19 research outputs found
The extent of the psychological impairment of prosthodontic outpatients at a German University Hospital
An Evaluation of Sex Differences in Patients With Chronic Pain Undergoing an Interdisciplinary Pain Treatment Program
Characterizing developing adverse pressure gradient flows subject to surface roughness
An experimental study was conducted to
examine the effects of surface roughness and adverse
pressure gradient (APG) on the development of a turbulent
boundary layer. Hot-wire anemometry measurements were
carried out using single and X-wire probes in all regions of
a developing APG flow in an open return wind tunnel test
section. The same experimental conditions (i.e., T?, Uref,
and Cp) were maintained for smooth, k? = 0, and rough,
k? = 41–60, surfaces with Reynolds number based on
momentum thickness, 3,000\Reh\40,000. The experiment
was carefully designed such that the x-dependence in
the flow field was known. Despite this fact, only a very
small region of the boundary layer showed a balance of the
various terms in the integrated boundary layer equation.
The skin friction computed from this technique showed up
to a 58% increase due to the surface roughness. Various
equilibrium parameters were studied and the effect of
roughness was investigated. The generated flow was not in
equilibrium according to the Clauser (J Aero Sci 21:91–
108, 1954) definition due to its developing nature. After a
development region, the flow reached the equilibrium
condition as defined by Castillo and George (2001), where
K = const, is the pressure gradient parameter. Moreover, it
was found that this equilibrium condition can be used to
classify developing APG flows. Furthermore, the Zagarola
and Smits (J Fluid Mech 373:33–79, 1998a) scaling of the
mean velocity deficit, U?d*/d, can also be used as a criteria
to classify developing APG flows which supports the
equilibrium condition of Castillo and George (2001). With
this information a ‘full APG region’ was defined
Psychological Factors that Influence Decision-Making Regarding Trauma-Related Pain in Adolescents with Temporomandibular Disorder
Etiopathogenesis and pathophysiology of malaria
Malaria is a parasitic disease caused by Plasmodium protozoan parasites and transmitted by Anopheles mosquitoes. The disease is diffused in tropical areas, where it is associated with high morbidity and mortality. P. falciparum is the most dangerous species, mainly affecting young children. The parasite cycle occurs both in humans (asexual stages) and in mosquitoes (sexual stages). In humans, Plasmodium grows and multiplies within red blood cells using hemoglobin as essential source of nutrients and energy. However, this process generates toxic heme that the parasite aggregates into an insoluble inert biocrystal called hemozoin. This molecule sequesters in various organs (liver, spleen, and brain), potentially contributing to the development of malaria immunopathogenesis. Uncomplicated falciparum malaria clinical frame ranges from asymptomatic infection to classic symptoms such as fever, chills, sweating, headache, and muscle aches. However, malaria can also evolve into severe life-threatening complications, including cerebral malaria, severe anemia, respiratory distress, and acute renal failure