Unique Observations of a Geomagnetic SI^+ -- SI^- Pair: Solar Sources and Associated Solar Wind Fluctuations

The paper describes the occurrence of a pair of oppositely directed sudden impulses (SI), in the geomagnetic field ($\Delta$X), at ground stations, called SI${^{+}}$ -- SI${^{-}}$ pairs, that occurred between 1835 UT and 2300 UT on 23 April 1998. The SI${^{+}}$ -- SI${^{-}}$ pair, was closely correlated with corresponding variations in the solar wind density, while solar wind velocity and the southward component of the interplanetary magnetic field (Bz) did not show any correspondence. Further, this event had no source on the visible solar disk. However, a rear-side partial halo coronal mass ejection (CME) and an M1.4 class solar flare behind the west limb, took place on 20 April 1998, the date corresponding to the traceback location of the solar wind flows. This event presents empirical evidence, which to our knowledge, is the best convincing evidence for the association of specific solar events to the observations of an SI${^{+}}$ -- SI${^{-}}$ pair. In addition, it shows that it is possible for a rear side solar flare to propagate a shock towards the earth.Comment: The paper has just been accepted in the Journal of Geophysical Research (Space Physics) on 20 September 2010. It is 17 pages with 4 figure

Gaia CCDs: charge transfer inefficiency measurements between five years of flight

The European Space Agency’s Gaia spacecraft was launched in December 2013 and has been in orbit at the Earth-Sun Lagrange point 2 (L2) for over 6 years. The spacecraft measures the positions, distances, space motions and many other physical characteristics of around one billion stars in the Milky Way and beyond. It has a focal plane of 106 Charge-Coupled Devices (CCDs) which have all been performing well but have been measuring a small but quantifiable degradation in performance in time due to Non-Ionizing Energy Loss (NIEL) damage from interstellar radiation. This NIEL damage produces trap defects which can capture charge from signals and reduces the quality of the data. Gaia’s original mission lifetime was planned to be around 5 years and the pre-flight testing and radiation damage analysis was tailored around those timescales as well as with the projected solar activity before launch. Closer to the time of launch and during Gaia’s years of orbit, it has been noted that the solar activity was lower than what was initially predicted. From the previous analysis of in-flight data in 2016, it was calculated that Gaia was experiencing an order of magnitude less radiation damage than was predicted. This paper describes the analysis of charge calibration data and corresponding Charge Transfer Inefficiency (CTI) measurements from the in-flight CCDs, both near the beginning of the mission and after more than 5 years in orbit to quantify the radiation damage impact. These sets of results can be compared with those from the pre-flight tests which can be used to evaluate and understand the differences between the on-ground and in-flight results

Relationship between solar energetic oxygen flux and MHD shock Mach number

This study correlates the time-intensity profile of a magnetohydrodynamic (MHD) shock with the corresponding solar energetic oxygen for a coronal mass ejection (CME) event that occurred on October 28, 2003. The intensity of MHD shock, in terms of Mach number, is simulated using a 1.5D MHD code, whereas the solar energetic oxygen flux is observed by the Solar Isotope Spectrometer (SIS) on board the Advanced Composition Explorer (ACE) spacecraft. A good correlation (Pearson correlation coefficient: r = 0.70 – 0.84) is found between the forward fast-mode shock Mach number and the hourly-averaged, logarithmic oxygen differential energy flux for 7 energy channels (7.3 – 63.8 MeV). We suspect that the intensity-time profile of high energy SEP events is manifested by the strength (Mach number) of CME-driven propagation shocks. While further studies with more events are required to be more conclusive, this study result provides a direction for future studies or predictions of SEP fluxes

