88 research outputs found
<i>Trypanosoma brucei rhodesiense</i> transmitted by a single tsetse fly bite in vervet monkeys as a model of human African trypanosomiasis
- Author
- AA Poltera
- AA Poltera
- B Bouteille
- C Gichuki
- C. M. R. Turner
- CE Clayton
- CMR Turner
- D Ripamonti
- DA Moore
- Daniel Masiga
- David Mwangangi
- DH Smith
- DK Masiga
- DL Emery
- E Fink
- Enock Matovu
- FM Fevre
- FW Jennings
- GC Jenkins
- H Schmidt
- J Naessens
- JD Dargie
- JE Chisi
- JM Ndung'u
- JM Sternberg
- John K. Thuita
- John M. Kagira
- JR Baker
- L MacLean
- M Odiit
- N Mabbot
- NW Maina
- PGE Kennedy
- S Bisser
- S Gross
- S Urwyler
- Serap Aksoy
- SO Omotainse
- TW Miezan
- V Jamonneau
- WE Ormerod
- WI Morrison
- WJ Herbert
- WJ Herbert
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2008
- Field of study
Sleeping sickness is caused by a species of trypanosome blood parasite that is transmitted by tsetse flies. To understand better how infection with this parasite leads to disease, we provide here the most detailed description yet of the course of infection and disease onset in vervet monkeys. One infected tsetse fly was allowed to feed on each host individual, and in all cases infections were successful. The characteristics of infection and disease were similar in all hosts, but the rate of progression varied considerably. Parasites were first detected in the blood 4-10 days after infection, showing that migration of parasites from the site of fly bite was very rapid. Anaemia was a key feature of disease, with a reduction in the numbers and average size of red blood cells and associated decline in numbers of platelets and white blood cells. One to six weeks after infection, parasites were observed in the cerebrospinal fluid (CSF), indicating that they had moved from the blood into the brain; this was associated with a white cell infiltration. This study shows that fly-transmitted infection in vervets accurately mimics human disease and provides a robust model to understand better how sleeping sickness develops
The effects of symmetry on the dynamics of antigenic variation
- Author
- A Fässler
- A Marcello
- B Adams
- B Fiedler
- C Jones
- C Newbold
- C Stockdale
- CA Pinto
- CMR Turner
- CO Buckee
- CO Buckee
- DG Aronson
- DJ Smith
- DL Martin
- H Farkas
- I Stewart
- IM Rouzine
- JD Barry
- JD Murray
- JHP Dawes
- JL Mitchell
- JL Muñoz-Jordán
- JR Gog
- JW Swift
- KA Lythgoe
- KB Blyuss
- Konstantin B. Blyuss
- KW Deitsch
- LJ Abu-Raddad
- LM Pecora
- M Dellnitz
- M Golubitsky
- M Golubitsky
- M Golubitsky
- M Golubitsky
- M Recker
- M Recker
- M Recker
- M Recker
- M Recker
- MGM Gomes
- MJ Gardner
- O Bagasra
- P Ashwin
- P Ashwin
- P Borst
- P De Leenheer
- P Hagblom
- P Minaev
- PL Buono
- R Antia
- S Gupta
- S Gupta
- S Gupta
- S Kyes
- S Lutenschlager
- SA Frank
- SA Frank
- SH Strogatz
- V Calvez
- Y Jiang
- Z Agur
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 20/02/2012
- Field of study
In the studies of dynamics of pathogens and their interactions with a host
immune system, an important role is played by the structure of antigenic
variants associated with a pathogen. Using the example of a model of antigenic
variation in malaria, we show how many of the observed dynamical regimes can be
explained in terms of the symmetry of interactions between different antigenic
variants. The results of this analysis are quite generic, and have wider
