177 research outputs found
Sorafenib, Rapamycin, and Venetoclax Attenuate Doxorubicin-Induced Senescence and Promote Apoptosis in HCT116 Cells
- Author
- Publication venue
- VCU Scholars Compass
- Publication date
- 31/12/2021
- Field of study
Emerging evidence has shown that the therapy-induced senescent growth arrest in cancer cells is of durable nature whereby a subset of cells can reinstate proliferative capacity. Promising new drugs named senolytics selectively target senescent cells and commit them into apoptosis. Accordingly, senolytics have been proposed as adjuvant cancer treatment to cull senescent tumor cells, and thus, screening for agents that exhibit senolytic properties is highly warranted. Our study aimed to investigate three agents, sorafenib, rapamycin, and venetoclax for their senolytic potential in doxorubicin-induced senescence in HCT116 cells. HCT116 cells were treated with one of the three agents, sorafenib (5 ”M), rapamycin (100 nM), or venetoclax (10 ”M), in the absence or presence of doxorubicin (1 ”M). Senescence was evaluated using microscopy-based and flow cytometry-based Senescence-associated-ÎČ-galactosidase staining (SA-ÎČ-gal), while apoptosis was assessed using annexin V-FITC/PI, and Muse caspase-3/-7 activity assays. We screened for potential genes through which the three drugs exerted senolytic-like action using the Human Cancer Pathway Finder PCR array. The three agents reduced doxorubicin-induced senescent cell subpopulations and significantly enhanced the apoptotic effect of doxorubicin compared with those treated only with doxorubicin. The senescence genes IGFBP5 and BMI1 and the apoptosis genes CASP7 and CASP9 emerged as candidate genes through which the three drugs exhibited senolytic-like properties. These results suggest that the attenuation of doxorubicin-induced senescence might have shifted HCT116 cells to apoptosis by exposure to the tested pharmacological agents. Our work argues for the use of senolytics to reduce senescence-mediated resistance in tumor cells and to enhance chemotherapy efficacy
Role of Interleukin-1 in Radiation-Induced Cardiomyopathy
- Author
- Publication venue
- VCU Scholars Compass
- Publication date
- 01/01/2015
- Field of study
Thoracic X-ray therapy (XRT), used in cancer treatment, is associated with increased risk of heart failure. XRT-mediated injury to the heart induces an inflammatory response leading to cardiomyopathy. The aim of this study was to determine the role of interleukin (IL)-1 in response to XRT injury to the heart and on the cardiomyopathy development in the mouse. Female mice with genetic deletion of the IL-1 receptor type I (IL-1R1 knockout mice [IL-1R1 KO]) and treatment with recombinant human IL-1 receptor antagonist anakinra, 10 mg/kg twice daily for 7 d, were used as independent approaches to determine the role of IL-1. Wild-type (wt) or IL-1R1 KO mice were treated with a single session of XRT (20 or 14 gray [Gy]). Echocardiography (before and after isoproterenol challenge) and left ventricular (LV) catheterization were performed to evaluate changes in LV dimensions and function. Massonâs trichrome was used to assess myocardial fibrosis and pericardial thickening. After 20 Gy, the contractile reserve was impaired in wt mice at d 3, and the LV ejection fraction (EF) was reduced after 4 months when compared with sham-XRT. IL-1R1 KO mice had preserved contractile reserve at 3 d and 4 months and LVEF at 4 months after XRT. Anakinra treatment for 1 d before and 7 d after XRT prevented the impairment in contractile reserve. A significant increase in LV end-diastolic pressure, associated with increased myocardial interstitial fibrosis and pericardial thickening, was observed in wt mice, as well as in IL-1R1 KOâor anakinra-treated mice. In conclusion, induction of IL-1 by XRT mediates the development of some, such as the contractile impairment, but not all aspects of the XRT-induced cardiomyopathy, such as myocardial fibrosis or pericardial thickening
The BET inhibitor/degrader ARV-825 prolongs the growth arrest response to Fulvestrant + Palbociclib and suppresses proliferative recovery in ER-positive breast cancer
- Author
