Counseling the Media: Bridging Pop Cultural Borders with the Mental Health Profession

Professional counseling is often defined as a professional relationship meant to empower diverse people towards their mental health and wellness goals (Kaplan, Tarvydas, & Gladding, 2014). While counselors continue to provide professional services in many settings, counseling is not always portrayed in a positive light within television and film. Shows such as 13 Reasons Why and How I Met Your Mother both portray an inaccurate perception of counseling characters who disregard their ethical duties despite the American Counseling Association’s Code of Ethics (2014) mandates for safe-guarding the wellbeing of those seeking counseling, honoring multicultural diversity, and advocating for social justice. This frequent portrayal of unethical behaviors conflates mental health stigma with needed healthcare services for marginalized populations. Pop culture thus serves as a border, separating fiction from reality and superimposing a socially constructed barrier between optimal health and exaggerated entertainment. To correct these concerns, professional counseling can bridge gaps created by these borders by respecting pop cultural influences and disallowing societal oppression (Tarvydas, Vazquez‐Ramos, & Estrada‐Hernandez, 2015). Integration of literary and enacted characters in diverse media can serve as metaphors to enhance the therapeutic experience for clients, overcome life challenges, and promote optimal health goals (Rubin, 2008; Schmidt, 2009). Therefore, this experiential presentation will provide audience members a unique opportunity to critically analyze media portrayals of counseling services and discuss the impact on mental health stigma. Attendees will learn how pop culture can be ethically integrated within a counseling setting to respect each individual’s needs and diversity without promoting stigma

Solving spin quantum-master equations with matrix continued-fraction methods: application to superparamagnets

We implement continued-fraction techniques to solve exactly quantum master equations for a spin with arbitrary S coupled to a (bosonic) thermal bath. The full spin density matrix is obtained, so that along with relaxation and thermoactivation, coherent dynamics is included (precession, tunnel, etc.). The method is applied to study isotropic spins and spins in a bistable anisotropy potential (superparamagnets). We present examples of static response, the dynamical susceptibility including the contribution of the different relaxation modes, and of spin resonance in transverse fields.Comment: Resubmitted to J. Phys. A: Math. Gen. Some rewriting here and there. Discussion on positivity in App.D3 at request of one refere

Progress along developmental tracks for electronic health records implementation in the United States

The development and implementation of electronic health records (EHR) have occurred slowly in the United States. To date, these approaches have, for the most part, followed four developmental tracks: (a) Enhancement of immunization registries and linkage with other health records to produce Child Health Profiles (CHP), (b) Regional Health Information Organization (RHIO) demonstration projects to link together patient medical records, (c) Insurance company projects linked to ICD-9 codes and patient records for cost-benefit assessments, and (d) Consortia of EHR developers collaborating to model systems requirements and standards for data linkage. Until recently, these separate efforts have been conducted in the very silos that they had intended to eliminate, and there is still considerable debate concerning health professionals access to as well as commitment to using EHR if these systems are provided. This paper will describe these four developmental tracks, patient rights and the legal environment for EHR, international comparisons, and future projections for EHR expansion across health networks in the United States

Information-based TMS to mid-lateral prefrontal cortex disrupts action goals during emotional processing.

The ability to respond to emotional events in a context-sensitive and goal-oriented manner is essential for adaptive functioning. In models of behavioral and emotion regulation, the lateral prefrontal cortex (LPFC) is postulated to maintain goal-relevant representations that promote cognitive control, an idea rarely tested with causal inference. Here, we altered mid-LPFC function in healthy individuals using a putatively inhibitory brain stimulation protocol (continuous theta burst; cTBS), followed by fMRI scanning. Participants performed the Affective Go/No-Go task, which requires goal-oriented action during affective processing. We targeted mid-LPFC (vs. a Control site) based on the individualized location of action-goal representations observed during the task. cTBS to mid-LPFC reduced action-goal representations in mid-LPFC and impaired goal-oriented action, particularly during processing of negative emotional cues. During negative-cue processing, cTBS to mid-LPFC reduced functional coupling between mid-LPFC and nodes of the default mode network, including frontopolar cortex-a region thought to modulate LPFC control signals according to internal states. Collectively, these results indicate that mid-LPFC goal-relevant representations play a causal role in governing context-sensitive cognitive control during emotional processing

SEA CAR STRUCTURAL AND ACOUSTIC MODELLING

paper #38

Escape from X inactivation of two new genes associated with DXS6974E and DXS7020E

Most genes on the X chromosome undergo ''inactivation,'' being transcribed from only one copy in female somatic cells, but several human genes have been shown to be expressed from both the active and the otherwise inactivated homologue. To assess further the fraction and location of genes that escape inactivation, we have analyzed the inactivation status of a set of 73 expressed sequence tags that were derived from the sequencing of cDNA collections and mapped to the X chromosome. Of 33 that were expressed in cultured cells, as assessed by reverse transcription and PCR, 4 (about 12%) were transcribed from both the active and the inactive X chromosome, Two, RPS4 and PCTAIRE1, are already known to escape inactivation; the other 2, of unknown function, include a short cDNA with a full open reading frame and a transcript with no detectable open reading frame. They map, respectively, to Xp11.3-p11.4 and Xp22.2; both regions were previously reported to encode sequences transcribed from the inactive X. Neither transcript has a corresponding sequence on the Y. Thus, they exhibit double dosage in females compared to males, and inactivation status may be inconsequential for these transcribed sequences. (C) 1997 Academic Press

Expressed STSs and transcription of human Xq28

STSs, which have been used to build and format clone contigs, have been used here to assemble a transcriptional map across a cytogenetic band. Of fifty one STSs in Xq28, 20 were positive by RT-PCR. Thus, an additional 20 possible ESTs were detected among the STSs, and seven of these also identified cDNAs in at least one library. The transcripts confirm the high expression level of this region, correlated with its GC compositional map and CpG island content

DNMT3B mutations in ICF syndrome are associated to epigenetic alterations of microRNAs having targets involved in immune function.

Immunodeficiency, centromeric region instability, cacial anomalies (IcF; OMIM #242860) syndrome, due to mutations in the DNMT3B gene, is characterized by inheritance of aberrant patterns of DNa methylation and heterochromatin defects. patients show variable agammaglobulinemia and a reduced number of T cells, making them prone to infections and death before adulthood. Other variable symptoms include facial dysmorphism, growth and mental retardation. Despite the recent advances in identifying the dysregulated genes, the molecular mechanisms, which underlie the altered gene expression causing IcF phenotype complexity, are not well understood. held the recently-shown tight correlation between epigenetics and microRNas (miRNas), we searched for miRNas regulated by DNMT3B activity, comparing cell lines from IcF patients with those from healthy individuals. We observe that 89 miRNas, some of which involved in immune function, development and neurogenesis, are dysregulated in IcF (LcLs) compared to wild-type cells. significant DNa hypomethylation of miRNa cpG islands was not observed in cases of miRNa upregulation in IcF cells, suggesting a more subtle effect of DNMT3B deficiency on their regulation; however, a modification of histone marks, especially h3K27 and h3K4 trimethylation, and h4 acetylation, was observed concomitantly with changes in microRNa expression. Functional correlation between miRNa and mRNa expression of their targets allow us to suppose a regulation either at mRNa level or at protein level. These results provide a better understanding of how DNa methylation and histone code interact to regulate the class of microRNa genes and enable us to predict molecular events possibly contributing to IcF condition