11 research outputs found
The LysR-type transcriptional regulator CbbR controlling autotrophic CO2 fixation by Xanthobacter flavus is an NADPH sensor
- Publication venue
- Publication date
- 01/03/1998
- Field of study
Get PDFThe LysR-type transcriptional regulator CbbR controlling autotrophic CO2 fixation by Xanthobacter flavus is an NADPH sensor
- Publication venue
- Publication date
- 01/03/1998
- Field of study
Get PDFNeo-Newtonian cosmology: An intermediate step towards General Relativity
- Author
- Bondi H.
- Cartan E.
- Chisari N.E.
- Einstein A.
- Einstein A.
- Einstein A.
- Eisenstaedt J.
- Fabris J.C.
- Fabris J.C.
- Friedmann A.
- Friedrichs K.
- Green S.R.
- Harrison E.R.
- Hawking S.W.
- Hubble E.P.
- Kuhn T.S.
- Landau L.
- Layzer D.
- Lemaître G.
- Lima J.A.S.
- Malament D.
- Mavridès S.
- McCrea W.H.
- McCrea W.H.
- Milne E.A.
- Milne E.A.
- Norton J.
- Norton J.
- Norton J.
- Ollitrault J-Y.
- Rasanen S.
- Ribeiro M.B.
- Shirata A.
- Tipler F.J.
- van den Bergh S.
- von Seeliger H.
- Waga I.
- Whittaker E.T.
- Publication venue
- Publication date
- 01/01/2012
- Field of study
Full text linkCosmology is a field of physics in which the use of General Relativity theory
is indispensable. However, a cosmology based on Newtonian gravity theory for
gravity is possible in certain circumstances. The applicability of Newtonian
theory can be substantially extended if it is modified in such way that
pressure has a more active role as source of the gravitational field. This was
done in the neo-Newtonian cosmology. The limitation on the construction of a
Newtonian cosmology, and the need for a relativistic theory in cosmology are
reviewed. The neo-Newtonian proposal is presented, and its consequences for
cosmology are discussed.Comment: 10 pages. Portuguese version submitted to RBE
New insights into the genetic etiology of Alzheimer's disease and related dementias
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- Aaltonen V.
- Aarsland Dag
- Aavikko M.
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- Aguilera N.
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- Alarcón-Martín Emilio
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Get PDFCharacterization of the genetic landscape of Alzheimer's disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/'proxy' AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE ε4 allele
Induction of the gap-pgk operon encoding glyceraldehyde-3-phosphate dehydrogenase and 3-phosphoglycerate kinase of Xanthobacter flavus requires the LysR-type transcriptional activator CbbR
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No full textInduction of the gap-pgk operon encoding glyceraldehyde-3-phosphate dehydrogenase and 3-phosphoglycerate kinase of Xanthobacter flavus requires the LysR-type transcriptional activator CbbR
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No full textB-Type D-Branes in Toric Calabi–Yau Varieties
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No full textPulsars: their origin and evolution
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No full textApplication of the emerging technologies in toxicogenomics: An overview
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No full textNew insights into the genetic etiology of Alzheimer’s disease and related dementias
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- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2022
- Field of study
No full textCharacterization of the genetic landscape of Alzheimer’s disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/‘proxy’ AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE ε4 allele. © 2022, The Author(s)
