Pion, kaon, proton and anti-proton transverse momentum distributions from p+p and d+Au collisions at sNN=200\sqrt{s_{NN}} = 200 GeV

Identified mid-rapidity particle spectra of $\pi^{\pm}$, $K^{\pm}$, and $p(\bar{p})$ from 200 GeV p+p and d+Au collisions are reported. A time-of-flight detector based on multi-gap resistive plate chamber technology is used for particle identification. The particle-species dependence of the Cronin effect is observed to be significantly smaller than that at lower energies. The ratio of the nuclear modification factor ($R_{dAu}$) between protons $(p+\bar{p})$ and charged hadrons ($h$) in the transverse momentum range $1.2<{p_{T}}<3.0$ GeV/c is measured to be $1.19\pm0.05$(stat)$\pm0.03$(syst) in minimum-bias collisions and shows little centrality dependence. The yield ratio of $(p+\bar{p})/h$ in minimum-bias d+Au collisions is found to be a factor of 2 lower than that in Au+Au collisions, indicating that the Cronin effect alone is not enough to account for the relative baryon enhancement observed in heavy ion collisions at RHIC.Comment: 6 pages, 4 figures, 1 table. We extended the pion spectra from transverse momentum 1.8 GeV/c to 3. GeV/

Demonstration of the temporal matter-wave Talbot effect for trapped matter waves

We demonstrate the temporal Talbot effect for trapped matter waves using ultracold atoms in an optical lattice. We investigate the phase evolution of an array of essentially non-interacting matter waves and observe matter-wave collapse and revival in the form of a Talbot interference pattern. By using long expansion times, we image momentum space with sub-recoil resolution, allowing us to observe fractional Talbot fringes up to 10th order.Comment: 17 pages, 7 figure

Kaon Production and Kaon to Pion Ratio in Au+Au Collisions at \snn=130 GeV

Mid-rapidity transverse mass spectra and multiplicity densities of charged and neutral kaons are reported for Au+Au collisions at \snn=130 GeV at RHIC. The spectra are exponential in transverse mass, with an inverse slope of about 280 MeV in central collisions. The multiplicity densities for these particles scale with the negative hadron pseudo-rapidity density. The charged kaon to pion ratios are $K^+/\pi^- = 0.161 \pm 0.002 {\rm (stat)} \pm 0.024 {\rm (syst)}$ and $K^-/\pi^- = 0.146 \pm 0.002 {\rm (stat)} \pm 0.022 {\rm (syst)}$ for the most central collisions. The $K^+/\pi^-$ ratio is lower than the same ratio observed at the SPS while the $K^-/\pi^-$ is higher than the SPS result. Both ratios are enhanced by about 50% relative to p+p and $\bar{\rm p}$+p collision data at similar energies.Comment: 6 pages, 3 figures, 1 tabl

Azimuthal anisotropy at RHIC: the first and fourth harmonics

We report the first observations of the first harmonic (directed flow, v_1), and the fourth harmonic (v_4), in the azimuthal distribution of particles with respect to the reaction plane in Au+Au collisions at the Relativistic Heavy Ion Collider (RHIC). Both measurements were done taking advantage of the large elliptic flow (v_2) generated at RHIC. From the correlation of v_2 with v_1 it is determined that v_2 is positive, or {\it in-plane}. The integrated v_4 is about a factor of 10 smaller than v_2. For the sixth (v_6) and eighth (v_8) harmonics upper limits on the magnitudes are reported.Comment: 6 pages with 3 figures, as accepted for Phys. Rev. Letters The data tables are at http://www.star.bnl.gov/central/publications/pubDetail.php?id=3

Mid-rapidity anti-proton to proton ratio from Au+Au collisions at sNN=130 \sqrt{s_{NN}} = 130 GeV

We report results on the ratio of mid-rapidity anti-proton to proton yields in Au+Au collisions at \rts = 130 GeV per nucleon pair as measured by the STAR experiment at RHIC. Within the rapidity and transverse momentum range of $|y|<0.5$ and 0.4 $<p_t<$ 1.0 GeV/$c$, the ratio is essentially independent of either transverse momentum or rapidity, with an average of $0.65\pm 0.01_{\rm (stat.)} \pm 0.07_{\rm (syst.)}$ for minimum bias collisions. Within errors, no strong centrality dependence is observed. The results indicate that at this RHIC energy, although the $p$-\pb pair production becomes important at mid-rapidity, a significant excess of baryons over anti-baryons is still present.Comment: 5 pages, 3 figures, accepted by Phys. Rev. Let

Strange anti-particle to particle ratios at mid-rapidity in sqrt(s_NN)= 130 GeV Au+Au Collisions

Values of the ratios in the mid-rapidity yields of anti-Lambda/Lambda = 0.71 +/- 0.01(stat.) +/- 0.04(sys.), anti-Xi+/Xi- = 0.83 +/- 0.04(stat.) +/- 0.05 (sys.), anti-Omega+/Omega- = 0.95 +/- 0.15(stat) +/- 0.05(sys.) and K+/K- 1.092 +/- 0.023(combined) were obtained in central sqrt(s_NN) = 130 GeV Au+Au collisions using the STAR detector. The ratios indicate that a fraction of the net-baryon number from the initial system is present in the excess of hyperons over anti-hyperons at mid-rapidity. The trend in the progression of the baryon ratios, with increasing strange quark content, is similar to that observed in heavy-ion collisions at lower energies. The value of these ratios may be related to the charged kaon ratio in the framework of simple quark-counting and thermal models.Comment: 6 pages, 3 figures, revtex4, now accepted by Physics Letters B. All figures improved for clarity, fig. 2 now has kaon ratio separated by technique, fig. 3 now has additional other RHIC data points. Minor clarifications in text in response to referee comments. Updated ref