implications for understanding the dynamics of immune escape of other
parasites, as well as for the dynamics of multi-strain diseases.Comment: 21 pages, 4 figures; J. Math. Biol. (2012), Online Firs
The origins of the trypanosome genome strains Trypanosoma brucei brucei TREU 927, T. b. gambiense DAL 972, T. vivax Y486 and T. congolense IL3000
- Author
- A MacLeod
- A MacLeod
- ALW De Gee
- AP Cortez
- AP Jackson
- AP Jackson
- B Dero
- B Wellde
- C Felu
- CA Hoare
- CJ Young
- CMR Turner
- CMR Turner
- D Jefferies
- D Mehlitz
- D Richner
- D Steverding
- DG Godfrey
- DK Masiga
- E Goedbloed
- E Goedbloed
- E Zweygarth
- ER Adams
- F Mathieu-Daude
- FJ van Deursen
- FK Dar
- G Hide
- G Hide
- GD Kanmogne
- GD Kanmogne
- H Hirumi
- H Hirumi
- H Hirumi
- HKW Gathuo
- I Morlais
- ID Gumm
- JD Barry
- JK Gashumba
- JK Stiles
- JR Helm
- JR Stevens
- JR Stevens
- JW Magona
- L Peacock
- LE Stephen
- M Berriman
- M Radwanska
- MA Gray
- N Biteau
- N Downey
- N Downey
- O Balmer
- P Butikofer
- P Dukes
- P Dukes
- P Leeflang
- PAO Majiwa
- PAO Majiwa
- PB Hamilton
- PR Gardiner
- PR Gardiner
- R Brun
- RA Masake
- RAL Bayne
- RP Beecroft
- S D'Archivio
- S Utz
- SE Melville
- SK Dickin
- SK Moloo
- SK Moloo
- T Bromidge
- T Sakurai
- V Coustou
- W Gibson
- W Gibson
- W Gibson
- WC Gibson
- WC Gibson
- WC Gibson
- Wendy Gibson
- Y Eshita
- Publication venue
- BioMed Central
- Publication date
- 01/01/2012
- Field of study
The genomes of several tsetse-transmitted African trypanosomes (Trypanosoma brucei brucei, T. b. gambiense, T. vivax, T. congolense) have been sequenced and are available to search online. The trypanosome strains chosen for the genome sequencing projects were selected because they had been well characterised in the laboratory, but all were isolated several decades ago. The purpose of this short review is to provide some background information on the origins and biological characterisation of these strains as a source of reference for future users of the genome data. With high throughput sequencing of many more trypanosome genomes in prospect, it is important to understand the phylogenetic relationships of the genome strains
Phylogeography and Taxonomy of Trypanosoma brucei
- Author
- A MacLeod
- A MacLeod
- A MacLeod
- A MacLeod
- A Tait
- Adalgisa Caccone
- C Cordon-Obras
- C De Greef
- CA Hoare
- CMR Turner
- CMR Turner
- D Ebert
- D Mehlitz
- D Posada
- D Swofford
- DG Godfrey
- EC Agbo
- EK Komba
- EM Fevre
- F Njiokou
- F Ronquist
- G Evanno
- G Hide
- G Hide
- G Hide
- G Simo
- HV Xong
- JAA Nylander
- JJ Bull
- JK Pritchard
- Jon S. Beadell
- JR Baker
- L Berrang-Ford
- L Jenni
- L Jost
- L MacLean
- L MacLean
- LJ Morrison
- M Clement
- M Hasegawa
- M Koffi
- M Koffi
- M Radwanska
- MP Barrett
- N Biteau
- NG Crawford
- O Balmer
- O Balmer
- O Balmer
- Oliver Balmer
- P Cattand
- P Librado
- P Maser
- P Paindavoine
- P Truc
- PG Coleman
- Philippe Solano
- PJ Hotez
- PP Simarro
- R Brun
- R Brun
- R Heller
- RE Cibulskis
- SC Welburn
- T Jombart
- V Jamonneau
- W Bitter
- WC Gibson
- WC Gibson
- WC Gibson
- WC Gibson
- WC Gibson
- WC Gibson
- WC Gibson
- WC Gibson
- WC Gibson
- WC Gibson
- Wendy Gibson
- ZK Njiru
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
