Cal Poly Microgrid Fixed PV Array

The Mechanical Engineering Department at California Polytechnic State University of San Luis Obispo would like an adjustable, fixed angle solar panel mount to help educate students on basic solar energy principles. Our team has developed a unique sawhorse design utilizing ideation techniques and design selection tools. The selected design allows for multiple panel adjustability and control of both azimuth and tilt angle. Safety concerns are addressed with action plans to mitigate risk. Concept prototypes to justify gearbox functionality and subsystem cohesion was utilized to reduce manufacturing issues. Manufacturing began in March 2020 and proceed through until the end of the month. The manufacturing of the mount was halted due to COVID-19, forcing the design to end strictly in a what-if manufacturing procedure to allow the construction of it to be done in future time

Individual Difference Models for Studying Rorschach Perception: An 'Indirect' Approach to Personality Assessment

162 p.Thesis (Ph.D.)--University of Illinois at Urbana-Champaign, 1970.U of I OnlyRestricted to the U of I community idenfinitely during batch ingest of legacy ETD

Monitoring volcanoes : techniques and strategies used by the staff of the Cascades Volcano Observatory, 1980-90 /

Includes bibliographical references.Mode of access: Internet

Major histocompatibility (B) complex control of the growth pattern of v-src DNA-induced primary tumors

Observations that the major histocompatibility (B) complex is a determinant of the growth pattern of Rous sarcoma virus (RSV)-induced tumors raised the question as to whether control is exerted at the level of a v-src-determined, i.e., transformation-specific, function. To investigate this point, the tumor size scores and tumor profile indices of v-src-induced tumors were compared in two lines of chickens congenic for B complex genotypes. The finding that the growth patterns of tumors, induced by v-src DNA inoculation at 6 weeks posthatch, differ in these two lines establishes that the B complex exerts control over tumor growth at the level of a v-src-determined function. The potential importance of this control, in terms of the naturally occurring case of an avian sarcoma virus infection, is suggested by the observation that the patterns of tumor growth in a given congenic line are similar whether the tumors are induced by v-src DNA or by RSV

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Cell culture and reagents Human neonatal foreskin keratinocyte cultures were initiated and propagated in MCDB153 complete medium, as previously describe

Electrophysiological assessment and pharmacological treatment of blast-induced tinnitus.

Tinnitus, the phantom perception of sound, often occurs as a clinical sequela of auditory traumas. In an effort to develop an objective test and therapeutic approach for tinnitus, the present study was performed in blast-exposed rats and focused on measurements of auditory brainstem responses (ABRs), prepulse inhibition of the acoustic startle response, and presynaptic ribbon densities on cochlear inner hair cells (IHCs). Although the exact mechanism is unknown, the "central gain theory" posits that tinnitus is a perceptual indicator of abnormal increases in the gain (or neural amplification) of the central auditory system to compensate for peripheral loss of sensory input from the cochlea. Our data from vehicle-treated rats supports this rationale; namely, blast-induced cochlear synaptopathy correlated with imbalanced elevations in the ratio of centrally-derived ABR wave V amplitudes to peripherally-derived wave I amplitudes, resulting in behavioral evidence of tinnitus. Logistic regression modeling demonstrated that the ABR wave V/I amplitude ratio served as a reliable metric for objectively identifying tinnitus. Furthermore, histopathological examinations in blast-exposed rats revealed tinnitus-related changes in the expression patterns of key plasticity factors in the central auditory pathway, including chronic loss of Arc/Arg3.1 mobilization. Using a formulation of N-acetylcysteine (NAC) and disodium 2,4-disulfophenyl-N-tert-butylnitrone (HPN-07) as a therapeutic for addressing blast-induced neurodegeneration, we measured a significant treatment effect on preservation or restoration of IHC ribbon synapses, normalization of ABR wave V/I amplitude ratios, and reduced behavioral evidence of tinnitus in blast-exposed rats, all of which accorded with mitigated histopathological evidence of tinnitus-related neuropathy and maladaptive neuroplasticity