Two phase galaxy formation: The Evolutionary Properties of Galaxies

We use our model for the formation and evolution of galaxies within a two-phase galaxy formation scenario, showing that the high-redshift domain typically supports the growth of spheroidal systems, whereas at low redshifts the predominant baryonic growth mechanism is quiescent and may therefore support the growth of a disc structure. Under this framework we investigate the evolving galaxy population by comparing key observations at both low and high-redshifts, finding generally good agreement. By analysing the evolutionary properties of this model, we are able to recreate several features of the evolving galaxy population with redshift, naturally reproducing number counts of massive star-forming galaxies at high redshifts, along with the galaxy scaling relations, star formation rate density and evolution of the stellar mass function. Building upon these encouraging agreements, we make model predictions that can be tested by future observations. In particular, we present the expected evolution to z=2 of the super-massive black hole mass function, and we show that the gas fraction in galaxies should decrease with increasing redshift in a mass, with more and more evolution going to higher and higher masses. Also, the characteristic transition mass from disc to bulge dominated system should decrease with increasing redshift.Comment: 15 pages, 11 figures. Version polished for publication in MNRA

A Physical Model for Co-evolution of QSOs and of their Spheroidal Hosts

At variance with most semi-analytic models, in the Anti-hierarchical Baryon Collapse scenario (Granato et al. 2001, 2004) the main driver of the galaxy formation and evolution is not the merging sequence but are baryon processes. This approach emphasizes, still in the framework of the hierarchical clustering paradigm for dark matter halos, feedback processes from supernova explosions and from active nuclei, that tie together star formation in spheroidal galaxies and the growth of black holes at their centers. We review some recent results showing the remarkably successful predictive power of this scenario, which allows us to account for the evolution with cosmic time of a broad variety of properties of galaxies and active nuclei, which proved to be very challenging for competing models.Comment: Invited talk at the Specola Vaticana Workshop on "AGN and Galaxy Evolution", Castel Gandolfo, 3-6 October 2005, 10 pages, 2 figure

The impact of the FMR and starburst galaxies on the (low metallicity) cosmic star formation history

The question how much star formation is occurring at low metallicity throughout the cosmic history appears crucial for the discussion of the origin of various energetic transients, and possibly double black hole mergers. We revisit the observation-based distribution of birth metallicities of stars (fSFR(Z,z)), focusing on several factors that strongly affect its low metallicity part: (i) the method used to describe the metallicity distribution of galaxies (redshift-dependent mass metallicity relation - MZR, or redshift-invariant fundamental metallicity relation - FMR), (ii) the contribution of starburst galaxies and (iii) the slope of the MZR. We empirically construct the FMR based on the low-redshift scaling relations, which allows us to capture the systematic differences in the relation caused by the choice of metallicity and star formation rate (SFR) determination techniques and discuss the related fSFR(Z,z) uncertainty. We indicate factors that dominate the fSFR(Z,z) uncertainty in different metallicity and redshift regimes. The low metallicity part of the distribution is poorly constrained even at low redshifts (even a factor of ∼200 difference between the model variations) The non-evolving FMR implies a much shallower metallicity evolution than the extrapolated MZR, however, its effect on the low metallicity part of the fSFR(Z,z) is counterbalanced by the contribution of starbursts (assuming that they follow the FMR). A non-negligible fraction of starbursts in our model may be necessary to satisfy the recent high-redshift SFR density constraints

A direct and robust method to observationally constrain the halo mass function via the submillimeter magnification bias: Proof of concept