Trypanosoma brucei, the parasite causing human African trypanosomiasis (sleeping sickness) across sub-Saharan Africa is traditionally split into three subspecies: T. b. gambiense (Tbg), causing a chronic form of human disease in West and Central Africa; T. b. rhodesiense (Tbr), causing an acute form of human disease in East and Southern Africa; and T. b. brucei (Tbb), which is restricted to animals. Tbg is further split into Tbg group 1 and Tbg group 2. Better understanding the evolutionary relationships between these groups may help to shed light on the epidemiology of sleeping sickness. Here, we used three different types of genetic markers to investigate the phylogeographic relationships among the four groups across a large portion of their range. Our results confirm the distinctiveness of Tbg group 1 while highlighting the extremely close relationships among the other three taxa. In particular, Tbg group 2 was closely related to Tbb, while Tbr appeared to be a variant of Tbb, differing only in its phenotype of human infectivity. The wide geographic distribution of the gene conferring human infectivity (SRA) and the fact that it is readily exchanged among lineages of T. brucei in eastern Africa suggests that human-infective trypanosomes have access to an extensive gene pool with which to respond to selective pressures such as drugs
Leishmania HASP and SHERP Genes are Required for In Vivo Differentiation, Parasite Transmission and Virulence Attenuation in the Host
- Author
- A Cassola
- A Dostálová
- A Selvapandiyan
- A Warburg
- A Zinoviev
- A-LL Bañuls
- B Moore
- BL Herwaldt
- C Mary
- CMR Turner
- CMR Turner
- D Moreira
- D Paape
- DA Baker
- Deborah F. Smith
- DL Sacks
- DL Sacks
- DP Depledge
- DP Depledge
- E Gómez Lahoz
- E Knuepfer
- E Vassella
- F Colbére-Garapin
- G Cohen-Freue
- G Dick
- G Leslie
- Hamide Aslan
- HC Lima
- HHP Price
- HM FIinn
- HM Flinn
- HP Price
- J Myskova
- J Sádlová
- J Sádlová
- J Sádlová
- J. Volf
- Jan Votýpka
- JH LeBowitz
- Johannes S. P. Doehl
- Jovana Sádlová
- JS Oliveira
- K Blochlinger
- Kateřina Pružinová
- KG Le Roch
- L Nicolas
- LL Walters
- LL Walters
- LM Maclean
- M Engstler
- M Kimura
- MD Abràmoff
- ME Rogers
- ME Rogers
- ME Rogers
- ME Rogers
- ML Cunningham
- N Kimblin
- NC Peters
- P Chakraborty
- P Kropf
- P MacGregor
- P Volf
- P Volf
- PA Bates
- PD Ready
- Petr Volf
- PG McKean
- PG McKean
- PG McKean
- PG Nugent
- PW Denny
- R Brun
- R Lainson
- R Wilson
- RG Titus
- RMR Coulson
- S Besteiro
- S Dean
- S Kamhawi
- Shaden Kamhawi
- SM Gossage
- SM Lei
- Sonia Metangmo
- Stephen M. Beverley
- TM Alce
- V Saf’janova
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2017
- Field of study
Differentiation of extracellular Leishmania promastigotes within their sand fly vector, termed metacyclogenesis, is considered to be essential for parasites to regain mammalian host infectivity. Metacyclogenesis is accompanied by changes in the local parasite environment, including secretion of complex glycoconjugates within the promastigote secretory gel and colonization and degradation of the sand fly stomodeal valve. Deletion of the stage-regulated HASP and SHERP genes on chromosome 23 of Leishmania major is known to stall metacyclogenesis in the sand fly but not in in vitro culture. Here, parasite mutants deficient in specific genes within the HASP/SHERP chromosomal region have been used to investigate their role in metacyclogenesis, parasite transmission and establishment of infection. Metacyclogenesis was stalled in HASP/SHERP mutants