Constraining ^(26)Al+p resonances using ^(26)Al(^3He,d)^(27)Si

The ^(26)Al(^3He,d)^(27)Si reaction was measured from 0°≤θ_(c.m.)≤35° at E(^3He)=20 MeV using a quadrupole-dipole-dipole-dipole magnetic spectrometer. States in ^(27)Si were observed above the background at 7652 and 7741 keV and upper limits were set for the state at 7592 keV. Implications for the ^(26)Al(p,γ)^(27)Si stellar reaction rate are discussed

Charge Imbalance and Bilayer 2D Electron Systems at νT=1\nu_T = 1

We use interlayer tunneling to study bilayer 2D electron systems at $\nu_T = 1$ over a wide range of charge density imbalance, $\Delta \nu =\nu_1-\nu_2$, between the two layers. We find that the strongly enhanced tunneling associated with the coherent excitonic $\nu_T = 1$ phase at small layer separation can survive at least up to an imbalance of $\Delta \nu$ = 0.5, i.e $(\nu_1, \nu_2)$ = (3/4, 1/4). Phase transitions between the excitonic $\nu_T = 1$ state and bilayer states which lack significant interlayer correlations can be induced in three different ways: by increasing the effective interlayer spacing $d/\ell$, the temperature $T$, or the charge imbalance, $\Delta \nu$. We observe that close to the phase boundary the coherent $\nu_T = 1$ phase can be absent at $\Delta \nu$ = 0, present at intermediate $\Delta \nu$, but then absent again at large $\Delta \nu$, thus indicating an intricate phase competition between it and incoherent quasi-independent layer states. At zero imbalance, the critical $d/\ell$ shifts linearly with temperature, while at $\Delta \nu$ = 1/3 the critical $d/\ell$ is only weakly dependent on $T$. At $\Delta \nu$ = 1/3 we report the first observation of a direct phase transition between the coherent excitonic $\nu_T = 1$ bilayer integer quantum Hall phase and the pair of single layer fractional quantized Hall states at $\nu_1$ = 2/3 and $\nu_2=1/3$.Comment: 13 pages, 8 postscript figures. Final published versio

Maternal modulation of paternal effects on offspring development.

The paternal transmission of environmentally induced phenotypes across generations has been reported to occur following a number of qualitatively different exposures and appear to be driven, at least in part, by epigenetic factors that are inherited via the sperm. However, previous studies of paternal germline transmission have not addressed the role of mothers in the propagation of paternal effects to offspring. We hypothesized that paternal exposure to nutritional restriction would impact male mate quality and subsequent maternal reproductive investment with consequences for the transmission of paternal germline effects. In the current report, using embryo transfer in mice, we demonstrate that sperm factors in adult food restricted males can influence growth rate, hypothalamic gene expression and behaviour in female offspring. However, under natural mating conditions females mated with food restricted males show increased pre- and postnatal care, and phenotypic outcomes observed during embryo transfer conditions are absent or reversed. We demonstrate that these compensatory changes in maternal investment are associated with a reduced mate preference for food restricted males and elevated gene expression within the maternal hypothalamus. Therefore, paternal experience can influence offspring development via germline inheritance, but mothers can serve as a modulating factor in determining the impact of paternal influences on offspring development