Support for Higher Education in the 1992-1993 Louisiana Legislature.

The public higher education system in Louisiana is facing a variety of problems. The Louisiana Legislature plays a vital role in the determination of policy regarding higher education in the state. Legislators have the responsibility of addressing current and emerging issues and finding solutions to problems. The purpose of the study was to determine the voting record of members of the 1992 Louisiana Legislature on issues facing higher education so as to infer legislative support for higher education. This purpose was accomplished by studying roll call votes on bills considered important to higher education in relation to personal characteristics of legislators. Data were collected on personal characteristics for all members of the 1992 legislature, including replacements. A panel of experts rated selected roll call bills voted on during the 1992-1993 regular and special sessions to determine the mean score for each bill. The mean score of each bill was applied to the appropriate yea or nay vote in determining a support for higher education score for each legislator. A model was developed for the senate that explained 33.1% of the variance in support for higher education. Seven variables in the stepwise regression model included legislative experience, business occupation, black race, number of bills introduced, education committee membership, legal occupation, and legislative committee leadership. A model developed for the house explained 21.4% of the variance in support for higher education. Five variables entered the stepwise regression model, including male gender, other occupation, democrat party affiliation, white race, legislative experience. The findings of the study indicated that there were more differences than similarities between the senate and the house research models in support for higher education. Legislative experience, race, and occupations, three variables common to both models, influence support for higher education differently in the two chambers. It is recommended that further study of the legislature include survey techniques to explore the influence of other factors contributing to the unexplained variance of legislative support for higher education

F.A.R.O.G. FORUM, Vol. 5 No. 7

https://digitalcommons.library.umaine.edu/francoamericain_forum/1019/thumbnail.jp

F.A.R.O.G. FORUM, Vol. 5 No. 2

https://digitalcommons.library.umaine.edu/francoamericain_forum/1060/thumbnail.jp

Current concept of abdominal sepsis : WSES position paper

Peer reviewe

Persistent circulation anomalies in observations and in a general circulation model

A comparative diagnostic study of upper-air persistent atmospheric events, as simulated by a general circulation model (GCM) and as observed, is presented. We start with an overview of the several theories that attempt to explain such phenomena. Particular emphasis is put on the model approach of Shutts (1983). We next show that the spatial distributions of persistent events is qualitatively similar in the GCM and observational data. The North-Atlantic events are extracted and a rotated empirical orthogonal function (REOF) analysis is done on the resulting data sets. The two REOF sets that are thus obtained are shown to greatly resemble one another. Both explain roughly 50% of their original data's variance. The relationships between the modes within a set are presented, so as to understand their probable combined evolution. The fourth chapter contains an evaluation of Shutt's theory. There, the third chapter's results are used to isolate a particular class of events, namely the strong +ATL2 dipoles. The time-tendencies associated to short time-scale synoptic waves are evaluated, using an E-vectors approach, taking care to distinguish between the onset, mature and demise phases of the events. It seems that these synoptic waves have a significant impact of the average life-cycle of this +ATL2 type of events, whether they be simulated by a GCM or obtained from a NMC set of analyses

LA FIBROSE SUPERFICIELLE RADIO-INDUITE (ETUDE DES MECANISMES D'ACTIVATION DU MYOFIBROBLASTE ET CARACTERISATION DE L'EPIDERME CICATRICIEL)

LA FIBROSE CUTANEE RADIO-INDUITE CONSTITUE UNE DES SEQUELLES FREQUENTES DES IRRADIATIONS THERAPEUTIQUES ET ACCIDENTELLES. ELLE SE CARACTERISE PAR UNE ACTIVATION DES FIBROBLASTES, UNE ACCUMULATION DE MATRICE EXTRA-CELLULAIRE ET UN INFILTRAT INFLAMMATOIRE PERSISTANT. CES PROCESSUS SONT ENTRETENUS PAR L'EXPRESSION ACCRUE DU TGF-BETAL. LE MYOFIBROBLASTE CONSTITUE LA CELLULE CLE DE CETTE PATHOLOGIE PAR SA CAPACITE DE SYNTHESE DE LA MATRICE EXTRA-CELLULAIRE. A PARTIR DE CELLULES ISSUES D'UN MODELE PORCIN DE FIBROSE RADIO-INDUITE, NOUS AVONS MONTRE QUE LES MYOFIBROBLASTES ECHAPPAIENT A L'EFFET ANTI-PROLIFERATIF DU TGF-BETAL, ET CE PROBABLEMENT A CAUSE D'UNE ALTERATION DE LA VOIE DE SIGNALISATION DU TGF-BETAL AU NIVEAU DE LA PROTEINE SMAD3 DONT LA TRANSLOCATION NUCLEAIRE EST DEFECTUEUSE. PAR AILLEURS, NOUS AVONS MONTRE, DANS UN MODELE DE PEAU RECONSTRUITE, QUE LES MYOFIBROBLASTES CONSTITUAIENT UNE CIBLE DIRECTE DE LA SOD ET QU'ILS REPONDAIENT A CET AGENT ANTI-FIBROSANT PAR UNE REVERSION DE LEUR PHENOTYPE. ENFIN, NOUS AVONS ELARGI L'ETUDE DE LA FIBROSE CUTANEE AU COMPARTIMENT EPIDERMIQUE JUSQU'ICI TRES PEU DECRIT. NOUS AVONS MONTRE QUE DES LESIONS HUMAINES DE FIBRO-NECROSE, INDUITES PAR UNE IRRADIATION THERAPEUTIQUE OU ACCIDENTELLE, PRESENTAIENT UNE HYPERPLASIE DE L'EPIDERME ASSOCIEE A UNE HYPERPROLIFERATION ET A UNE DIFFERENCIATION ANORMALE DES KERATINOCYTES DONT LES JONCTIONS COMMUNICANTES ETAIENT ALTEREES. DE PLUS, CES CELLULES PRESENTAIENT UNE ALTERATION DE LA VOIE DE SIGNALISATION DU TGF-1, DEFAUT QUI POURRAIT CONTRIBUER A LEUR PHENOTYPE HYPERPROLIFERATIF. EN CONCLUSION, NOUS AVONS PU PROGRESSER DANS LA COMPREHENSION DU ROLE DU MYOFIBROBLASTE LORS DU DEVELOPPEMENT ET DE LA REGRESSION DE LA FIBROSE. PAR AILLEURS, NOUS MONTRONS QU'UNE ALTERATION DE L'EPIDERME CONTIGUE AU DERME SE DEVELOPPE EN PARALLELE DU PROCESSUS DE FIBROSE, SUGGERANT UNE CONTRIBUTION ACTIVE DES KERATINOCYTES LORS DU DEVELOPPEMENT DE CETTE SEQUELLE RADIO-INDUITE.PARIS-BIUSJ-Thèses (751052125) / SudocCentre Technique Livre Ens. Sup. (774682301) / SudocPARIS-BIUSJ-Physique recherche (751052113) / SudocSudocFranceF