in vivo and, although still capable of osmotaxis, these mutants failed to secrete promastigote secretory gel, correlating with a lack of parasite accumulation in the thoracic midgut and failure to colonise the stomodeal valve. These defects prevented parasite transmission to a new mammalian host. Sand fly midgut homogenates modulated parasite behaviour in vitro, suggesting a role for molecular interactions between parasite and vector in Leishmania development within the sand fly. For the first time, stage-regulated expression of the small HASPA proteins in Leishmania (Leishmania) has been demonstrated: HASPA2 is expressed only in extracellular promastigotes and HASPA1 only in intracellular amastigotes. Despite its lack of expression in amastigotes, replacement of HASPA2 into the null locus background delays onset of pathology in BALB/c mice. This HASPA2-dependent effect is reversed by HASPA1 gene addition, suggesting that the HASPAs may have a role in host immunomodulation
Gene Conversion Transfers the GAF-A Domain of Phosphodiesterase TbrPDEB1 to One Allele of TbrPDEB2 of Trypanosoma brucei
- Author
- AJ Jeffreys
- C Stockdale
- CL Hartley
- CMR Turner
- D Horn
- D Le Ray
- E Pays
- E Pays
- E Wirtz
- Edith Luginbuehl
- F Hesse
- G Burkard
- GAM Cross
- GAM Cross
- H Wang
- JA Beavo
- Jayne Raper
- JD Bangs
- JE Taylor
- JF Watkins
- JM Chen
- K Schlaeppi
- L Peacock
- L Wentzinger
- LHT Van der Ploeg
- M Berriman
- M Conti
- M Oberholzer
- M Oberholzer
- MN Papadakis
- O Dreesen
- R Brun
- R McCullough
- RL Barnes
- S Kunz
- S Laxman
- S Martinez
- SR Judd
- Stefan Kunz
- Thomas Seebeck
- W Gibson
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2009
- Field of study
Cyclic nucleotide specific phosphodiesterases are important regulators of cyclic nucleotide signalling in eukaryotes. In many organisms, including humans and trypanosomes, some of these enzymes contain specific domains (GAF domains) that bind cyclic nucleotides, and that are involved in the regulation of the catalytic domain. In the parasitic protozoon that causes human sleeping sickness, Trypanosoma brucei, two closely related phosphodiesterases each contain two such GAF domains, GAF-A and GAF-B. Their genes are tandemly located on chromosome 9, spaced by only a few thousand nucleotides. We here show that a gene conversion event has exchanged the region that codes for the GAF-A domain of the downstream gene by the closely similar corresponding sequence of the upstream gene. This domain exchange has no effect on intracellular localization of the two enzymes. The gene conversion event has occurred in one particular strain of trypanosomes (Lister427) and is found in all its derivatives, but not in any other strain or isolate. The presence or absence of this gene conversion represents a useful analytical marker for the stringent discrimination of Lister427 derivatives from other trypanosome strains
Measurement of single top-quark production in association with a W boson in the single-lepton channel at \sqrt{s} = 8\,\text {TeV} with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abud AA
- Abulaiti Y
- Acharya BS
- Achkar B
- Adachi S
- Adam L
- Adamczyk L
- Adamek L
- Adelman J
- Adersberger M
- Adiguzel A
- Adorni S
- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agaras MN
- Aggarwal A
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahmadov F
- Ahmed WS
- Ai X
