Adaptation of Journal Article Tag Suite XML for Japanese humanities papers

Out of East Asian languages which do not use the Latin alphabet, Japanese is a very complicated writing system that uses “kanji,” which are ideograms, and “kana,” which are phonetic characters. Most of the Japanese papers published so far using Journal Article Tag Suite (JATS) are science, technology, and medicine fields adapting horizontal writing systems, which are structurally consistent with English papers. Most of them only replace Latin letters with Japanese characters. In this presentation, we suggested method of presenting vertically oriented Japanese humanities articles in JATS XML. For vertical description of Chinese numeric, we would like to propose the introduction of an element which specifies description direction. Alternatively, could be used as a hidden command when creating a document. We propose the following notation in the part of the number that can be converted: 六五. Chinese numeric 六五 is a Arabic numeric 65. With this, it is shown that 六五 of Chinese numerals can be converted to 65 in Arabic numerals. For vertical text description with JATS, we would like to suggest adding @ writing-mode as an attribute to :. Furthermore, note and references should be differentiated for example, between a and a in the future. As Kanji are ideograms, there are variations that cannot be expressed with UTF-8. If these difficult Kanji are included in the JATS text, it will be necessary to decide on their description method. For the propagation of use of JATS XML for non-Latin characters articles, the structure of the document for example, vertical description, and special presentation should be considered more widely

Interaction between Engineered Cementitious Composites Lining and Foundation Subsurface Drain

The effect of cyclic loads on the surface profiles of ECC linings cast on foundations comprising crushed stone and compacted soil was investigated. A geotextile was embedded between the crushed stone and ECC lining for some of the samples. After 28 days of water curing, the hardened surfaces were loaded and monitored for roughness and crack development by measuring surface levels and crack widths, respectively. Neither cracking nor significant variations in the lateral profiles were observed on all the samples for all the loads applied. However, significant variations which depended on the foundation types were observed in the vertical profiles. It was concluded that while ECC can resist cracking due to its high strain capacity, its flexibility causes ECC linings to assume the shape of the foundation material, which can increase the surface roughness at certain loading configurations

Urocortin 1 reduces food intake and ghrelin secretion via CRF2 receptors

Although it is known that urocortin 1 (UCN) acts on both corticotropin-releasing factor receptors (CRF1 and CRF2), the mechanisms underlying UCN-induced anorexia remain unclear. In contrast, ghrelin, the endogenous ligand for the growth hormone secretagogue receptor, stimulates food intake. In the present study, we examined the effects of CRF1 and CRF2 receptor antagonists (CRF1a and CRF2a) on ghrelin secretion and synthesis, c-fos mRNA expression in the caudal brain stem, and food intake following intracerebroventricular administration of UCN. Eight-week-old, male Sprague-Dawley rats were used after 24-h food deprivation. Acylated and des-acylated ghrelin levels were measured by enzyme-linked immunosorbent assay. The mRNA expressions of preproghrelin and c-fos were measured by real-time RT-PCR. The present study provided the following important insights into the mechanisms underlying the anorectic effects of UCN: 1) UCN increased acylated and des-acylated ghrelin levels in the gastric body and decreased their levels in the plasma; 2) UCN decreased preproghrelin mRNA levels in the gastric body; 3) UCN-induced reduction of plasma ghrelin and food intake were restored by CRF2a but not CRF1a; 4) UCN-induced increase of c-fos mRNA levels in the caudal brain stem containing the nucleus of the solitary tract (NTS) was inhibited by CRF2a; and 5) UCN-induced reduction of food intake was restored by exogenous ghrelin and rikkunshito, an endogenous ghrelin secretion regulator. Thus, UCN increases neuronal activation in the caudal brain stem containing NTS via CRF2 receptors, which may be related to UCN-induced inhibition of both ghrelin secretion and food intake