- Publication venue
- 'Frontiers Media SA'
- Publication date
- 01/01/2023
- Field of study
Anti-estrogens or aromatase inhibitors in combination with cyclin-dependent kinase 4 and 6 (CDK4/6) inhibitors are the current standard of care for estrogen receptor-positive (ER+) Her-2 negative metastatic breast cancer. Although these combination therapies prolong progression-free survival compared to endocrine therapy alone, the growth-arrested state of residual tumor cells is clearly transient. Tumor cells that escape what might be considered a dormant or quiescent state and regain proliferative capacity often acquire resistance to further therapies. Our studies are based upon the observation that breast tumor cells arrested by Fulvestrant + Palbociclib enter into states of both autophagy and senescence from which a subpopulation ultimately escapes, potentially contributing to recurrent disease. Autophagy inhibition utilizing pharmacologic or genetic approaches only moderately enhanced the response to Fulvestrant + Palbociclib in ER+ MCF-7 breast tumor cells, slightly delaying proliferative recovery. In contrast, the BET inhibitor/degrader, ARV-825, prolonged the growth arrested state in both p53 wild type MCF-7 cells and p53 mutant T-47D cells and significantly delayed proliferative recovery. In addition, ARV-825 added after the Fulvestrant + Palbociclib combination promoted apoptosis and demonstrated efficacy in resistant RB deficient cell lines. These studies indicate that administration of BET inhibitors/degraders, which are currently being investigated in multiple clinical trials, may potentially improve standard of care therapy in metastatic ER+ breast cancer patients and may further prolong progression-free survival
Autophagy Inhibition for Chemosensitization and Radiosensitization in Cancer: Do the Preclinical Data Support This Therapeutic Strategy?
- Author
- Publication venue
- 'American Society for Pharmacology & Experimental Therapeutics (ASPET)'
- Publication date
- Field of study
The HSP70 modulator MAL3-101 inhibits Merkel cell carcinoma
- Author
- A Rodina
- AB Bakker
- Anne Baeurle
- C Garrido
- C Pincelli
- C Willmes
- Christian Adam
- CS Sullivan
- CS Sullivan
- D Green
- D Schrama
- DA Gewirtz
- David Schrama
- E Schmitt
- E Schmitt
- ET Sawai
- G Meier
- HJ Kwun
- HY Kim
- J Albores-Saavedra
- J Cheng
- J Nylandsted
- J Zalvide
- JA DeCaprio
- JC Young
- Jeffrey L. Brodsky
- JK Myung
- JP Duprat
- JĂŒrgen Christian Becker
- k Daily
- KS Campbell
- L Brocchieri
- L Ravagnan
- M Agelli
- M Fakiha
- M Rohde
- M Shuda
- M Shuda
- M Van Gele
- Michael Sherman
- MJ Braunstein
- MS Davids
- NJ Miller
- P Boukamp
- Peter Wipf
- PT Tai
- Q Sheng
- R Arora
- R Houben
- R Houben
- RA Howard
- RE Wang
- Roland Houben
- S Gillis
- S Gurbuxani
- SG Ronan
- SW Fewell
- T Liu
- U Moens
- U Wieland
- WE Samlowski
- Y Tutar
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2013
- Field of study
Merkel Cell Carcinoma (MCC) is a rare and highly aggressive neuroendocrine skin cancer for which no effective treatment is available. MCC represents a human cancer with the best experimental evidence for a causal role of a polyoma virus. Large T antigens (LTA) encoded by polyoma viruses are oncoproteins, which are thought to require support of cellular heat shock protein 70 (HSP70) to exert their transforming activity. Here we evaluated the capability of MAL3-101, a synthetic HSP70 inhibitor, to limit proliferation and survival of various MCC cell lines. Remarkably, MAL3-101 treatment resulted in considerable apoptosis in 5 out of 7 MCC cell lines. While this effect was not associated with the viral status of the MCC cells, quantitative mRNA expression analysis of the known HSP70 isoforms revealed a significant correlation between MAL3-101 sensitivity and HSC70 expression, the most prominent isoform in all cell lines. Moreover, MAL3-101 also exhibited in vivo antitumor activity in an MCC xenograft model suggesting that this substance or related compounds are potential therapeutics for the treatment of MCC in the future. © 2014 Adam et al
School choice, consumerism and the ethical strand in talk
- Author
- Andrew Wilkins
- Apple M.
