341 research outputs found
Galaxy Zoo: Are Bars Responsible for the Feeding of Active Galactic Nuclei at 0.2 < z < 1.0?
We present a new study investigating whether active galactic nuclei (AGN)
beyond the local universe are preferentially fed via large-scale bars. Our
investigation combines data from Chandra and Galaxy Zoo: Hubble (GZH) in the
AEGIS, COSMOS, and GOODS-S surveys to create samples of face-on, disc galaxies
at 0.2 < z < 1.0. We use a novel method to robustly compare a sample of 120 AGN
host galaxies, defined to have 10^42 erg/s < L_X < 10^44 erg/s, with inactive
control galaxies matched in stellar mass, rest-frame colour, size, Sersic
index, and redshift. Using the GZH bar classifications of each sample, we
demonstrate that AGN hosts show no statistically significant enhancement in bar
fraction or average bar likelihood compared to closely-matched inactive
galaxies. In detail, we find that the AGN bar fraction cannot be enhanced above
the control bar fraction by more than a factor of two, at 99.7% confidence. We
similarly find no significant difference in the AGN fraction among barred and
non-barred galaxies. Thus we find no compelling evidence that large-scale bars
directly fuel AGN at 0.2<z<1.0. This result, coupled with previous results at
z=0, implies that moderate-luminosity AGN have not been preferentially fed by
large-scale bars since z=1. Furthermore, given the low bar fractions at z>1,
our findings suggest that large-scale bars have likely never directly been a
dominant fueling mechanism for supermassive black hole growth.Comment: 13 pages, 5 figures, 2 tables, accepted by MNRA
Genomic and transcriptional analysis of protein heterogeneity of the honeybee venom allergen Api m 6
Several components of honeybee venom are known to cause allergenic responses in humans and other vertebrates. One such component, the minor allergen Api m 6, has been known to show amino acid variation but the genetic mechanism for this variation is unknown. Here we show that Api m 6 is derived from a single locus, and that substantial protein-level variation has a simple genome-level cause, without the need to invoke multiple loci or alternatively spliced exons. Api m 6 sits near a misassembled section of the honeybee genome sequence, and we propose that a substantial number of indels at and near Api m 6 might be the root cause of this misassembly. We suggest that genes such as Api m 6 with coding-region or untranslated region indels might have had a strong effect on the assembly of this draft of the honeybee genome
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