- Aielli G
- Akatsuka S
- Akesson TPA
- Akilli E
- Akimov A
- Al Khoury K
- Alberghi GL
- Albert J
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexandre D
- Alexopoulos T
- Alfonsi A
- Alfonsi F
- Alhroob M
- Ali B
- Aliev M
- Alimonti G
- Alison J
- Alkire SP
- Allaire C
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alvarez Estevez M
- Alvarez Piqueras D
- Alviggi MG
- Ambler A
- Ambroz L
- Amelung C
- Amidei D
- Amoroso S
- Amrouche CS
- An F
- Anastopoulos C
- Andari N
- Andeen T
- Anders CF
- Anders JK
- Andreazza A
- Andrei V
- Anelli CR
- Angelidakis S
- Angerami A
- Anisenkov A
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Antrim DJA
- Anulli F
- Aoki M
- Aparisi Pozo JA
- Arabidze G
- Araque JP
- Araya ST
- Arcangeletti C
- Arce ATH
- Arduh FA
- Arguin J-F
- Argyropoulos S
- Arling J-H
- Armadans RC
- Armbruster AJ
- Armstrong A
- Arnaez O
- Arnold H
- Artoni G
- Artz S
- Asai S
- Asbah N
- Asimakopoulou EM
- Asquith L
- Assahsah J
- Assamagan K
- Astalos R
- Astigarraga MEP
- Atkin RJ
- Atkinson M
- Atlay NB
- Atmani H
- Augsten K
- Avolio G
- Avramidou R
- Ayoub MK
- Azoulay AM
- Azuelos G
- Bachacou H
- Bachas K
- Backes M
- Backman F
- Bagnaia P
- Bahmani M
- Bahrasemani H
- Bailey AJ
- Bailey VR
- Baines JT
- Bajic M
- Bakalis C
- Baker OK
- Bakker PJ
- Balaji S
- Baldin EM
- Balek P
- Balli F
- Balunas WK
- Balz J
- Banas E
- Bandyopadhyay A
- Banerjee S
- Bannoura AAE
- Barak L
- Barbe WM
- Barberio EL
- Barberis D
- Barbero M
- Barbour G
- Barillari T
- Barisits M-S
- Barkeloo J
- Barklow T
- Barnea R
- Barnes SL
- Barnett BM
- Barnett RM
- Barnovska-Blenessy Z
- Baroncelli A
- Barone G
- Barr AJ
- Barreiro F
- Barsov S
- Bartoldus R
- Bartolini G
- Barton AE
- Bartos P
- Basalaev A
- Bassalat A
- Basso MJ
- Bates RL
- Batlamous S
- Batley JR
- Batool B
- Battaglia M
- Bauce M
- Bauer F
- Bauer KT
- Bawa HS
- Beacham JB
- Beau T
- Beauchemin PH
- Becherer F
- Bechtle P
- Beck HC
- Beck HP
- Becker K
- Becker M
- Becot C
- Beddall A
- Beddall AJ
- Bednyakov VA
- Bedognetti M
- Bee CP
- Beermann TA
- Begalli M
- Begel M
- Behera A
- Behr JK
- Beisiegel F
- Bell AS
- Bella G
- Bella LA
- Bellagamba L
- Bellerive A
- Bellos P
- Beloborodov K
- Belotskiy K
- Belyaev NL
- Benchekroun D
- Benekos N
- Benhammou Y
- Benjamin DP
- Benoit M
- Bensinger JR
- Bentvelsen S
- Beresford L
- Beretta M
- Berge D
- Berger N
- Bergmann B
- Bergsten LJ
- Beringer J
- Berlendis S
- Berlingen JM
- Bernardi G
- Bernius C
- Bernlochner FU
- Berry T
- Bertella C
- Bertram IA
- Besson N
- Bethani A
- Bethke S
- Betti A
- Bevan AJ
- Beyer J
- Bhattacharya DS
- Bhattarai P
- Bi R
- Bianchi RM
- Biebel O
- Biedermann D
- Bielski R
- Bierwagen K
- Biesuz N
- Biglietti M
- Billoud TR
- Bindi M
- Bingul A
- Bini C
- Biondi S
- Birman M
- Bisanz T
- Biswal JP
- Biswas D
- Bitadze A
- Bittrich C
- Bjorke K
- Black KM
- Blazek T
- Bloch I
- Blocker C
- Blue A
- Blumenschein U
- Bobbink GJ
- Bobrovnikov VS
- Bocchetta SS
- Bocci A
- Boeriu OEV
- Bogavac D
- Bogdanchikov AG
- Bohm C
- Boisvert V
- Bokan P
- Bold T
- Boldyrev AS
- Bolz AE
- Bomben M
- Bona M
- Bonilla JS
- Boonekamp M
- Booth CD
- Borecka-Bielska HM
- Borisov A
- Borissov G
- Bortfeldt J
- Bortoletto D
- Boscherini D
- Bosman M
- Bostanabad MG
- Bouaouda K
- Boudreau J
- Bouhova-Thacker E
- Boumediene D
- Bourdarios CA
- Boutle SK
- Boveia A
- Boyd J
- Boye D
- Boyko IR
- Bozson AJ
