3,807 research outputs found

    Joint relay scheduling, channel access, and power allocation for green cognitive radio communications

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    PublishedJournal Article© 1983-2012 IEEE. The capacity of cognitive radio (CR) systems can be enhanced significantly by deploying relay nodes to exploit the spatial diversity. However, the inevitable imperfect sensing in CR has vital effects on the policy of relay selection, channel access, and power allocation that play pivotal roles in the system capacity. The increase in transmission power can improve the system capacity, but results in high energy consumption, which incurs the increase of carbon emission and network operational cost. Most of the existing schemes for CR systems have not jointly considered the imperfect sensing scenario and the tradeoff between the system capacity and energy consumption. To fill in this gap, this paper proposes an energy-aware centralized relay selection scheme that takes into account the relay selection, channel access, and power allocation jointly in CR with imperfect sensing. Specifically, the CR system is formulated as a partially observable Markov decision process (POMDP) to achieve the goal of balancing the system capacity and energy consumption as well as maximizing the system reward. The optimal policy for relay selection, channel access, and power allocation is then derived by virtue of a dynamic programming approach. A dimension reduction strategy is further applied to reduce its high computation complexity. Extensive simulation experiments and results are presented and analysed to demonstrate the significant performance improvement compared to the existing schemes. The performance results show that the received reward increases more than 50% and the network lifetime increases more than 35%, but the system capacity is reduced less than 6% only.This work was supported by the National Natural Science Foundation of China under Grants 61201219, 61171111, 61472150, and 61173045 and in part by the Fundamental Research Funds for the Central Universities under Grant 2013QN122

    Multi-Antenna Covert Communication via Full-Duplex Jamming Against a Warden With Uncertain Locations

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    Covert communication can hide the information transmission process from the warden to prevent adversarial eavesdropping. However, it becomes challenging when the location of warden is uncertain. In this paper, we propose a covert communication scheme against a warden with uncertain locations, which maximizes the connectivity throughput between a multi-antenna transmitter and a full-duplex jamming receiver with the limit of covert outage probability (the probability of the transmission found by the warden). First, we analyze the monotonicity of the covert outage probability to obtain the optimal location for the warden. Then, under this worst situation, we optimize the transmission rate, the transmit power and the jamming power of covert communication to maximize the connection throughput. This problem is solved in two stages. First, we derive the transmit-to-jamming power ratio limit from the maximum allowed covert outage probability. With this constraint, the connection probability is maximized over the transmit-to-jamming power ratio for a fixed transmission rate. Since the connection probability and the transmission rate are coupled, the bisection method is applied to maximize the connectivity throughput via optimizing the transmission rate iteratively. Simulation results are presented to evaluate the effectiveness of the proposed scheme

    Identification and Characterisation of the Early Differentiating Cells in Neural Differentiation of Human Embryonic Stem Cells

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    One of the challenges in studying early differentiation of human embryonic stem cells (hESCs) is being able to discriminate the initial differentiated cells from the original pluripotent stem cells and their committed progenies. It remains unclear how a pluripotent stem cell becomes a lineage-specific cell type during early development, and how, or if, pluripotent genes, such as Oct4 and Sox2, play a role in this transition. Here, by studying the dynamic changes in the expression of embryonic surface antigens, we identified the sequential loss of Tra-1-81 and SSEA4 during hESC neural differentiation and isolated a transient Tra-1-81(−)/SSEA4(+) (TR−/S4+) cell population in the early stage of neural differentiation. These cells are distinct from both undifferentiated hESCs and their committed neural progenitor cells (NPCs) in their gene expression profiles and response to extracellular signalling; they co-express both the pluripotent gene Oct4 and the neural marker Pax6. Furthermore, these TR−/S4+ cells are able to produce cells of both neural and non-neural lineages, depending on their environmental cues. Our results demonstrate that expression of the pluripotent factor Oct4 is progressively downregulated and is accompanied by the gradual upregulation of neural genes, whereas the pluripotent factor Sox2 is consistently expressed at high levels, indicating that these pluripotent factors may play different roles in the regulation of neural differentiation. The identification of TR-S4+ cells provides a cell model for further elucidation of the molecular mechanisms underlying hESC neural differentiation

    Photon-induced conduction modulation in SiO 2 thin films embedded with Ge nanocrystals

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    The authors report the photon-induced conduction modulation in Si O2 thin films embedded with germanium nanocrystals (nc-Ge). The conduction of the oxide could be switched to a higher- or lower-conductance state by a ultraviolet (UV) illumination. The conduction modulation is caused by charging and discharging in the nc-Ge due to the UV illumination. If the charging process is dominant, the oxide conductance is reduced; however, if the discharging process is dominant, the oxide conductance is increased. As the conduction can be modulated by UV illumination, it could have potential applications in silicon-based optical memory devices. © 2007 American Institute of Physics.published_or_final_versio

