372,968 research outputs found
A week with Professor Giacomo Rizzolatti (30th of November - 6th of December 2015)
Professor Giacomo Rizzolatti, Professor Emeritus in
physiology at the University of Parma, is the discoverer
of `mirror neurons' and director of the `Social and Mo-
tor Cognition' centre at the Italian Institute of Technology. From January 2016 he has been appointed by the
Rector Professor Jaunito Camilleri as Senior Researcher
Advisor by the University of Malta within the Department of Physiology and Biochemistry.
Award winner and world-renowned neuroscientist,
Professor Giacomo Rizzolatti received the `Brain Prize'
in 2014, a prestigious prize for brain research bestowed
by the Grete Lundbeck European Brain Research Prize
Foundation in Copenhagen.
Professor Giacomo Rizzolatti was invited by Professor
Giuseppe Di Giovanni on behalf of the Malta Neuroscience Network Program of the University of Malta
as key-note speaker at the IX Malta Medical School
Conference (MMSC), the most important event for the
local medical community, which this year was held in
St. Julian at the Hilton Hotel from the 3rd to the 5th of
December. The conference, for the second time, had different sessions in Neuroscience and for the first time a
commune session in neurology-neuroscience.peer-reviewe
Two-photon imaging of cell-specific fluorophores in transgenic mice – an exploratory tool to study mechanisms of white matter injury
Relatively little is known about specific pathways leading to structural and functional disruption of axons and glial cells in white matter. Because focal cerebral ischemia in humans damages both gray and white matter, an understanding of white matter injury is important in devising potential therapeutic approaches. We have developed a novel brain slice model from transgenic mice under control of cell-specific promoters to understand interactions between oligodendrocytes and axons under high resolution twophoton microscopy. Our data extends over previous findings the vulnerability of oligodendrocytes and axons both in culture and in slice preparations to glutamate toxicity during stroke and hypoglycemia. Conditions as different as stroke, trauma, perinatal brain injury, and multiple sclerosis may share common mechanisms of white matter injury.N/
Iatros 1984
Iatros is the yearbook of the School of Medicine at the University of Massachusetts Medical School in Worcester, MA. The name of the yearbook comes from the Greek word for healer or physician . This is the yearbook for the Class of 1984.https://escholarship.umassmed.edu/yearbooks/1001/thumbnail.jp
Iatros 1974-1978
Iatros is the yearbook of the School of Medicine at the University of Massachusetts Medical School in Worcester, MA. The name of the yearbook comes from the Greek word for healer or physician . This is the inaugural yearbook, covering the classes of 1974-1975, 1975-1976, 1976-1977, and 1977-1978.https://escholarship.umassmed.edu/yearbooks/1000/thumbnail.jp
Medical School of Maine at Bowdoin College: Commencing February 8th, 1883
Information about the School of Medicine of Maine when at Bowdoin College
Our medical school : quo vadis?
The Medical School of the University of Malta is truly an institution with a unique historical background and mission, as has been shown time and again since its foundation in 1676, when the School of Medicine was referred to as the School of Anatomy and Surgery. Over subsequent centuries, it grew and evolved to meet the changing needs of a population often undergoing rapid social change and occasionally beleagured by wars. The Medical School responded well to the fresh challenges brought by the emergence of certain medical problems and against a background initially of colonialism and subsequently independence.peer-reviewe
The Malta Medical School Conference
The next Malta Medical School Conference, the ninth one in the series, is being held towards the end of 2015.peer-reviewe
L-lactate reduces ischemic white matter injury and modulates HCA1 oligodendrocyte expression in an in vivo mouse model of focal ischemia
L-lactate is a metabolite that is oxidized preferentially to glucose under conditions of high metabolic stress. The discovery and localization of the lactate receptor HCA1 in various brain regions suggests that lactate is additionally an important signaling molecule in the brain. Lactate is neuroprotective in various ischemia paradigms, reduces axonal injury in vitro and is avidly utilized by oligodendrocytes (OLs). The protective potential of L-lactate to reduce white matter (WM) injury in a mouse stroke model was investigated.N/
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