- Bracinik J
- Brahimi N
- Brandt G
- Brandt O
- Braren F
- Brau B
- Brau JE
- Brendlinger K
- Brenner L
- Brenner R
- Bressler S
- Bret MC
- Brickwedde B
- Briglin DL
- Britton D
- Britzger D
- Brock I
- Brock R
- Brooijmans G
- Brooks WK
- Brost E
- Broughton JH
- Bruncko D
- Bruni A
- Bruni G
- Bruni LS
- Bruno S
- Bruschi M
- Bruscino N
- Bryant P
- Bryngemark L
- Buanes T
- Buat Q
- Buchholz P
- Buckley AG
- Budagov IA
- Bugge MK
- Buhrer F
- Bulekov O
- Burch TJ
- Burdin S
- Burgard CD
- Burger AM
- Burghgrave B
- Burr JTP
- Burton CD
- Burzynski JC
- Buscher V
- Buschmann E
- Bussey PJ
- Butler JM
- Buttar CM
- Butterworth JM
- Butti P
- Buttinger W
- Buzatu A
- Buzykaev AR
- Bylund OB
- Cabras G
- Cabrera Urban S
- Caforio D
- Cai H
- Cairo VMM
- Cakir IT
- Cakir O
- Calace N
- Calafiura P
- Calandri A
- Calderini G
- Calfayan P
- Callea G
- Caloba LP
- Caltabiano A
- Calvente Lopez S
- Calvet D
- Calvet S
- Calvet TP
- Calvetti M
- Camarda S
- Camarero Munoz D
- Camarri P
- Cameron D
- Camincher C
- Campana S
- Campanelli M
- Camplani A
- Campoverde A
- Canale V
- Canesse A
- Cantero J
- Cao T
- Cao Y
- Capua M
- Cardarelli R
- Cardillo F
- Carducci G
- Carli I
- Carli T
- Carlino G
- Carlson BT
- Carminati L
- Carney RMD
- Caron S
- Carquin E
- Carra S
- Carter JWS
- Casado MP
- Casha AF
- Casper DW
- Castelijn R
- Castillo Gimenez V
- Castillo FL
- Castillo LRF
- Castro NF
- Catinaccio A
- Catmore JR
- Cattai A
- Caudron J
- Cavaliere V
- Cavallaro E
- Cavalli-Sforza M
- Cavasinni V
- Celebi E
- Ceradini F
- Cerda Alberich L
- Cerny K
- Cerqueira AS
- Cerri A
- Cerrito L
- Cerutti F
- Cervelli A
- Cetin SA
- Chadi Z
- Chakraborty D
- Chan SK
- Chan WS
- Chan WY
- Chapman JD
- Chargeishvili B
- Charlton DG
- Charman TP
- Chau CC
- Che S
- Chekanov S
- Chekulaev S
- Chelkov GA
- Chelstowska MA
- Chen B
- Chen C
- Chen CH
- Chen H
- Chen J
- Chen J
- Chen S
- Chen SJ
- Chen X
- Chen Y
- Chen Y-H
- Cheng HC
- Cheng HJ
- Cheplakov A
- Cheremushkina E
- Cheu E
- Cheung K
- Chevalerias TJA
- Chevalier L
- Chiarella V
- Chiarelli G
- Chiodini G
- Chisholm AS
- Chitan A
- Chiu I
- Chiu YH
- Chizhov M
- Choi K
- Chomont AR
- Chouridou S
- Chow YS
- Chu MC
- Chu X
- Chudoba J
- Chuinard AJ
- Chwastowski JJ
- Chytka L
- Cieri D
- Ciesla KM
- Cinca D
- Cindro V
- Ciocio A
- Cirotto F
- Citron ZH
- Citterio M
- Ciubotaru DA
- Ciungu BM
- Clark A
- Clark MR
- Clark PJ
- Clement C
- Coadou Y
- Cobal M
- Coccaro A
- Cochran J
- Cohen H
- Coimbra AEC
- Colasurdo L
- Cole B
- Colijn AP
- Collot J
- Coniavitis E
- Connell SH
- Connelly IA
- Constantinescu S
- Conventi F
- Cooper-Sarkar AM
- Corga KDV
- Cormier F
- Cormier KJR
- Cornell SD
- Corpe LD
- Corradi M
- Correia AMH
- Corrigan EE
- Corriveau F
- Costa MJ
- Costanza F
- Costanzo D
- Coutinho YA
- Cowan G
- Cowley JW
- Crane J
- Cranmer K
- Crawley SJ
- Creager RA
- Crepe-Renaudin S
- Crescioli F
- Cristinziani M
- Croft V
- Crosetti G
- Cueto A
- Cukierman AR
- Cunningham WR
- Czekierda S
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- Zhemchugov A
- Zheng Z
- Zhong D
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- Zieminska D
- Zimine N
- Zimmermann S
- Zinonos Z
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- Zorbas TG
- Zou R
- Zu Theenhausen HM
- Zwalinski L
- Publication venue
- 'Springer Fachmedien Wiesbaden GmbH'
- Publication date
- 01/01/2021
- Field of study
