both 1.4 GHz and 0.6 GHz the pulses are characterized by a fast rise time and an exponential decay time which are correlated. The pulse broadening with its exponential decay form is most likely the result of multipath propagation in intervening ionized gas.Comment: LaTeX, 18 pages, 7 figures, accepted for publication in The Astrophysical Journa

Pandemic H1N1 Influenza Isolated from Free-Ranging Northern Elephant Seals in 2010 off the Central California Coast

Interspecies transmission of influenza A is an important factor in the evolution and ecology of influenza viruses. Marine mammals are in contact with a number of influenza reservoirs, including aquatic birds and humans, and this may facilitate transmission among avian and mammalian hosts. Virus isolation, whole genome sequencing, and hemagluttination inhibition assay confirmed that exposure to pandemic H1N1 influenza virus occurred among free-ranging Northern Elephant Seals (Mirounga angustirostris) in 2010. Nasal swabs were collected from 42 adult female seals in April 2010, just after the animals had returned to the central California coast from their short post-breeding migration in the northeast Pacific. Swabs from two seals tested positive by RT-PCR for the matrix gene, and virus was isolated from each by inoculation into embryonic chicken eggs. Whole genome sequencing revealed greater than 99% homology with A/California/04/2009 (H1N1) that emerged in humans from swine in 2009. Analysis of more than 300 serum samples showed that samples collected early in 2010 (n = 100) were negative and by April animals began to test positive for antibodies against the pH1N1 virus (HI titer of ≥ 1∶40), supporting the molecular findings. In vitro characterizations studies revealed that viral replication was indistinguishable from that of reference strains of pH1N1 in canine kidney cells, but replication was inefficient in human epithelial respiratory cells, indicating these isolates may be elephant seal adapted viruses. Thus findings confirmed that exposure to pandemic H1N1 that was circulating in people in 2009 occurred among free-ranging Northern Elephant Seals in 2010 off the central California coast. This is the first report of pH1N1 (A/Elephant seal/California/1/2010) in any marine mammal and provides evidence for cross species transmission of influenza viruses in free-ranging wildlife and movement of influenza viruses between humans and wildlife

Features of Moyal Trajectories

We study the Moyal evolution of the canonical position and momentum variables. We compare it with the classical evolution and show that, contrary to what is commonly found in the literature, the two dynamics do not coincide. We prove that this divergence is quite general by studying Hamiltonians of the form $p^2 /2m + V(q)$. Several alternative formulations of Moyal dynamics are then suggested. We introduce the concept of starfunction and use it to reformulate the Moyal equations in terms of a system of ordinary differential equations on the noncommutative Moyal plane. We then use this formulation to study the semiclassical expansion of Moyal trajectories, which is cast in terms of a (order by order in $\hbar$) recursive hierarchy of i) first order partial differential equations as well as ii) systems of first order ordinary differential equations. The latter formulation is derived independently for analytic Hamiltonians as well as for the more general case of smooth local integrable ones. We present various examples illustrating these results

Immune deconvolution and temporal mapping identifies stromal targets and developmental intervals for abrogating murine low-grade optic glioma formation

BACKGROUND: Brain tumor formation and progression are dictated by cooperative interactions between neoplastic and non-neoplastic cells. This stromal dependence is nicely illustrated by tumors arising in the Neurofibromatosis type 1 (NF1) cancer predisposition syndrome, where children develop low-grade optic pathway gliomas (OPGs). Using several authenticated METHODS: A combination of bulk and single-cell RNA mouse optic nerve sequencing, immunohistochemistry, T cell assays, and pharmacologic and antibody-mediated inhibition methods were used in these experiments. RESULTS: We show that T cells and microglia are the main non-neoplastic immune cell populations in both murine and human LGGs. Moreover, we demonstrate that CD8 CONCLUSIONS: Collectively, these findings provide proof-of-concept demonstrations that targeting stromal support during early gliomagenesis durably blocks murine LGG formation