A Better Choice? A Case Study of Polish Migrants' Educational Aspirations and School Choice for Their Children

Using a case study of Polish parents living in North London and Nottingham, this thesis analyses their interaction and strategies within two localised education markets. Drawing on qualitative research, perceptions of social divisions in British society are explored in an attempt to assess whether these influence their educational strategies and practices used in secondary school choice for their children. It is found that the Polish symbolisation of Catholicism as representing their national cultural “morals” and “values” becomes a guiding influence in school choice. It also offers them a support network in gathering information about the education market. “Whiteness” and the stigmatising of disadvantaged sections of society also becomes a channel for some of the Polish participants to position themselves against others and within a perceived “hierarchy” which ultimately impacts upon their strategies used in parental school choice. A Bourdieusian framework, exploring the influence of reciprocal “importing” and “exporting” societal structures, is used to analyse the findings

Structuring East to West Migration: A Case Study of Central and Eastern European Migrants to Britain

Focusing on Central and Eastern European migration from accession states to England, this paper seeks to explore how migration has been affected by global level socioeconomic and political transformations that have occurred as part of wider global integration. The study explores Central and Eastern European migrant’s experiences of globalisation at a micro level. The argument in this paper seeks to conceptualised East to West migration as a structuration process: using the analytical categories of social structure and human agency, and applying the structuration model to explain the reciprocal influence of migrant’s home and host societal structures in shaping their activities and goals (Morawska, 2001). The paper also aims to show that East to West migration is determined by market forces and that EU accession is used as an enabling structure by the migrants to regularise the already existing process of Central and Eastern European migration flows

Dancing in the moon : teaching poetry to children : an honors thesis (HONRS 499)

There is no abstract available for this thesis.Thesis (B.?.)Honors Colleg

The combination of valsartan and sacubitril does not improve effectiveness against AngII-induced AAAs

Abdominal aortic aneurysm (AAA) is defined as permanent dilation of the abdominal aorta by more than 50% of its normal diameter. As AAAs grow progressively, the aorta can rupture and cause life-threatening bleeding. Preliminary data demonstrates that angiotensin II (AngII), which can induce AAA, plus testosterone were powerful stimulants for expression of neprilysin in abdominal aortic smooth muscle cells from male mice. Neprilysin is a metalloendopeptidase, which can cleave and inactivate AngII and natriuretic peptides. Because natriuretic peptides might be beneficial for reducing the growth of AAAs through decreasing blood pressure, inhibition of neprilysin along with blocking AngII receptor would be predicted to decrease AAA formation. Sacubitril is a competitive inhibitor of neprilysin and has been utilized with valsartan, an AngII receptor blocker, in the treatment of heart failure; however, this combination has not been tested against AAA formation. Therefore, in this study we evaluated the effects of valsartan, sacubitril, and their combination on the formation of AngII-induced AAAs. We treated the male mice with either solvent (control), valsartan (0.3, 0.5, 1, 6, or 20 mg/kg/day), sacubitril (1, 6, and 9 mg/kg/day), or the combination, which were 0.3 mg/kg/day valsartan with 1 mg/kg/day sacubitril or 0.5 mg/kg/day valsartan with 9 mg/kg/day sacubitril. All the treatments were infused (sc) for seven days prior to administering AngII and then along with AngII infusion for 28 days. We found that valsartan, except for the 0.3 mg/kg/day dose, dose-dependently inhibited AngII-infused AAA formation compared to the control. Sacubitril did not inhibit AAA formation at any dose examined. Combinational drug therapy also showed no additional effect on reducing AAA formation beyond that of valsartan alone