- Ball S.J.
- Ball S.J.
- Barnett C.
- Billig M.
- Blinkhorn A.
- Clarke J.
- Clarke J.
- Clarke J.
- David M.
- David M.E.
- Deacon A.
- Dierkes J.
- Dunleavy P.
- Dwyer P.
- Edwards D.
- Etzioni A.
- Gewirtz S.
- Giddens A.
- Harvey D.
- Hauptmann E.
- Johansson H.
- Jones K.
- Le Grand J.
- Narodowski M.
- Oria A.
- Phillips K.D.
- Pollitt C.
- Reay D.
- Reay D.
- Reay D.
- Reay D.
- Reay D.
- Reay D.
- Rooney B.
- Rose N.
- Srivastava P.
- Walkerdine V.
- Wetherell M.
- Wetherell M.
- Wilkins A.
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2011
- Field of study
Research on school choice highlights the extent to which a communitarian impulse informs the way some parents engage with their role as chooser. This suggests that the responsibilities of parents as consumers are often negotiated in collective as well as individualizing terms. Drawing on data from a group of mothers of diverse social class and racial backgrounds, this paper builds on some of these perspectives through deploying elements of a critical discursive analytic approach. Its aim is to explore how some mothers engage with the meaning and practice of school choice. Focusing on the emotional labouring that often underpins mothersâ rationalizations of choice, this paper examines the discursive role of emotion in these contexts as a form of social action geared towards achieving certain ends. In turn I discuss the implications of this for thinking through choice as a framing, function and discourse inhabited and performed by mothers
Genetic effects on gene expression across human tissues
- Author
- Abell Nathan S.
- Abell Nathan S.
- Addington Anjene
- Addington Anjene M.
- Aguet François
- Aguet François
- Akey Joshua M.
- Ardlie Kristin G.
- Ardlie Kristin G.
- Balliu Brunilda
- Balliu Brunilda
- Barcus Mary E.
- Barcus Mary E.
- Barker Laura K.
- Barshir Ruth
- Basha Omer
- Bates Daniel
- Battle Alexis
- Billy Li Jin
- Bogu Gireesh K.
- Branton Philip A.
- Branton Philip A.
- Bridge Jason
- Bridge Jason
- Brigham Lori E.
- Brigham Lori E.
- Brown Andrew
- Brown Andrew A.
- Brown Christopher D.
- Bustamante Carlos D.
- Carithers Latarsha J.
- Castel Stephane E.
- Castel Stephane E.
- Chan Joanne
- Chen Lin S.
- Chen Lin S.
- Chiang Colby
- Claussnitzer Melina
- Conrad Donald F.
- Conrad Donald F.
- Cox Nancy J.
- Cox Nancy J.
- Craft Brian
- Cummings Beryl B.
- Cummings Beryl B.
- Damani Farhan N.
- Davis David A.
- Davis David A.
- Davis Joe R.
- Davis Joe R.
- Delaneau Olivier
- Delaneau Olivier
- Demanelis Kathryn
- Dermitzakis Emmanouil T.
- Dermitzakis Emmanouil T.
- Diegel Morgan
- Doherty Jennifer A.
- Engelhardt Barbara E.
- Eskin Eleazar
- Eskin Eleazar
- Feinberg Andrew P.
- Fernando Marian S.
- Ferreira Pedro G.
- Flicek Paul
- Foster Barbara A.
- Foster Barbara A.
- Frésard Laure
- Frésard Laure
- Gamazon Eric R.
- Gamazon Eric R.
- Garrido-MartĂn Diego
- Garrido-MartĂn Diego
- Gelfand Ellen T.
- Gelfand Ellen T.
- Getz Gad
- Getz Gad
- Gewirtz Ariel D.H.
- Gewirtz Ariel D.H.
- Gillard Bryan M.
- Gillard Bryan M.
- Gliner Genna
- Gliner Genna
- Gloudemans Michael J.
- Gloudemans Michael J.
- Goldman Mary
- Gould Sarah E.