    Extracranial head and neck schwannomas: a study of the nerve of origin

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    Schwannoma is a type of benign nerve sheath tumour arising from the Schwann cell. Because of the close relationship between the tumour and the nerve of origin (NOO), the operation of extracranial head and neck schwannoma may lead to palsy of major nerve. For this reason, an accurate diagnosis of schwannoma with the identification of the NOO is crucial to the management. The aim of this review was to find out the distribution of the NOO and the usefulness of the investigations in the diagnosis of schwannoma. Medical records of the patients who underwent operation of the extracranial head and neck schwannoma in our division were reviewed. Between January 2000 and December 2009, 30 cases of extracranial head and neck schwannoma were operated. Sympathetic trunk (10, 33%) and vagus nerve (6, 20%) were the two most common NOOs. In five (17%) cases, the NOO was not found to be arising from any major nerve. For these 30 patients, 20 received fine needle aspiration cytology (FNAC) and 26 underwent imaging studies (computed tomography or magnetic resonance imaging) before operation. The specificity of FNAC and imaging studies in making the diagnosis of schwannoma was 20 and 38%, respectively. For the patients who had nerve palsies on presentation, their deficits remained after operation. The rate of nerve palsy after tumour excision with division of NOO and intracapsular enucleation was 100 and 67%, respectively. The diagnosis of schwannoma is suggested by clinical features and supported by investigations. Most of the time, the diagnosis can only be confirmed on the histological study of the surgical specimen. Sympathetic trunk and vagus nerve are the two common NOOs. MRI is the investigation of choice in the diagnosis of schwannoma and the identification of NOO

    Influence of charge trapping on electroluminescence from Si-nanocrystal light emitting structure

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    We report a study on the influence of charge trapping on electroluminescence (EL) from Si nanocrystal (nc-Si) distributed throughout a 30 nm Si O2 thin film synthesized by Si+ implantation into an oxide film thermally grown on a p -type Si substrate. The electron and hole trapping in the nc-Si located near the indium tin oxide gate and the Si substrate, respectively, cause a reduction in the EL intensity. The reduced EL intensity can be recovered after the trapped charges are released. A partial recovery can be easily achieved by the application of a positive gate voltage or thermal annealing at hot temperatures (e.g., 120 °C) for a short duration. The present study highlights the impact of charging in the nc-Si on the light emission efficiency and its stability of nc-Si light-emitting devices. © 2007 American Institute of Physics.published_or_final_versio

    TGF-beta(2)- and H2O2-Induced Biological Changes in Optic Nerve Head Astrocytes Are Reduced by the Antioxidant Alpha-Lipoic Acid

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    Background/Aims: The goal of the present study was to determine whether transforming growth factor-beta(2) (TGF-beta(2))- and oxidative stress-induced cellular changes in cultured human optic nerve head (ONH) astrocytes could be reduced by pretreatment with the antioxidant alpha-lipoic acid (LA). Methods: Cultured ONH astrocytes were treated with 1.0 ng/ml TGF-beta(2) for 24 h or 200 mu M hydrogen peroxide (H2O2) for 1 h. Lipid peroxidation was measured by a decrease in cis-pari-naric acid fluorescence. Additionally, cells were pretreated with different concentrations of LA before TGF-beta 2 or H2O2 exposure. Expressions of the heat shock protein (Hsp) alpha B-crystallin and Hsp27, the extracellular matrix (ECM) component fibronectin and the ECM-modulating protein connective tissue growth factor (CTGF) were examined with immunohistochemistry and real-time PCR analysis. Results: Both TGF-beta(2) and H2O2 increased lipid peroxidation. Treatment of astrocytes with TGF-beta(2) and H2O2 upregulated the expression of alpha B-crystallin, Hsp27, fibronectin and CTGF. Pretreatment with different concentrations of LA reduced the TGF-beta(2)- and H2O2-stimulated gene expressions. Conclusion: We showed that TGF-beta(2)- and H2O2-stimulated gene expressions could be prevented by pretreatment with the antioxidant LA in cultured human ONH astrocytes. Therefore, it is tempting to speculate that the use of antioxidants could have protective effects in glaucomatous optic neuropathy. Copyright (C) 2012 S. Karger AG, Base

    Ageing test of the ATLAS RPCs at X5-GIF

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    An ageing test of three ATLAS production RPC stations is in course at X5-GIF, the CERN irradiation facility. The chamber efficiencies are monitored using cosmic rays triggered by a scintillator hodoscope. Higher statistics measurements are made when the X5 muon beam is available. We report here the measurements of the efficiency versus operating voltage at different source intensities, up to a maximum counting rate of about 700Hz/cm^2. We describe the performance of the chambers during the test up to an overall ageing of 4 ATLAS equivalent years corresponding to an integrated charge of 0.12C/cm^2, including a safety factor of 5.Comment: 4 pages. Presented at the VII Workshop on Resistive Plate Chambers and Related Detectors; Clermont-Ferrand October 20th-22nd, 200
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