Aims. The main purpose of this work is to provide a proof-of-concept method to derive tabulated observational constraints on the halo mass function (HMF) by studying the magnification bias effect on high-redshift submillimeter galaxies. Under the assumption of universality, we parametrize the HMF according to two traditional models, namely the Sheth and Tormen (ST) and Tinker fits, derive posterior distributions for their parameters, and assess their performance in explaining the measured data within the \u39b cold dark matter model. We also study the potential influence of the halo occupation distribution (HOD) parameters in this analysis and discuss two aspects regarding the HMF parametrization, namely its normalization and the possibility of allowing negative values for the parameters. Methods. We measure the cross-correlation function between a foreground sample of GAMA galaxies with spectroscopic redshifts in the range 0.2 < z < 0.8 and a background sample of H-ATLAS galaxies with photometric redshifts in the range 1.2 < z < 4.0 and carry out a Markov chain Monte Carlo algorithm in the context of Bayesian inference to check this observable against its mathematical prediction within the halo model formalism, which depends on both the HOD and HMF parameters. Results. Under the assumption that all HMF parameters are positive, the ST fit only seems to fully explain the measurements by forcing the mean number of satellite galaxies in a halo to increase substantially from its prior mean value. The Tinker fit, on the other hand, provides a robust description of the data without relevant changes in the HOD parameters, but with some dependence on the prior range of two of its parameters. When the normalization condition for the HMF is dropped and we allow negative values of the p1 parameter in the ST fit, all the involved parameters are better determined, unlike the previous models, thus deriving the most general HMF constraints. While all the aforementioned cases are in agreement with the traditional fits within the uncertainties, the last one hints at a slightly higher number of halos at intermediate and high masses, raising the important point of the allowed parameter range

Coordination of the Co2+and Ni2+Ions in Tf2N-Based Ionic Liquids: A Combined X-ray Absorption and Molecular Dynamics Study

Molecular dynamics (MD) simulations and X-ray absorption spectroscopy (XAS) have been combined to study the coordination of the Co2+ and Ni2+ ions in ionic liquids (ILs) based on the bis(trifluoromethylsulfonyl)imide ([Tf2N]-) anion and having different organic cations, namely, 1-butyl-3-methylimidazolium ([C4mim]+), 1,8-bis(3-methylimidazolium-1-yl)octane ([C8(mim)2]2+), N,N,N-trimethyl-N-(2-hydroxyethyl)ammonium ([choline]+), and butyltrimethylammonium ([BTMA]+). Co and Ni K-edge XAS data have been collected on 0.1 mol L-1 Co(Tf2N)2 and Ni(Tf2N)2 solutions and on the metallic salts. MD simulations have been carried out to obtain structural information on the metal ion coordination. The analysis of the extended X-ray absorption fine structure (EXAFS) spectra of the solutions has been carried out based on the atomistic description provided by MD, and the studied ILs have been found to be able to dissolve both the Co(Tf2N)2 and Ni(Tf2N)2 salts giving rise to a different structural arrangement around the metal ions as compared to the solid state. The combined EXAFS and MD results showed that the Co2+ and Ni2+ ions are surrounded by a first solvation shell formed by six [Tf2N]- anions, each coordinating in a monodentate fashion by means of the oxygen atoms. The nature of the IL organic cation has little or no influence on the overall spatial arrangement of the [Tf2N]- anions, so that stable octahedral complexes of the type [M(Tf2N)6]4- (M = Co, Ni) have been observed in all the investigated ILs

Punching of reinforced concrete flat slabs – Rational use of high strength concrete

PTDC/ECI-EST/30511/2017; SFRH/BD/76242/2011; The authors are grateful to CIMPOR for providing the cement and Sika for providing VISCOCRETE 20HE and SIKACRETE HD.This paper deals with punching of reinforced high strength concrete (HSC) flat slabs. Despite the use of HSC increased significantly in the last years, the experimental research on punching behavior of HSC slabs is still limited. Furthermore, most of this past research adopted concrete compressive strength lower than 90 MPa. In a previous work by this research group three specimens with concrete compressive strength around 120 MPa and one with normal strength concrete (NSC) were tested. The present work represents the continuation of the previous activity and it is focused on the rational use of HSC. Four specimens with HSC and one of NSC were tested under monotonic vertical loading. The HSC was placed only in the slab-column connection region and it was limited to a thin layer in the compressive zone, in order to have a more economical and sustainable solution. This rational use of the HSC showed excellent results in terms of punching strength. Limiting the HSC to a thin layer in the compressive zone resulted in an almost equal punching strength to that obtained with the slab entirely casted in HSC.authorsversionpublishe

