Credit allocation based on journal impact factor and coauthorship contribution

Some research institutions demand researchers to distribute the incomes they earn from publishing papers to their researchers and/or co-authors. In this study, we deal with the Impact Factor-based ranking journal as a criteria for the correct distribution of these incomes. We also include the Authorship Credit factor for distribution of the incomes among authors, using the geometric progression of Cantor's theory and the Harmonic Credit Index. Depending on the ranking of the journal, the proposed model develops a proper publication credit allocation among all authors. Moreover, our tool can be deployed in the evaluation of an institution for a funding program, as well as calculating the amounts necessary to incentivize research among personnel.Comment: 9 pages; 3 figures; 2 table

Software-based Implementation of Secure AES Frame Encryption (SAFE) for CAN FD in Multicore Environment

The SAFE kernel software implementation was born as an inexpensive alternative from the original implementation which uses a field programmable gate array (FPGA). This paper proposes to replace the FPGA with a microprocessor, which will drastically reduce the cost of the implementation. The new implementation includes the MIMXRT1170 EVK that integrates two microprocessors, a Cortex M4 running at 400 MHz and a Cortex M7 running at 1 GHz. After the software implementation, the multicore environment met the required time deadlines and achieved the desire performance, keeping the CAN FD bus occupation at 100% and the encryption and decryption time less than 50 microseconds, which represents 25% of the microprocessor overhead. In future works the implementation could be adjusted to one microprocessor, such as the cortex M7 due to it is powerful capability to process data which also reduces the cost and the complexity of the implementation.ITESO, A. C

Credit Allocation Based on Journal Impact Factor and Co-authorship Contribution

Abstract. Some research institutions demand researchers to distribute the incomes they earn from publishing papers to their researchers and/or co-authors. In this study, we deal with the Impact Factor-based ranking journal as a criteria for the correct distribution of these incomes. We also include the Authorship Credit factor for distribution of the incomes among authors, using the geometric progression of Cantor’s theory and the Harmonic Credit Index. Depending on the ranking of the journal, the proposed model develops a proper publication credit allocation among all authors. Moreover, our tool can be deployed in the evaluation of an institution for a funding program, as well as calculating the amounts necessary to incentivize research among personnel.Keywords. Co-author credit; Impact factor; Ranking; Cantor’s succession; Harmonic credit.JEL. A12, C02, C10

Mfd Protects Against Oxidative Stress in Bacillus Subtilis Independently of its Canonical Function in DNA Repair

Background: Previous reports showed that mutagenesis in nutrient-limiting conditions is dependent on Mfd in Bacillus subtilis. Mfd initiates one type of transcription-coupled repair (TCR); this type of repair is known to target bulky lesions, like those associated with UV exposure. Interestingly, the roles of Mfd in repair of oxidative-promoted DNA damage and regulation of transcription differ. Here, we used a genetic approach to test whether Mfd protected B. subtilis from exposure to two different oxidants. Results: Wild-type cells survived tert-butyl hydroperoxide (t-BHP) exposure significantly better than Mfd-deficient cells. This protective effect was independent of UvrA, a component of the canonical TCR/nucleotide excision repair (NER) pathway. Further, our results suggest that Mfd and MutY, a DNA glycosylase that processes 8-oxoG DNA mismatches, work together to protect cells from lesions generated by oxidative damage. We also tested the role of Mfd in mutagenesis in starved cells exposed to t-BHP. In conditions of oxidative stress, Mfd and MutY may work together in the formation of mutations. Unexpectedly, Mfd increased survival when cells were exposed to the protein oxidant diamide. Under this type of oxidative stress, cells survival was not affected by MutY or UvrA. Conclusions: These results are significant because they show that Mfd mediates error-prone repair of DNA and protects cells against oxidation of proteins by affecting gene expression; Mfd deficiency resulted in increased gene expression of the OhrR repressor which controls the cellular response to organic peroxide exposure. These observations point to Mfd functioning beyond a DNA repair factor in cells experiencing oxidative stress