The Spread of Opinions and Proportional Voting

Election results are determined by numerous social factors that affect the formation of opinion of the voters, including the network of interactions between them and the dynamics of opinion influence. In this work we study the result of proportional elections using an opinion dynamics model similar to simple opinion spreading over a complex network. Erdos-Renyi, Barabasi-Albert, regular lattices and randomly augmented lattices are considered as models of the underlying social networks. The model reproduces the power law behavior of number of candidates with a given number of votes found in real elections with the correct slope, a cutoff for larger number of votes and a plateau for small number of votes. It is found that the small world property of the underlying network is fundamental for the emergence of the power law regime.Comment: 10 pages, 7 figure

Social contagions on interdependent lattice networks

Although an increasing amount of research is being done on the dynamical processes on interdependent spatial networks, knowledge of how interdependent spatial networks influence the dynamics of social contagion in them is sparse. Here we present a novel non-Markovian social contagion model on interdependent spatial networks composed of two identical two-dimensional lattices. We compare the dynamics of social contagion on networks with different fractions of dependency links and find that the density of final recovered nodes increases as the number of dependency links is increased. We use a finite-size analysis method to identify the type of phase transition in the giant connected components (GCC) of the final adopted nodes and find that as we increase the fraction of dependency links, the phase transition switches from second-order to first-order. In strong interdependent spatial networks with abundant dependency links, increasing the fraction of initial adopted nodes can induce the switch from a first-order to second-order phase transition associated with social contagion dynamics. In networks with a small number of dependency links, the phase transition remains second-order. In addition, both the second-order and first-order phase transition points can be decreased by increasing the fraction of dependency links or the number of initially-adopted nodes.This work was partially supported by National Natural Science Foundation of China (Grant Nos 61501358, 61673085), and the Fundamental Research Funds for the Central Universities. (61501358 - National Natural Science Foundation of China; 61673085 - National Natural Science Foundation of China; Fundamental Research Funds for the Central Universities)Published versio

Evolutionary game theory: Temporal and spatial effects beyond replicator dynamics

Evolutionary game dynamics is one of the most fruitful frameworks for studying evolution in different disciplines, from Biology to Economics. Within this context, the approach of choice for many researchers is the so-called replicator equation, that describes mathematically the idea that those individuals performing better have more offspring and thus their frequency in the population grows. While very many interesting results have been obtained with this equation in the three decades elapsed since it was first proposed, it is important to realize the limits of its applicability. One particularly relevant issue in this respect is that of non-mean-field effects, that may arise from temporal fluctuations or from spatial correlations, both neglected in the replicator equation. This review discusses these temporal and spatial effects focusing on the non-trivial modifications they induce when compared to the outcome of replicator dynamics. Alongside this question, the hypothesis of linearity and its relation to the choice of the rule for strategy update is also analyzed. The discussion is presented in terms of the emergence of cooperation, as one of the current key problems in Biology and in other disciplines.Comment: Review, 48 pages, 26 figure

The Effect of Disease-induced Mortality on Structural Network Properties

As the understanding of the importance of social contact networks in the spread of infectious diseases has increased, so has the interest in understanding the feedback process of the disease altering the social network. While many studies have explored the influence of individual epidemiological parameters and/or underlying network topologies on the resulting disease dynamics, we here provide a systematic overview of the interactions between these two influences on population-level disease outcomes. We show that the sensitivity of the population-level disease outcomes to the combination of epidemiological parameters that describe the disease are critically dependent on the topological structure of the population's contact network. We introduce a new metric for assessing disease-driven structural damage to a network as a population-level outcome. Lastly, we discuss how the expected individual-level disease burden is influenced by the complete suite of epidemiological characteristics for the circulating disease and the ongoing process of network compromise. Our results have broad implications for prediction and mitigation of outbreaks in both natural and human populations.Comment: 23 pages, 6 figure

Binding Social and Cultural Networks: A Model

Until now, most studies carried onto social or semantic networks have considered each of these networks independently. Our goal here is to bring a formal frame for studying both networks empirically as well as to point out stylized facts that would explain their reciprocal influence and the emergence of clusters of agents, which may also be regarded as ''cultural cliques''. We show how to apply the Galois lattice theory to the modeling of the coevolution of social and conceptual networks, and the characterization of cultural communities. Basing our approach on Barabasi-Albert's models, we however extend the usual preferential attachment probability in order to take into account the reciprocal influence of both networks, therefore introducing the notion of dual distance. In addition to providing a theoretic frame we draw here a program of empirical tests which should give root to a more analytical model and the consequent simulation and validation. In a broader view, adopting and actually implementing the paradigm of cultural epidemiology, we could proceed further with the study of knowledge diffusion and explain how the social network structure affects concept propagation and in return how concept propagation affects the social network.Comment: 8 pages, 3 figures (v2: typos, minor corrections in section 3.2) (v3: examples, figures added

Evolutionary Games on Networks and Payoff Invariance Under Replicator Dynamics

The commonly used accumulated payoff scheme is not invariant with respect to shifts of payoff values when applied locally in degree-inhomogeneous population structures. We propose a suitably modified payoff scheme and we show both formally and by numerical simulation, that it leaves the replicator dynamics invariant with respect to affine transformations of the game payoff matrix. We then show empirically that, using the modified payoff scheme, an interesting amount of cooperation can be reached in three paradigmatic non-cooperative two-person games in populations that are structured according to graphs that have a marked degree inhomogeneity, similar to actual graphs found in society. The three games are the Prisoner's Dilemma, the Hawks-Doves and the Stag-Hunt. This confirms previous important observations that, under certain conditions, cooperation may emerge in such network-structured populations, even though standard replicator dynamics for mixing populations prescribes equilibria in which cooperation is totally absent in the Prisoner's Dilemma, and it is less widespread in the other two games.Comment: 20 pages, 8 figures; to appear on BioSystem

Mass media destabilizes the cultural homogeneous regime in Axelrod's model

An important feature of Axelrod's model for culture dissemination or social influence is the emergence of many multicultural absorbing states, despite the fact that the local rules that specify the agents interactions are explicitly designed to decrease the cultural differences between agents. Here we re-examine the problem of introducing an external, global interaction -- the mass media -- in the rules of Axelrod's model: in addition to their nearest-neighbors, each agent has a certain probability $p$ to interact with a virtual neighbor whose cultural features are fixed from the outset. Most surprisingly, this apparently homogenizing effect actually increases the cultural diversity of the population. We show that, contrary to previous claims in the literature, even a vanishingly small value of $p$ is sufficient to destabilize the homogeneous regime for very large lattice sizes

Nonequilibrium transitions in complex networks: a model of social interaction

We analyze the non-equilibrium order-disorder transition of Axelrod's model of social interaction in several complex networks. In a small world network, we find a transition between an ordered homogeneous state and a disordered state. The transition point is shifted by the degree of spatial disorder of the underlying network, the network disorder favoring ordered configurations. In random scale-free networks the transition is only observed for finite size systems, showing system size scaling, while in the thermodynamic limit only ordered configurations are always obtained. Thus in the thermodynamic limit the transition disappears. However, in structured scale-free networks, the phase transition between an ordered and a disordered phase is restored.Comment: 7 pages revtex4, 10 figures, related material at http://www.imedea.uib.es/PhysDept/Nonlinear/research_topics/Social