The Generic Supersymmetric Standard Model as the Complete Theory of Supersymmetry without R-parity

The generic supersymmetric standard model is a model built from a supersymmetrized standard model field spectrum the gauge symmetries only. The popular minimal supersymmetric standard model differs from the generic version in having R-parity imposed by hand. We review an efficient formulation of the model in which all the admissible R-parity violating terms are incorporated without bias. The model gives many new interesting R-parity violating phenomenological features only started to be studied recently. Some of our recent results will be discussed, including newly identified 1-loop contributions to neutrino masses and electric dipole moments of neutron and electron. This is related to the largely overlooked R-parity violating contributions to squark and slepton mixings, which we also present in detail.Comment: 10 pages latex using espcrc2.sty (included) with a latex table and 3 eps- figure files incoporated, typos in a couple of expressions fixe

Light pseudoscalar eta and H->eta eta decay in the simplest little Higgs mode

The SU(3) simplest little Higgs model in its original framework without the so-called mu term inevitably involves a massless pseudoscalar boson eta, which is problematic for b-physics and cosmological axion limit. With the mu term introduced by hand, the eta boson acquires mass m_eta ~ mu, which can be lighter than half the Higgs boson mass in a large portion of the parameter space. In addition, the introduced mu term generates sizable coupling of H-eta-eta. The Higgs boson can dominantly decay into a pair of eta's especially when mH below the WW threshold. Another new decay channel of H->Z+eta can be dominant or compatible with H -> WW for mH above the Z+eta threshold. We show that the LEP bound on the Higgs boson mass is loosened to some extent due to this new H->eta eta decay channel as well as the reduced coupling of H-Z-Z. The Higgs boson mass bound falls to about 110 GeV for f=3-4 TeV. Since the eta boson decays mainly into a bb pair, H-> eta eta -> 4b and H-> Z eta -> Z bb open up other interesting search channels in the pursuit of the Higgs boson in the future experiments. We discuss on these issues.Comment: major modification considering the simplest little Higgs model with the mu ter

Engineered single nucleotide polymorphisms in the mosquito MEK docking site alter Plasmodium berghei development in Anopheles gambiae.

BackgroundSusceptibility to Plasmodium infection in Anopheles gambiae has been proposed to result from naturally occurring polymorphisms that alter the strength of endogenous innate defenses. Despite the fact that some of these mutations are known to introduce non-synonymous substitutions in coding sequences, these mutations have largely been used to rationalize knockdown of associated target proteins to query the effects on parasite development in the mosquito host. Here, we assay the effects of engineered mutations on an immune signaling protein target that is known to control parasite sporogonic development. By this proof-of-principle work, we have established that naturally occurring mutations can be queried for their effects on mosquito protein function and on parasite development and that this important signaling pathway can be genetically manipulated to enhance mosquito resistance.MethodsWe introduced SNPs into the A. gambiae MAPK kinase MEK to alter key residues in the N-terminal docking site (D-site), thus interfering with its ability to interact with the downstream kinase target ERK. ERK phosphorylation levels in vitro and in vivo were evaluated to confirm the effects of MEK D-site mutations. In addition, overexpression of various MEK D-site alleles was used to assess P. berghei infection in A. gambiae.ResultsThe MEK D-site contains conserved lysine residues predicted to mediate protein-protein interaction with ERK. As anticipated, each of the D-site mutations (K3M, K6M) suppressed ERK phosphorylation and this inhibition was significant when both mutations were present. Tissue-targeted overexpression of alleles encoding MEK D-site polymorphisms resulted in reduced ERK phosphorylation in the midgut of A. gambiae. Furthermore, as expected, inhibition of MEK-ERK signaling due to D-site mutations resulted in reduction in P. berghei development relative to infection in the presence of overexpressed catalytically active MEK.ConclusionMEK-ERK signaling in A. gambiae, as in model organisms and humans, depends on the integrity of conserved key residues within the MEK D-site. Disruption of signal transmission via engineered SNPs provides a purposeful proof-of-principle model for the study of naturally occurring mutations that may be associated with mosquito resistance to parasite infection as well as an alternative genetic basis for manipulation of this important immune signaling pathway

How Does Employees\u27 Branding and Employees\u27 Behavior Influence Hotel Service Quality?

The primary aim of this research is to investigate whether organisational citizenship behaviors, in-role behaviors, employees’ empowerment and employees’ branding can influence customers’ perceived service quality. A model is proposed based on three interlinked literature strands that consider human resources management, brand management, and hospitality management. The survey instruments will be developed based on the results of the focus groups and the literature. A set of relationships among employees’ behaviors, employees’ branding and service quality will be examined by structural equation modeling. The findings would encourage hotels to allocate resources to improve those critical elements that can help to achieve the best results

Off-the-record target zones:theory with an application to Hong Kong’s Currency Board

This paper provides a modelling framework for evaluating the exchange rate dynamics of a target zone regime with undisclosed bands. We generalize the literature to allow for asymmetric one-sided regimes. Market participants' beliefs concerning an undisclosed band change as they learn more about central bank intervention policy. We apply the model to Hong Kong's one-sided currency board mechanism. In autumn 2003, the Hong Kong dollar appreciated from close to 7.80 per US dollar to 7.70, as investors feared that the currency board would be abandoned. In the wake of this appreciation, the monetary authorities finally revamped the regime as a symmetric two-sided system with a narrow exchange rate band