Scholarship of Teaching & Learning: Who, what, when, where, why, and how?

Published in Proceedings of the 39th Georgia Educational Research Association Conferenc

The Scholarship of Teaching & Learning: Who, What, When, Where, Why, and How?

This session will present a panel discussion of the Scholarship of Teaching & Learning [SoTL]. Panelists include: scholars new to SoTL and those with over a decade’s experience with it, the editor of a pre-eminent international SoTL journal, the chair of a pre-eminent international SoTL conference, and a University System of Georgia SoTL Award winner. The panel will discuss what SoTL is (and is not), the value of SoTL, how to get started with SoTL research projects, how to contextualize and theoretically ground SoTL research, how SoTL both informs and dovetails with teaching, and will provide examples of SoTL projects at both the undergraduate and graduate level

The Scholarship of Teaching & Learning: Who, what, when, where, why, and how?

Panel symposium presented at the 2014 Georgia Educational Research Association Conference, Savannah, GA. This session will present a panel discussion of the Scholarship of Teaching & Learning [SoTL]. Panelists include: scholars new to SoTL and those with over a decade’s experience with it, the editor of a pre-eminent international SoTL journal, the chair of a pre-eminent international SoTL conference, and a University System of Georgia SoTL Award winner. The panel will discuss what SoTL is (and is not), the value of SoTL, how to get started with SoTL research projects, how to contextualize and theoretically ground SoTL research, how SoTL both informs and dovetails with teaching, and will provide examples of SoTL projects at both the undergraduate and graduate level

Incorporation of a Dietary Omega 3 Fatty Acid Impairs Murine Macrophage Responses to Mycobacterium tuberculosis

by creating an immunosuppressive environment. We hypothesized that incorporation of n-3 PUFA suppresses activation of macrophage antimycobacterial responses and favors bacterial growth, in part, by modulating the IFNγ-mediated signaling pathway.. The fatty acid composition of macrophage membranes was modified significantly by DHA treatment. DHA-treated macrophages were less effective in controlling intracellular mycobacteria and showed impaired oxidative metabolism and reduced phagolysosome maturation. Incorporation of DHA resulted in defective macrophage activation, as characterized by reduced production of pro-inflammatory cytokines (TNFα, IL-6 and MCP-1), and lower expression of co-stimulatory molecules (CD40 and CD86). DHA treatment impaired STAT1 phosphorylation and colocalization of the IFNγ receptor with lipid rafts, without affecting surface expression of IFNγ receptor. in response to activation by IFNγ, by modulation of IFNγ receptor signaling and function, suggesting that n-3 PUFA-enriched diets may have a detrimental effect on host immunity to tuberculosis