- Guan Ping
- Guan Ping
- Guigo Roderic
- GuigĂł Roderic
- Hadley Kane
- Hadley Kane
- Haeussler Maximilian
- Hall Ira M.
- Halow Jessica
- Han Buhm
- Han Buhm
- Handsaker Robert E.
- Hansen Kasper D.
- Hariharan Pushpa
- Hasz Richard
- Hasz Richard
- Haugen Eric
- He Amy Z.
- He Yuan
- He Yuan
- Hickey Peter F.
- Hormozdiari Farhad
- Hormozdiari Farhad
- Hou Lei
- Howald Cedric
- Huang Katherine H.
- Huang Katherine H.
- Hunter Marcus
- Hunter Marcus
- Hunter Steven
- Jasmine Farzana
- Jewell Scott D.
- Jewell Scott D.
- Jian Ruiqi
- Jiang Lihua
- Jo Brian
- Jo Brian
- Johns Christopher
- Johns Christopher
- Johnson Audra
- Johnson Mark
- Johnson Mark
- Juettemann Thomas
- Kang Eun Yong
- Karasik Ellen
- Karasik Ellen
- Karczewski Konrad J.
- Kashin Seva
- Kaul Rajinder
- Kellis Manolis
- Kellis Manolis
- Kent W. James
- Kibriya Muhammad G.
- Kim Yungil
- Kim-Hellmuth Sarah
- Koester Susan
- Koester Susan E.
- Kopen Gene
- Kopen Gene
- Kumar Rachna
- Kumar Rachna
- Kyung Im Hae
- Lappalainen Tuuli
- Lappalainen Tuuli
- Lee Christopher M.
- Lee Kristen
- Leinweber William F.
- Leinweber William F.
- Lek Monkol
- Lek Monkol
- Li Gen
- Li Gen
- Li Qin
- Li Xiao
- Li Xiao
- Li Xiao
- Li Xin
- Li Xin
- Lin Jessica
- Lin Shin
- Linder Sandra
- Linke Caroline
- Little A. Roger
- Little A. Roger
- Liu Boxiang
- Liu Boxiang
- Liu Yaping
- Lockart Nicole C.
- Lockhart Nicole C.
- Lonsdale John T.
- Lonsdale John T.
- MacArthur Daniel G.
- MacArthur Daniel G.
- Mangul Serghei
- Martin Casey
- Mash Deborah C.
- Mash Deborah C.
- Matose Takunda
- Maurano Matthew T.
- McCarthy Mark I.
- McCarthy Mark I.
- McDonald Alisa
- McDonald Alisa
- McDowell Ian C.
- McDowell Ian C.
- McLean Jeffrey A.
- Mestichelli Bernadette
- Mestichelli Bernadette
- Miklos Mark
- Miklos Mark
- Mohammadi Pejman
- Mohammadi Pejman
- Molinie Benoit
- Monlong Jean
- Montgomery Stephen B.
- Montgomery Stephen B.
- Montroy Robert G.
- Montroy Robert G.
- Moore Helen M.
- Moore Helen M.
- Mosavel Maghboeba
- Moser Michael T.
- Moser Michael T.
- Muñoz-Aguirre Manuel
- Myer Kevin
- Myer Kevin
- Ndungu Anne W.
- Nedzel Jared L.
- Nedzel Jared L.
- Nelson Jemma
- Neri Fidencio J.
- Nguyen Duyen T.
- Nguyen Duyen Y.
- Nicolae Dan L.
- Nierras Concepcion R.
- Nobel Andrew B.
- Nobel Andrew B.
- Noble Michael S.
- Noble Michael S.
- Oliva Meritxell
- Oliva Meritxell
- Ongen Halit
- Ongen Halit
- Palowitch John J.
- Palowitch John J.
- Panousis Nikolaos
- Papasaikas Panagiotis
- Park Yongjin
- Park YoSon
- Park YoSon
- Parsana Princy
- Parsana Princy
- Paten Benedict
- Payne Anthony J.
- Peterson Christine B.
- Peterson Christine B.
- Pierce Brandon L.