Event-wise ⟨p\u3csub\u3et\u3c/sub\u3e⟩ fluctuations in Au-Au collisions at √s\u3csub\u3eNN\u3c/sub\u3e = 130 GeV

We present the first large-acceptance measurement of event-wise mean transverse momentum ⟨pt⟩ fluctuations for Au-Au collisions at nucleon-nucleon center-of-momentum collision energy √sNN = 130 GeV. The observed nonstatistical ⟨pt⟩ fluctuations substantially exceed in magnitude fluctuations expected from the finite number of particles produced in a typical collision. The r.m.s. fractional width excess of the event-wise ⟨pt⟩ distribution is 13.7±0.1(stat) ±1.3(syst)% relative to a statistical reference, for the 15% most-central collisions and for charged hadrons within pseudorapidity range |η|c. The width excess varies smoothly but nonmonotonically with collision centrality and does not display rapid changes with centrality which might indicate the presence of critical fluctuations. The reported ⟨pt⟩ fluctuation excess is qualitatively larger than those observed at lower energies and differs markedly from theoretical expectations. Contributions to ⟨pt⟩ fluctuations from semihard parton scattering in the initial state and dissipation in the bulk colored medium are discussed

Event-wise ⟨p\u3csub\u3et\u3c/sub\u3e⟩ fluctuations in Au-Au collisions at √s\u3csub\u3eNN\u3c/sub\u3e = 130 GeV

We present the first large-acceptance measurement of event-wise mean transverse momentum ⟨pt⟩ fluctuations for Au-Au collisions at nucleon-nucleon center-of-momentum collision energy √sNN = 130 GeV. The observed nonstatistical ⟨pt⟩ fluctuations substantially exceed in magnitude fluctuations expected from the finite number of particles produced in a typical collision. The r.m.s. fractional width excess of the event-wise ⟨pt⟩ distribution is 13.7±0.1(stat) ±1.3(syst)% relative to a statistical reference, for the 15% most-central collisions and for charged hadrons within pseudorapidity range |η|c. The width excess varies smoothly but nonmonotonically with collision centrality and does not display rapid changes with centrality which might indicate the presence of critical fluctuations. The reported ⟨pt⟩ fluctuation excess is qualitatively larger than those observed at lower energies and differs markedly from theoretical expectations. Contributions to ⟨pt⟩ fluctuations from semihard parton scattering in the initial state and dissipation in the bulk colored medium are discussed

СИСТЕМНЫЙ ЛИПИДНЫЙ ДИСТРЕСС-СИНДРОМ ПРИ ТРАВМАТИЧЕСКОЙ БОЛЕЗНИ

The purpose of work was studying of a role of systemic lipid metabolism disturbances in a pathogenesis of homeostasis disorders in the early posttraumatic period at fractures of pelvic bones in experiment and clinic. Pilot and clinical studies are put in a basis of work with application of the techniques estimating structure phospholipid membranes of erythrocytes, cells of a liver, kidneys, heart, lungs, and also intensity of the processes participating in a regulation of a lipid metabolism. Results. It is established that at a pelvic trauma change of lipids structure arise not only in a blood, but also other tissues structures (a liver, kidneys, lungs, heart) that is caused by activization of phosfolipaze systems, an intensification of lipids peroxidation process. There is a systemic lipid distress syndrome. The arisen changes - one of significant in a pathogenesis of traumatic illness. At modification of lipid structure of a biomembrane the morphological and functional condition of a cell starts suffering. It is substrate of emergence of disregulation pathologies. The received scientific facts can be the base for intelligent medical actions for the purpose of the prevention of emergence of a "favorable" background of disregulation conditions by vectorial impact on one of its main pathogenetic links - a systemic lipide distress syndrome.Цель работы - изучение роли системных нарушений липидного метаболизма в патогенезе расстройств гомеостаза у пациентов с переломами таза. В основу работы положены экспериментальные и клинические исследования с применением методик, позволяющих оценить состав фосфолипидного бислоя мембран эритроцитов, клеток печени, почек, сердца, легких, а также интенсивность процессов, участвующих в регуляции липидного обмена. Результаты. Установлено, что при травме таза изменения состава липидов возникают не только в крови, но и других тканевых структурах (печени, почек, легких, сердце), что обусловлено активизацией фосфолипазных систем, интенсификацией процесса перекисного окисления липидов. Возникает системный липидный дистресс-синдром. Возникшие изменения - одни из значимых в патогенезе травматической болезни. Об этом в первую очередь свидетельствует то, что при модификации липидного состава биомембраны начинает страдать морфофункциональное состояние клетки - субстрат возникновения дизрегуляционных патологий. Полученные научные факты могут явиться фундаментом для осмысленных врачебных действий с целью предупреждения возникновения «благоприятного» фона дизрегуляционных состояний путем векторного воздействия на одно из основных его патогенетических звеньев - системный липидный дистресс-синдром