Eq upregulates protein expression of adhesion molecules in HUVECs.
<p>HUVECs were treated with various estrogens as described in <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0211462#pone.0211462.g001" target="_blank">Fig 1</a>. (<b>A</b>) E-selectin, (<b>B</b>) ICAM-1, (<b>C</b>) P-selectin, and (<b>D</b>) VCAM-1 protein levels were quantified by EIA. Data are expressed as the mean ± SEM of four experiments. *<i>P</i> < 0.05 vs. vehicle alone.</p
Biochemistry, Cell Biology, Genetics, Molecular Biology, Physiology, Immunology, Developmental Biology, Cancer, Biological Sciences not elsewhere classified, Chemical Sciences not elsewhere classified, intercellular adhesion molecule, equilin treatment, NF -κB activation, adhesion molecules E-selectin, adhesion molecules, protein expression levels, ER, atherosclerosi, equine estrogen increases monocyte-endothelial adhesion, E 2 treatment, estrogen-replacement therapy, polymerase chain reaction, HUVEC, Conjugated equine estrogen, NF -κB subunit p 65, U 937 monocytoid cells, E 2, NF -κB proteins, Equilin
DOI identifier: 10.1371/journal.pone.0211462.g002
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