Rat pial microvascular responses to melatonin during bilateral common carotid artery occlusion and reperfusion

The present study assessed the in vivo rat pial microvascular responses induced by melatonin during brain hypoperfusion and reperfusion (RE) injury. Pial microcirculation of male Wistar rats was visualized by fluorescence microscopy through a closed cranial window. Hypoperfusion was induced by bilateral common carotid artery occlusion (BCCAO, 30 min); thereafter, pial microcirculation was observed for 60 min. Arteriolar diameter, permeability increase, leukocyte adhesion to venular walls, perfused capillary length (PCL), and capillary red blood cell velocity (V(RBC) ) were investigated by computerized methods. Melatonin (0.5, 1, 2 mg/kg b.w.) was intravenously administered 10 min before BCCAO and at the beginning of RE. Pial arterioles were classified in five orders according to diameter, length, and branchings. In control group, BCCAO caused decrease in order 2 arteriole diameter (by 17.5 ± 3.0% of baseline) that was reduced by 11.8 ± 1.2% of baseline at the end of RE, accompanied by marked leakage and leukocyte adhesion. PCL and capillary V(RBC) decreased. At the end of BCCAO, melatonin highest dosage caused order 2 arteriole diameter reduction by 4.6 ± 2.0% of baseline. At RE, melatonin at the lower dosages caused different arteriolar responses. The highest dosage caused dilation in order 2 arteriole by 8.0 ± 1.5% of baseline, preventing leakage and leukocyte adhesion, while PCL and V(RBC) increased. Luzindole (4 mg/kg b.w.) prior to melatonin caused order 2 arteriole constriction by 12.0 ± 1.5% of baseline at RE, while leakage, leukocyte adhesion, PCL and V(RBC) were not affected. Prazosin (1 mg/kg b.w.) prior to melatonin did not significantly change melatonin's effects. In conclusion, melatonin caused different responses during hypoperfusion and RE, modulating pial arteriolar tone likely by MT1 and MT2 melatonin receptors while preventing blood-brain barrier changes through its free radical scavenging action

The Transcription Factor Nfix Requires RhoA-ROCK1 Dependent Phagocytosis to Mediate Macrophage Skewing during Skeletal Muscle Regeneration

Macrophages (MPs) are immune cells which are crucial for tissue repair. In skeletal muscle regeneration, pro-inflammatory cells first infiltrate to promote myogenic cell proliferation, then they switch into an anti-inflammatory phenotype to sustain myogenic cells differentiation and myofiber formation. This phenotypical switch is induced by dead cell phagocytosis. We previously demonstrated that the transcription factor Nfix, a member of the nuclear factor I (Nfi) family, plays a pivotal role during muscle development, regeneration and in the progression of muscular dystrophies. Here, we show that Nfix is mainly expressed by anti-inflammatory macrophages. Upon acute injury, mice deleted for Nfix in myeloid line displayed a significant defect in the process of muscle regeneration. Indeed, Nfix is involved in the macrophage phenotypical switch and macrophages lacking Nfix failed to adopt an anti-inflammatory phenotype and interact with myogenic cells. Moreover, we demonstrated that phagocytosis induced by the inhibition of the RhoA-ROCK1 pathway leads to Nfix expression and, consequently, to acquisition of the anti-inflammatory phenotype. Our study identified Nfix as a link between RhoA-ROCK1-dependent phagocytosis and the MP phenotypical switch, thus establishing a new role for Nfix in macrophage biology for the resolution of inflammation and tissue repair