- Qi Liqun
- Quan Jie
- Quon Gerald
- Rao Abhi
- Rao Abhi
- Reverter Ferran
- Rinaldi Nicola J.
- Ripke Stephan
- Rizzardi Lindsay F.
- Robinson Karna L.
- Roche Nancy V.
- Roe Brian
- Roe Bryan
- Rohrer Daniel C.
- Rohrer Daniel C.
- Rosenbloom Kate R.
- Ruffier Magali
- Sabatti Chiara
- Sabatti Chiara
- Saha Ashis
- Saha Ashis
- Salvatore Michael
- Salvatore Michael
- Sammeth Michael
- Sandstrom Richard
- Scott Alexandra J.
- SegrĂš Ayellet V.
- SegrĂš Ayellet V.
- Shabalin Andrey A.
- Shabalin Andrey A.
- Shad Saboor
- Shad Saboor
- Sheppard Dan
- Shimko Tyler C.
- Siminoff Laura A.
- Singh Shilpi
- Skol Andrew
- Smith Anna M.
- Smith Kevin S.
- Snyder Michael P.
- Sobin Leslie
- Sobin Leslie
- Sodaei Reza
- Stamatoyannopoulos John
- Stephens Matthew
- Stranger Barbara E.
- Stranger Barbara E.
- Stranger Barbara E.
- Strober Benjamin J.
- Strober Benjamin J.
- Struewing Jeffery P.
- Struewing Jeffery P.
- Sul Jae Hoon
- Sul Jae Hoon
- Sullivan Timothy J.
- Tabor David E.
- Tang Hua
- Taylor Kieron
- Teran Nicole A.
- Thomas Jeffrey A.
- Thomas Jeffrey A.
- Tomaszewski Maria M.
- Traino Heather M.
- Trevanion Stephen J.
- Trowbridge Casandra A.
- Tsang Emily K.
- Tsang Emily K.
- Tsang Emily K.
- Tukiainen Taru
- Tukiainen Taru
- Um Ki Sung
- Undale Anita H.
- Urbut Sarah
- Valentino Kimberly M.
- Valley Dana
- Valley Dana R.
- van de Bunt Martijn
- Van Wittenberghe Nicholas
- Vatanian Negin
- Vaught Jimmie B.
- Vivian John
- Volpi Simona
- Volpi Simona
- Walters Gary
- Walters Gary
- Wang Gao
- Wang Li
- Wang Meng
- Washington Michael
- Washington Michael
- Wen Xiaoquan
- Wen Xiaoquan
- Wheeler Joseph
- Wheeler Joseph
- Wright Fred A.
- Wright Fred A.
- Wu Fan
- Xi Hualin S.
- Yeger-Lotem Esti
- Yong Kang Eun
- Zappala Zachary
- Zappala Zachary
- Zaugg Judith B.
- Zerbino Daniel R.
- Zhang Hailei
- Zhang Rui
- Zhou Yi-Hui
- Zhou Yi-Hui
- Zhu Jingchun
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2017
- Field of study
Characterization of the molecular function of the human genome and its variation across individuals is essential for identifying the cellular mechanisms that underlie human genetic traits and diseases. The Genotype-Tissue Expression (GTEx) project aims to characterize variation in gene expression levels across individuals and diverse tissues of the human body, many of which are not easily accessible. Here we describe genetic effects on gene expression levels across 44 human tissues. We find that local genetic variation affects gene expression levels for the majority of genes, and we further identify inter-chromosomal genetic effects for 93 genes and 112 loci. On the basis of the identified genetic effects, we characterize patterns of tissue specificity, compare local and distal effects, and evaluate the functional properties of the genetic effects. We also demonstrate that multi-tissue, multi-individual data can be used to identify genes and pathways affected by human disease-associated variation, enabling a mechanistic interpretation of gene regulation and the genetic basis of diseas
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
- Author
- A. -G. Wu
- A. C. Ma
- A. K. Au
- Abdel-Aziz A. K.
- Abdelfatah S.
- Abdellatif M.
- Abdoli A.
- Abel S.
- Abeliovich H.
- Abildgaard M. H.
- Abudu Y. P.
- Acevedo-Arozena A.
- Adamopoulos I. E.