The production cross-section of a top quark in association with a W boson is measured using proton–proton collisions at \sqrt{s} = 8\,\text {TeV}. The dataset corresponds to an integrated luminosity of 20.2\,\text {fb}^{-1}, and was collected in 2012 by the ATLAS detector at the Large Hadron Collider at CERN. The analysis is performed in the single-lepton channel. Events are selected by requiring one isolated lepton (electron or muon) and at least three jets. A neural network is trained to separate the tW signal from the dominant t{\bar{t}} background. The cross-section is extracted from a binned profile maximum-likelihood fit to a two-dimensional discriminant built from the neural-network output and the invariant mass of the hadronically decaying W boson. The measured cross-section is \sigma _{tW} = 26 \pm 7\,\text {pb}, in good agreement with the Standard Model expectation
Measurements of Higgs bosons decaying to bottom quarks from vector boson fusion production with the ATLAS experiment at √=13TeV
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abud AA
- Abulaiti Y
- Acharya BS
- Achkar B
- Adam L
- Adamczyk L
- Adamek L
- Adelman J
- Adiguzel A
- Adorni S
- Adye T
- Affolder AA
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- Agaras MN
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- Agheorghiesei C
- Aguilar-Saavedra JA
- Agullo PM
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- Alberghi GL
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- Buschmann E
- Bussey PJ
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- Butterworth JM
- Buttinger W
- Buzykaev AR
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- Calafiura P
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- Camerlingo MT
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- Cheremushkina E
- Cheu E
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- Chevalérias TJA
- Chiarella V
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- Chitan A
- Chiu I
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- Cindro V
- Cioară IA
- Ciocio A
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- Dumitriu AE
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- Zinonos Z
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- Zorbas TG
- Zou R
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 23/06/2021
- Field of study
The paper presents a measurement of the Standard Model Higgs Boson decaying to b-quark pairs in the vector boson fusion (VBF) production mode. A sample corresponding to 126 fb−1 of s√=13TeV proton–proton collision data, collected with the ATLAS experiment at the Large Hadron Collider, is analyzed utilizing an adversarial neural network for event classification. The signal strength, defined as the ratio of the measured signal yield to that predicted by the Standard Model for VBF Higgs production, is measured to be 0.95+0.38−0.36 , corresponding to an observed (expected) significance of 2.6 (2.8) standard deviations from the background only hypothesis. The results are additionally combined with an analysis of Higgs bosons decaying to b-quarks, produced via VBF in association with a photon
Muon reconstruction and identification efficiency in ATLAS using the full Run 2 pp collision data set at \sqrt{s}=13 TeV
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
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- Zhemchugov A
