1,895 research outputs found

    SIC-POVMs and the Extended Clifford Group

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    We describe the structure of the extended Clifford Group (defined to be the group consisting of all operators, unitary and anti-unitary, which normalize the generalized Pauli group (or Weyl-Heisenberg group as it is often called)). We also obtain a number of results concerning the structure of the Clifford Group proper (i.e. the group consisting just of the unitary operators which normalize the generalized Pauli group). We then investigate the action of the extended Clifford group operators on symmetric informationally complete POVMs (or SIC-POVMs) covariant relative to the action of the generalized Pauli group. We show that each of the fiducial vectors which has been constructed so far (including all the vectors constructed numerically by Renes et al) is an eigenvector of one of a special class of order 3 Clifford unitaries. This suggests a strengthening of a conjuecture of Zauner's. We give a complete characterization of the orbits and stability groups in dimensions 2-7. Finally, we show that the problem of constructing fiducial vectors may be expected to simplify in the infinite sequence of dimensions 7, 13, 19, 21, 31,... . We illustrate this point by constructing exact expressions for fiducial vectors in dimensions 7 and 19.Comment: 27 pages. Version 2 contains some additional discussion of Zauner's original conjecture, and an alternative, possibly stronger version of the conjecture in version 1 of this paper; also a few other minor improvement

    Pauli Diagonal Channels Constant on Axes

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    We define and study the properties of channels which are analogous to unital qubit channels in several ways. A full treatment can be given only when the dimension d is a prime power, in which case each of the (d+1) mutually unbiased bases (MUB) defines an axis. Along each axis the channel looks like a depolarizing channel, but the degree of depolarization depends on the axis. When d is not a prime power, some of our results still hold, particularly in the case of channels with one symmetry axis. We describe the convex structure of this class of channels and the subclass of entanglement breaking channels. We find new bound entangled states for d = 3. For these channels, we show that the multiplicativity conjecture for maximal output p-norm holds for p=2. We also find channels with behavior not exhibited by unital qubit channels, including two pairs of orthogonal bases with equal output entropy in the absence of symmetry. This provides new numerical evidence for the additivity of minimal output entropy

    Qubit Channels Can Require More Than Two Inputs to Achieve Capacity

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    We give examples of qubit channels that require three input states in order to achieve the Holevo capacity.Comment: RevTex, 5 page, 4 figures

    Mutually unbiased bases: tomography of spin states and star-product scheme

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    Mutually unbiased bases (MUBs) are considered within the framework of a generic star-product scheme. We rederive that a full set of MUBs is adequate for a spin tomography, i.e. knowledge of all probabilities to find a system in each MUB-state is enough for a state reconstruction. Extending the ideas of the tomographic-probability representation and the star-product scheme to MUB-tomography, dequantizer and quantizer operators for MUB-symbols of spin states and operators are introduced, ordinary and dual star-product kernels are found. Since MUB-projectors are to obey specific rules of the star-product scheme, we reveal the Lie algebraic structure of MUB-projectors and derive new relations on triple- and four-products of MUB-projectors. Example of qubits is considered in detail. MUB-tomography by means of Stern-Gerlach apparatus is discussed.Comment: 11 pages, 1 table, partially presented at the 17th Central European Workshop on Quantum Optics (CEWQO'2010), June 6-11, 2010, St. Andrews, Scotland, U

    Extending additivity from symmetric to asymmetric channels

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    We prove a lemma which allows one to extend results about the additivity of the minimal output entropy from highly symmetric channels to a much larger class. A similar result holds for the maximal output pp-norm. Examples are given showing its use in a variety of situations. In particular, we prove the additivity and the multiplicativity for the shifted depolarising channel.Comment: 8 pages. This is the latest version of the first half of the original paper. The other half will appear in another pape

    Integrated whole transcriptome and DNA methylation analysis identifies gene networks specific to late-onset Alzheimer’s disease