- Adeli K.
- Adolph T. E.
- Adornetto A.
- Aflaki E.
- Agam G.
- Agarwal A.
- Aggarwal B. B.
- Agnello M.
- Agostinis P.
- Agrewala J. N.
- Agrotis A.
- Aguilar P. V.
- Ahmad S. T.
- Ahmed Z. M.
- Ahumada-Castro U.
- Aits S.
- Aizawa S.
- Akkoc Y.
- Akoumianaki T.
- Akpinar H. A.
- Al-Abd A. M.
- Al-Akra L.
- Al-Gharaibeh A.
- Alaoui-Jamali M. A.
- Alberti S.
- Alcocer-Gomez E.
- Alessandri C.
- Ali M.
- Alim Al-Bari M. A.
- Aliwaini S.
- Alizadeh J.
- Almacellas E.
- Almasan A.
- Alonso A.
- Alonso G. D.
- Altan-Bonnet N.
- Altieri D. C.
- Alvarez E. M. C.
- Alves da Costa C.
- Alves S.
- Alzaharna M. M.
- Amadio M.
- Amantini C.
- Amaral C.
- Ambrosio S.
- Amer A. O.
- Ammanathan V.
- An Z.
- Andersen S. U.
- Andrabi S. A.
- Andrade-Silva M.
- Andres A. M.
- Angelini S.
- Ann D.
- Anozie U. C.
- Ansari M. Y.
- Antas P.
- Antebi A.
- Anton Z.
- Anwar T.
- Apetoh L.
- Apostolova N.
- Araki T.
- Araki Y.
- Arasaki K.
- Araujo W. L.
- Araya J.
- Arden C.
- Arevalo M. -A.
- Arguelles S.
- Arias E.
- Arikkath J.
- Arimoto H.
- Ariosa A. R.
- Armstrong-James D.
- Arnaune-Pelloquin L.
- Aroca A.
- Arroyo D. S.
- Arsov I.
- Artero R.
- Asaro D. M. L.
- Aschner M.
- Ashrafizadeh M.
- Ashur-Fabian O.
- Atanasov A. G.
- Auberger P.
- Auner H. W.
- Aurelian L.
- Autelli R.
- Avagliano L.
- Avalos Y.
- Aveic S.
- Aveleira C. A.
- Avin-Wittenberg T.
- Aydin Y.
- Ayton S.
- Ayyadevara S.
- Azzopardi M.
- B. C. B. Ko
- Baba M.
- Backer J. M.
- Backues S. K.
- Bae D. -H.
- Bae O. -N.
- Bae S. H.
- Baehrecke E. H.
- Baek A.
- Baek S. -H.
- Baek S. H.
- Bagetta G.
- Bagniewska-Zadworna A.
- Bai H.
- Bai J.
- Bai X.
- Bai Y.
- Bairagi N.
- Baksi S.
- Balazadeh S.
- Balbi T.
- Baldari C. T.
- Balduini W.
- Ballabio A.
- Ballester M.
- Balzan R.
- Bandopadhyay R.
- Banerjee S.
- Banerjee S.
- Banreti A.
- Bao Y.
- Baptista M. S.
- Baracca A.
- Barbati C.
- Bargiela A.
- Barila D.
- Barlow P. G.
- Barmada S. J.
- Barreiro E.
- Barreto G. E.
- Bartek J.
- Bartel B.
- Bartolome A.
- Barve G. R.
- Basagoudanavar S. H.
- Bassham D. C.
- Bast R. C.
- Basu A.
- Batoko H.
- Batten I.
- Baulieu E. E.
- Baumgarner B. L.
- Bayry J.
- Beale R.
- Beau I.
- Beaumatin F.
- Bechara L. R. G.
- Beck G. R.
- Beers M. F.
- Begun J.
- Behl C.
- Behrends C.
- Behrens G. M. N.
- Bei R.
- Bejarano E.
- Bel S.
- Belaid A.
- Belgareh-Touze N.
- Bellarosa C.
- Belleudi F.
- Bello Perez M.
- Bello-Morales R.
- Beltran J. S. D. O.
- Beltran S.
- Benbrook D. M.
