The failure tolerance of mechatronic software systems to random and targeted attacks

This paper describes a complex networks approach to study the failure tolerance of mechatronic software systems under various types of hardware and/or software failures. We produce synthetic system architectures based on evidence of modular and hierarchical modular product architectures and known motifs for the interconnection of physical components to software. The system architectures are then subject to various forms of attack. The attacks simulate failure of critical hardware or software. Four types of attack are investigated: degree centrality, betweenness centrality, closeness centrality and random attack. Failure tolerance of the system is measured by a 'robustness coefficient', a topological 'size' metric of the connectedness of the attacked network. We find that the betweenness centrality attack results in the most significant reduction in the robustness coefficient, confirming betweenness centrality, rather than the number of connections (i.e. degree), as the most conservative metric of component importance. A counter-intuitive finding is that "designed" system architectures, including a bus, ring, and star architecture, are not significantly more failure-tolerant than interconnections with no prescribed architecture, that is, a random architecture. Our research provides a data-driven approach to engineer the architecture of mechatronic software systems for failure tolerance.Comment: Proceedings of the 2013 ASME International Design Engineering Technical Conferences & Computers and Information in Engineering Conference IDETC/CIE 2013 August 4-7, 2013, Portland, Oregon, USA (In Print

On the influence of topological characteristics on robustness of complex networks

In this paper, we explore the relationship between the topological characteristics of a complex network and its robustness to sustained targeted attacks. Using synthesised scale-free, small-world and random networks, we look at a number of network measures, including assortativity, modularity, average path length, clustering coefficient, rich club profiles and scale-free exponent (where applicable) of a network, and how each of these influence the robustness of a network under targeted attacks. We use an established robustness coefficient to measure topological robustness, and consider sustained targeted attacks by order of node degree. With respect to scale-free networks, we show that assortativity, modularity and average path length have a positive correlation with network robustness, whereas clustering coefficient has a negative correlation. We did not find any correlation between scale-free exponent and robustness, or rich-club profiles and robustness. The robustness of small-world networks on the other hand, show substantial positive correlations with assortativity, modularity, clustering coefficient and average path length. In comparison, the robustness of Erdos-Renyi random networks did not have any significant correlation with any of the network properties considered. A significant observation is that high clustering decreases topological robustness in scale-free networks, yet it increases topological robustness in small-world networks. Our results highlight the importance of topological characteristics in influencing network robustness, and illustrate design strategies network designers can use to increase the robustness of scale-free and small-world networks under sustained targeted attacks

Resolving structural variability in network models and the brain

Large-scale white matter pathways crisscrossing the cortex create a complex pattern of connectivity that underlies human cognitive function. Generative mechanisms for this architecture have been difficult to identify in part because little is known about mechanistic drivers of structured networks. Here we contrast network properties derived from diffusion spectrum imaging data of the human brain with 13 synthetic network models chosen to probe the roles of physical network embedding and temporal network growth. We characterize both the empirical and synthetic networks using familiar diagnostics presented in statistical form, as scatter plots and distributions, to reveal the full range of variability of each measure across scales in the network. We focus on the degree distribution, degree assortativity, hierarchy, topological Rentian scaling, and topological fractal scaling---in addition to several summary statistics, including the mean clustering coefficient, shortest path length, and network diameter. The models are investigated in a progressive, branching sequence, aimed at capturing different elements thought to be important in the brain, and range from simple random and regular networks, to models that incorporate specific growth rules and constraints. We find that synthetic models that constrain the network nodes to be embedded in anatomical brain regions tend to produce distributions that are similar to those extracted from the brain. We also find that network models hardcoded to display one network property do not in general also display a second, suggesting that multiple neurobiological mechanisms might be at play in the development of human brain network architecture. Together, the network models that we develop and employ provide a potentially useful starting point for the statistical inference of brain network structure from neuroimaging data.Comment: 24 pages, 11 figures, 1 table, supplementary material

Optimization methods for topological design of interconnected ring networks

Thesis (M.S.)--Massachusetts Institute of Technology, Dept. of Electrical Engineering and Computer Science, 1994.Includes bibliographical references (leaves 177-179).by Valery Brodsky.M.S