Spectral Analysis of the Chandra Comet Survey

We present results of the analysis of cometary X-ray spectra with an extended version of our charge exchange emission model (Bodewits et al. 2006). We have applied this model to the sample of 8 comets thus far observed with the Chandra X-ray observatory and ACIS spectrometer in the 300-1000 eV range. The surveyed comets are C/1999 S4 (LINEAR), C/1999 T1 (McNaught-Hartley), C/2000 WM1 (LINEAR), 153P/2002 (Ikeya-Zhang), 2P/2003 (Encke), C/2001 Q4 (NEAT), 9P/2005 (Tempel 1) and 73P/2006-B (Schwassmann-Wachmann 3) and the observations include a broad variety of comets, solar wind environments and observational conditions. The interaction model is based on state selective, velocity dependent charge exchange cross sections and is used to explore how cometary X-ray emission depend on cometary, observational and solar wind characteristics. It is further demonstrated that cometary X-ray spectra mainly reflect the state of the local solar wind. The current sample of Chandra observations was fit using the constrains of the charge exchange model, and relative solar wind abundances were derived from the X-ray spectra. Our analysis showed that spectral differences can be ascribed to different solar wind states, as such identifying comets interacting with (I) fast, cold wind, (II), slow, warm wind and (III) disturbed, fast, hot winds associated with interplanetary coronal mass ejections. We furthermore predict the existence of a fourth spectral class, associated with the cool, fast high latitude wind.Comment: 16 pages, 16 figures, and 7 Tables; accepted A&A (Due to space limits, this version has lower resolution jpeg images.

Solar Flares and Coronal Mass Ejections: A Statistically Determined Flare Flux-CME Mass Correlation

In an effort to examine the relationship between flare flux and corresponding CME mass, we temporally and spatially correlate all X-ray flares and CMEs in the LASCO and GOES archives from 1996 to 2006. We cross-reference 6,733 CMEs having well-measured masses against 12,050 X-ray flares having position information as determined from their optical counterparts. For a given flare, we search in time for CMEs which occur 10-80 minutes afterward, and we further require the flare and CME to occur within +/-45 degrees in position angle on the solar disk. There are 826 CME/flare pairs which fit these criteria. Comparing the flare fluxes with CME masses of these paired events, we find CME mass increases with flare flux, following an approximately log-linear, broken relationship: in the limit of lower flare fluxes, log(CME mass)~0.68*log(flare flux), and in the limit of higher flare fluxes, log(CME mass)~0.33*log(flare flux). We show that this broken power-law, and in particular the flatter slope at higher flare fluxes, may be due to an observational bias against CMEs associated with the most energetic flares: halo CMEs. Correcting for this bias yields a single power-law relationship of the form log(CME mass)~0.70*log(flare flux). This function describes the relationship between CME mass and flare flux over at least 3 dex in flare flux, from ~10^-7 to 10^-4 W m^-2.Comment: 28 pages, 16 figures, accepted to Solar Physic

Arrival times of Flare/Halo CME associated shocks at the Earth: comparison of the predictions of three numerical models with these observations

International audienceThe arrival times at L1 of eleven travelling shocks associated both with X-ray flaring and with halo CMEs recorded aboard SOHO/LASCO have been considered. Close to the Sun the velocities of these events were estimated using either Type II radio records or CME speeds. Close to the Earth the shocks were detected in the data of various solar wind plasma, interplanetary magnetic field (IMF) and energetic particle experiments aboard SOHO, ACE, WIND, INTERBALL-1 and IMP-8. The real-time shock arrival predictions of three numerical models, namely the Shock Time of Arrival Model (STOA), the Interplanetary Shock Propagation Model (ISPM) and the Hakamada-Akasofu-Fry Solar Wind Model (HAFv.2) were tested against these observations. This is the first time that energetic protons (tens of keV to a few MeV) have been used to complement plasma and IMF data in validating shock propagation models. The models were all generally successful in predicting shock arrivals. STOA provided the smallest values of the "predicted minus measured" arrival times and displayed a typical predictive precision better than about 8 h. The ratio of the calculated standard deviation of the transit times to Earth to the standard deviation of the measurements was estimated for each model (treating interacting events as composite shocks) and these ratios turned out to be 0.60, 1.15 and 1.02 for STOA, ISPM and HAFv.2, respectively. If an event in the sample for which the shock velocity was not well known is omitted from consideration, these ratios become 0.36, 0.76 and 0.81, respectively. Larger statistical samples should now be tested. The ratio of the in situ shock velocity and the "Sun to L1" transit velocity (Vsh /Vtr) was in the range of 0.7?0.9 for individual, non-interacting, shock events. HAFv.2 uniquely provided information on those changes in the COBpoint (the moving Connection point on the shock along the IMF to the OBserver) which directly influenced energetic particle rise times. This model also illustrated the non-uniform upstream conditions through which the various shocks propagated; furthermore it simulated shock deformation on a scale of fractions of an AU. On the spatial scale (300 RE ), where near-Earth spacecraft are located, the passing shocks, in conformity with the models, were found to be locally planar. The shocks also showed tilting relative to the Sun-Earth line, probably reflecting the inherent directionality associated with their solar origin. Key words. Interplanetary physics (energetic particles; interplanetary shocks; solar wind plasma