- Bendorius M.
- Benitez B. A.
- Benito-Cuesta I.
- Bensalem J.
- Berchtold M. W.
- Berezowska S.
- Bergamaschi D.
- Bergami M.
- Bergmann A.
- Berliocchi L.
- Berlioz-Torrent C.
- Bernard A.
- Berthoux L.
- Besirli C. G.
- Besteiro S.
- Betin V. M.
- Beyaert R.
- Bezbradica J. S.
- Bhaskar K.
- Bhatia-Kissova I.
- Bhattacharya R.
- Bhattacharya S.
- Bhattacharyya S.
- Bhuiyan M. S.
- Bhutia S. K.
- Bi L.
- Bi X.
- Biden T. J.
- Bijian K.
- Billes V. A.
- Binart N.
- Bincoletto C.
- Birgisdottir A. B.
- Bjorkoy G.
- Blanco G.
- Blas-Garcia A.
- Blasiak J.
- Blomgran R.
- Blomgren K.
- Blum J. S.
- Boada-Romero E.
- Boban M.
- Boesze-Battaglia K.
- Boeuf P.
- Boland B.
- Bomont P.
- Bonaldo P.
- Bonam S. R.
- Bonfili L.
- Bonifacino J. S.
- Boone B. A.
- Bootman M. D.
- Bordi M.
- Borner C.
- Bornhauser B. C.
- Borthakur G.
- Bosch J.
- Bose S.
- Botana L. M.
- Botas J.
- Boulanger C. M.
- Boulton M. E.
- Bourdenx M.
- Bourgeois B.
- Bourke N. M.
- Bousquet G.
- Boya P.
- Bozhkov P. V.
- Bozi L. H. M.
- Bozkurt T. O.
- Brackney D. E.
- Brand-Saberi B.
- Brandts C. H.
- Braun R. J.
- Braus G. H.
- Bravo-Sagua R.
- Bravo-San Pedro J. M.
- Brest P.
- Bringer M. -A.
- Briones-Herrera A.
- Broaddus V. C.
- Brodersen P.
- Brodsky J. L.
- Brody S. L.
- Bronson P. G.
- Bronstein J. M.
- Brown C. N.
- Brown R. E.
- Brum P. C.
- Brumell J. H.
- Brunetti-Pierri N.
- Bruno D.
- Bryson-Richardson R. J.
- Bucci C.
- Buch S.
- Buchan J. R.
- Buchrieser C.
- Buckingham E. M.
- Budak H.
- Budini M.
- Bueno M.
- Buitrago-Molina L. E.
- Bultynck G.
- Burada F.
- Buraschi S.
- Burgoyne J. R.
- Buron M. I.
- Bustos V.
- Buttner S.
- Butturini E.
- Byrd A.
- C. C. W. Ng
- C. H. Lo
- Cabas I.
- Cabrera-Benitez S.
- Cadwell K.
- Cai J.
- Cai L.
- Cai Q.
- Cairo M.
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- Publication venue
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- Publication date
- 01/01/2021
- Field of study
Autophagy in malignant transformation and cancer progression
- Author
- Alec Kimmelman
- Andrew M Thorburn
- Anne Simonsen
- Beth Levine
- David A Gewirtz
- David C Rubinsztein
- Eric H Baehrecke
- Faderl S
- Federico Pietrocola
- Francesco Cecconi
- Freitas AC
- GomezâManzano C
- Guido Kroemer
- Guillermo Velasco
- HansâUwe Simon
- Jayanta Debnath
- Josef Penninger
- JosĂ© Manuel BravoâSan Pedro
- Kevin M Ryan
- Lorenzo Galluzzi
- Lu Z
- Maria Chiara Maiuri
- Martoriati A
- Mauro Piacentini
- Miracco C
- Patrice Codogno
- Ravi K Amaravadi
- Rousselot P
- Seamus J Martin
- Seok S
- Sharad Kumar
- Shuin T
- Vassiliki Karantza
- Publication venue
- 'EMBO'
- Publication date
- Field of study
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.
