15 research outputs found

    Kiadói és nyomdai tevékenység a szerbek körében a középkor végén és az újkor hajnalán

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    We study the properties of 30 spectroscopically identified pairs of galaxies observed during the peak epoch of star formation in the universe. These systems are drawn from the MOSFIRE Deep Evolution Field (MOSDEF) Survey at 1.4 ≤ z ≤ 3.8, and are interpreted as early-stage galaxy mergers. Galaxy pairs in our sample are identified as two objects whose spectra were collected on the same Keck/MOSFIRE spectroscopic slit. Accordingly, all pairs in the sample have projected separations R proj ≤ 60 kpc. The velocity separation for pairs was required to be Δv ≤ 500 km s -1 , which is a standard threshold for defining interacting galaxy pairs at low redshift. Stellar mass ratios in our sample range from 1.1 to 550, with 12 ratios closer than or equal to 3:1, the common definition of a "major merger." Studies of merging pairs in the local universe indicate an enhancement in star formation activity and deficit in gas-phase oxygen abundance relative to isolated galaxies of the same mass. We compare the MOSDEF pairs sample to a control sample of isolated galaxies at the same redshift, finding no measurable SFR enhancement or metallicity deficit at fixed stellar mass for the pairs sample. The lack of significant difference between the average properties of pairs and control samples appears in contrast to results from low-redshift studies, although the small sample size and lower signal-to-noise of the high-redshift data limit definitive conclusions on redshift evolution. These results are consistent with some theoretical works, suggesting a reduced differential effect of precoalescence mergers on galaxy properties at high redshift - specifically that precoalescence mergers do not drive strong starbursts

    WT1 and GATA4 in the regulation of sex-specific gonadal gene expression.

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    <p>During testis development, WT1 is required for activating various testis promoting genes, i.e. <i>Amh</i>, <i>Amhr2</i>, <i>Nr5a1</i>(<i>Sf1)</i>, <i>Star</i>, <i>Sox9</i>, <i>Gli1</i>, <i>Gli2</i>, <i>Smo</i>, <i>Dhh and Ptch1</i>. GATA4 represses ovarian promoting transcripts such as <i>Ctnnb1</i>, <i>Bmp2</i>, and <i>Fst</i>, in the testis, and thereby may be essential for the maintenance of the testis phenotype. GATA4 is also involved in repressing <i>Dhh</i> and <i>Ptch1</i> transcript levels in embryonic testis, which may contribute to the fine-tuning of their threshold levels. Given such a pivotal role of GATA4, it is likely that sufficient GATA4 levels are ensured by a back-up mechanism provided by the GATA4 E1b isoform in the testis. During ovary development, i.e. in the absence of SRY, WNT4 and RSPO-1 levels are stabilized and promote canonical Wnt signaling. In the ovary, WT1 is required for <i>Dax1</i> expression. GATA4 acts as a repressor of <i>Ctnnb1</i> and serves as an activator for the transcripts <i>Gli2</i>, <i>Gli3</i>, and <i>Smo</i>, the latter of which may be regulated synergistically by both WT1 and GATA4. WT1 and GATA4 may also synergize on <i>Foxl2</i> transcription, which contributes to an ovarian specific signature. Suggested stimulatory (→) and inhibitory (⊥) pathways identified herein are marked in red color.</p
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