A simple view of linguistic complexity

Although a growing number of second language acquisition (SLA) studies take linguistic complexity as a dependent variable, the term is still poorly defined and often used with different meanings, thus posing serious problems for research synthesis and knowledge accumulation. This article proposes a simple, coherent view of the construct, which is defined in a purely structural way, i.e. the complexity directly arising from the number of linguistic elements and their interrelationships. Issues of cognitive cost (difficulty) or developmental dynamics (acquisition) are explicitly excluded from this theoretical definition and its operationalization. The article discusses how the complexity of an interlanguage system can be assessed based on the limited samples SLA researchers usually work with. For the areas of morphology, syntax and the lexicon, some measures are proposed which are coherent with the purely structural view advocated, and issues related to their operationalization are critically scrutinized

Use of chromatin immunoprecipitation (ChIP) to detect transcription factor binding to highly homologous promoters in chromatin isolated from unstimulated and activated primary human B cells

The Chromatin Immunoprecipiation (ChIP) provides a powerful technique for identifying the in vivo association of transcription factors with regulatory elements. However, obtaining meaningful information for promoter interactions is extremely challenging when the promoter is a member of a class of highly homologous elements. Use of PCR primers with small numbers of mutations can limit cross-hybridization with non-targeted sequences and distinguish a pattern of binding for factors with the regulatory element of interest. In this report, we demonstrate the selective in vivo association of NF-κB, p300 and CREB with the human Iγ1 promoter located in the intronic region upstream of the Cγ1 exons in the immunoglobulin heavy chain locus. These methods have the ability to extend ChIP analysis to promoters with a high degree of homology

Src Dependent Pancreatic Acinar Injury Can Be Initiated Independent of an Increase in Cytosolic Calcium

Several deleterious intra-acinar phenomena are simultaneously triggered on initiating acute pancreatitis. These culminate in acinar injury or inflammatory mediator generation in vitro and parenchymal damage in vivo. Supraphysiologic caerulein is one such initiator which simultaneously activates numerous signaling pathways including non-receptor tyrosine kinases such as of the Src family. It also causes a sustained increase in cytosolic calcium- a player thought to be crucial in regulating deleterious phenomena. We have shown Src to be involved in caerulein induced actin remodeling, and caerulein induced changes in the Golgi and post-Golgi trafficking to be involved in trypsinogen activation, which initiates acinar cell injury. However, it remains unclear whether an increase in cytosolic calcium is necessary to initiate acinar injury or if injury can be initiated at basal cytosolic calcium levels by an alternate pathway. To study the interplay between tyrosine kinase signaling and calcium, we treated mouse pancreatic acinar cells with the tyrosine phosphatase inhibitor pervanadate. We studied the effect of the clinically used Src inhibitor Dasatinib (BMS-354825) on pervanadate or caerulein induced changes in Src activation, trypsinogen activation, cell injury, upstream cytosolic calcium, actin and Golgi morphology. Pervanadate, like supraphysiologic caerulein, induced Src activation, redistribution of the F-actin from its normal location in the sub-apical area to the basolateral areas, and caused antegrade fragmentation of the Golgi. These changes, like those induced by supraphysiologic caerulein, were associated with trypsinogen activation and acinar injury, all of which were prevented by Dasatinib. Interestingly, however, pervanadate did not cause an increase in cytosolic calcium, and the caerulein induced increase in cytosolic calcium was not affected by Dasatinib. These findings suggest that intra-acinar deleterious phenomena may be initiated independent of an increase in cytosolic calcium. Other players resulting in acinar injury along with the Src family of tyrosine kinases remain to be explored. © 2013 Mishra et al