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- Yoo YH
- Yoshida K
- Yoshii SR
- Yoshimori T
- Yousefi B
- Yu B
- Yu H
- Yu J
- Yu J
- Yu L
- Yu M-L
- Yu S-W
- Yu VC
- Yu WH
- Yu Z
- Yu Z
- Yuan J
- Yuan L-Q
- Yuan S
- Yuan S-SF
- Yuan Y
- Yuan Z
- Yue J
- Yue Z
- Yun J
- Yung RL
- Zacks DN
- Zaffagnini G
- Zambelli VO
- Zanella I
- Zang QS
- Zanivan S
- Zappavigna S
- Zaragoza P
- Zarbalis KS
- Zarebkohan A
- Zarrouk A
- Zeitlin SO
- Zeng J
- Zeng J-D
- Zhan L
- Zhang B
- Zhang DD
- Zhang H
- Zhang H
- Zhang H
- Zhang H
- Zhang H
- Zhang H
- Zhang H
- Zhang H-L
- Zhang J
- Zhang J
- Zhang J-P
- Zhang KYB
- Zhang L
- Zhang L
- Zhang L
- Zhang L
- Zhang LW
- Zhang M
- Zhang P
- Zhang S
- Zhang W
- Zhang X
- Zhang X
- Zhang X
- Zhang X
- Zhang X
- Zhang X-W
- Zhang XD
- Zhang Y
- Zhang Y
- Zhang Y
- Zhang Y
- Zhang Y
- Zhang Y-D
- Zhang Y-Y
- Zhang Z
- Zhang Z
- Zhang Z
- Zhang Z
- Zhang Z
- Zhang Z
- Zhao H
- Zhao L
- Zhao S
- Zhao T
- Zhao X-F
- Zhao Y
- Zhao Y
- Zhao Y
- Zhao Y
- Zheng G
- Zheng K
- Zheng L
- Zheng S
- Zheng X-L
- Zheng Y
- Zheng Z-G
- Zhivotovsky B
- Zhong Q
- Zhou A
- Zhou B
- Zhou C
- Zhou G
- Zhou H
- Zhou H
- Zhou H
- Zhou J
- Zhou J
- Zhou J
- Zhou J
- Zhou K
- Zhou R
- Zhou X-J
- Zhou Y
- Zhou Y
- Zhou Y
- Zhou Z
- Zhou Z-Y
- Zhu B
- Zhu C
- Zhu G-Q
- Zhu H
- Zhu H
- Zhu H
- Zhu W-G
- Zhu Y
- Zhu Y
- Zhuang H
- Zhuang X
- Zientara-Rytter K
- Zimmermann CM
- Ziviani E
- Zoladek T
- Zong W-X
- Zorov DB
- Zorzano A
- Zou W
- Zou Z
- Zou Z
- Zuryn S
- Zwerschke W
- Ălvarez ĂMC
- Ăvalos Y
- Ănal G
- ĂstĂŒn S
- ÄoliÄ M
- ÄokiÄ J
- Ćœerovnik E
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
In 2008, we published the first set of guidelines for standardizing research in autophagy. Since then, this topic has received increasing attention, and many scientists have entered the field. Our knowledge base and relevant new technologies have also been expanding. Thus, it is important to formulate on a regular basis updated guidelines for monitoring autophagy in different organisms. Despite numerous reviews, there continues to be confusion regarding acceptable methods to evaluate autophagy, especially in multicellular eukaryotes. Here, we present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes. These guidelines are not meant to be a dogmatic set of rules, because the appropriateness of any assay largely depends on the question being asked and the system being used. Moreover, no individual assay is perfect for every situation, calling for the use of multiple techniques to properly monitor autophagy in each experimental setting. Finally, several core components of the autophagy machinery have been implicated in distinct autophagic processes (canonical and noncanonical autophagy), implying that genetic approaches to block autophagy should rely on targeting two or more autophagy-related genes that ideally participate in distinct steps of the pathway. Along similar lines, because multiple proteins involved in autophagy also regulate other cellular pathways including apoptosis, not all of them can be used as a specific marker for bona fide autophagic responses. Here, we critically discuss current methods of assessing autophagy and the information they can, or cannot, provide. Our ultimate goal is to encourage intellectual and technical innovation in the field
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