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    Previous transcriptome studies observed disrupted cellular processes in late-onset Alzheimer\u27s disease (LOAD), yet it is unclear whether these changes are specific to LOAD, or are common to general neurodegeneration. In this study, we address this question by examining transcription in LOAD and comparing it to cognitively normal controls and a cohort of disease controls. Differential transcription was examined using RNA-seq, which allows for the examination of protein coding genes, non-coding RNAs, and splicing. Significant transcription differences specific to LOAD were observed in five genes: C10orf105, DIO2, a lincRNA, RARRES3, and WIF1. These findings were replicated in two independent publicly available microarray data sets. Network analyses, performed on 2,504 genes with moderate transcription differences in LOAD, reveal that these genes aggregate into seven networks. Two networks involved in myelination and innate immune response specifically correlated to LOAD. FRMD4B and ST18, hub genes within the myelination network, were previously implicated in LOAD. Of the five significant genes, WIF1 and RARRES3 are directly implicated in the myelination process; the other three genes are located within the network. LOAD specific changes in DNA methylation were located throughout the genome and substantial changes in methylation were identified within the myelination network. Splicing differences specific to LOAD were observed across the genome and were decreased in all seven networks. DNA methylation had reduced influence on transcription within LOAD in the myelination network when compared to both controls. These results hint at the molecular underpinnings of LOAD and indicate several key processes, genes, and networks specific to the disease

    Cholinergic stimulation by pyridostigmine bromide before myocardial infarction prevent cardiac and autonomic dysfunction

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    Inflammatory processes and cardiovascular autonomic imbalance are very relevant characteristic of the enormous dynamic process that is a myocardial infarction (MI). In this sense, some studies are investigating pharmacological therapies using acetylcholinesterase inhibitors, such as pyridostigmine bromide (PYR), aiming to increase parasympathetic tone after MI. Here we hypothesized that the use of PYR before the MI might bring an additional positive effect to the autonomic function, and consequently, in the inflammatory response and cardiac function. The present study aimed to evaluate left ventricular function, baroreflex sensitivity, autonomic modulation, and inflammatory profile in PYR- treated rats previously to MI. Methods: Male Wistar rats (250-300 g) were treated for 60 days with PYR. After treatment, they were submitted to the MI. After the MI, the autonomic and ventricular function were evaluated, as well as the systemic, left ventricle, and adipose tissue inflammatory profile. Results: PYR, performed before MI, prevented HR increase, systolic function impairment, baroreflex sensitivity drop, as well as pulse interval variance, RMSSD, blood pressure and parasympathetic modulation reduction in treated rats compared to untreated rats. Also, this positive functional changes may have been a result of the reduced inflammatory parameters in the left ventricle (IFN-alpha, IL-6, and IL-1 beta), as well as increased IL-10 expression and IL-10/TNF-alpha ratio in treated animals before MI. Conclusion: Prior treatment with PYR prevents impairment of the autonomic nervous system after MI, which may be associated with the attenuated expression of inflammatory factors and heart dysfunction9CONSELHO NACIONAL DE DESENVOLVIMENTO CIENTÍFICO E TECNOLÓGICO - CNPQFUNDAÇÃO DE AMPARO À PESQUISA DO ESTADO DE SÃO PAULO - FAPESPsem informação2013/14788-9; 2014/06669-

    Classical information deficit and monotonicity on local operations

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    We investigate classical information deficit: a candidate for measure of classical correlations emerging from thermodynamical approach initiated in [Phys. Rev. Lett 89, 180402]. It is defined as a difference between amount of information that can be concentrated by use of LOCC and the information contained in subsystems. We show nonintuitive fact, that one way version of this quantity can increase under local operation, hence it does not possess property required for a good measure of classical correlations. Recently it was shown by Igor Devetak, that regularised version of this quantity is monotonic under LO. In this context, our result implies that regularization plays a role of "monotoniser".Comment: 6 pages, revte
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