- Zheng Z
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- Zivkovic L
- Zobernig G
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- Zorbas TG
- Zou R
- Zwalinski L
- Publication venue
- 'Springer Fachmedien Wiesbaden GmbH'
- Publication date
- 01/01/2021
- Field of study
This article documents the muon reconstruction and identification efficiency obtained by the ATLAS experiment for 139 \hbox {fb}^{-1} of pp collision data at \sqrt{s}=13 TeV collected between 2015 and 2018 during Run 2 of the LHC. The increased instantaneous luminosity delivered by the LHC over this period required a reoptimisation of the criteria for the identification of prompt muons. Improved and newly developed algorithms were deployed to preserve high muon identification efficiency with a low misidentification rate and good momentum resolution. The availability of large samples of Z\rightarrow \mu \mu and J/\psi \rightarrow \mu \mu decays, and the minimisation of systematic uncertainties, allows the efficiencies of criteria for muon identification, primary vertex association, and isolation to be measured with an accuracy at the per-mille level in the bulk of the phase space, and up to the percent level in complex kinematic configurations. Excellent performance is achieved over a range of transverse momenta from 3 GeV to several hundred GeV, and across the full muon detector acceptance of |\eta |<2.7
Dijet Resonance Search with Weak Supervision Using root S=13 TeV pp Collisions in the ATLAS Detector
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- Abhayasinghe DK
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- Zaripovas DR
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- Zemaityte G
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- Zhemchugov A
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- Zu Theenhausen HM
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- Publication venue
- AMER PHYSICAL SOC
- Publication date
- 25/09/2020
- Field of study
This Letter describes a search for narrowly resonant new physics using a machine-learning anomaly
detection procedure that does not rely on signal simulations for developing the analysis selection. Weakly
supervised learning is used to train classifiers directly on data to enhance potential signals. The targeted
topology is dijet events and the features used for machine learning are the masses of the two jets. The
resulting analysis is essentially a three-dimensional search A → BC, for mA ∼ OðTeVÞ, mB; mC ∼
Oð100 GeVÞ and B, C are reconstructed as large-radius jets, without paying a penalty associated with
a large trials factor in the scan of the masses of the two jets. The full run 2 ffiffi
s p ¼ 13 TeV pp collision
dataset of 139 fb−1 recorded by the ATLAS detector at the Large Hadron Collider is used for the search.
There is no significant evidence of a localized excess in the dijet invariant mass spectrum between 1.8 and
8.2 TeV. Cross-section limits for narrow-width A, B, and C particles vary with mA, mB, and mC. For
example, when mA ¼ 3 TeV and mB ≳ 200 GeV, a production cross section between 1 and 5 fb is
excluded at 95% confidence level, depending on mC. For certain masses, these limits are up to 10 times
more sensitive than those obtained by the inclusive dijet search. These results are complementary to the
dedicated searches for the case that B and C are standard model boson
- …