On the environments of Type Ia supernovae within host galaxies

We present constraints on supernovae type Ia (SNe Ia) progenitors through an analysis of the environments found at the explosion sites of 102 events within star-forming host galaxies. Hα and GALEX near-UV images are used to trace on-going and recent star formation (SF), while broad band B,R, J,K imaging is also analysed. Using pixel statistics we find that SNe Ia show the lowest degree of association with Hα emission of all supernova types. It is also found that they do not trace near-UV emission. As the latter traces SF on timescales less than 100Myr, this rules out any extreme ‘prompt’ delay-times as the dominant progenitor channel of SNe Ia. SNe Ia best trace the B-band light distribution of their host galaxies. This implies that the population within star-forming galaxies is dominated by relatively young progenitors. Splitting SNe by their (B-V) colours at maximumlight, ‘redder’ events show a higher degree of association to H II regions and are found more centrally within hosts. We discuss possible explanations of this result in terms of line of sight extinction and progenitor effects. No evidence for correlations between SN stretch and environment properties is observed. Key words: supernovae: general, galaxies: statistic

PESSTO monitoring of SN 2012hn: further heterogeneity among faint type I supernovae

We present optical and infrared monitoring data of SN 2012hn collected by the Public ESO Spectroscopic Survey for Transient Objects (PESSTO). We show that SN 2012hn has a faint peak magnitude (MR ~ -15.7) and shows no hydrogen and no clear evidence for helium in its spectral evolution. Instead, we detect prominent Ca II lines at all epochs, which relates this transient to previously described 'Ca-rich' or 'gap' transients. However, the photospheric spectra (from -3 to +32 d with respect to peak) of SN 2012hn show a series of absorption lines which are unique, and a red continuum that is likely intrinsic rather than due to extinction. Lines of Ti II and Cr II are visible. This may be a temperature effect, which could also explain the red photospheric colour. A nebular spectrum at +150d shows prominent CaII, OI, CI and possibly MgI lines which appear similar in strength to those displayed by core-collapse SNe. To add to the puzzle, SN 2012hn is located at a projected distance of 6 kpc from an E/S0 host and is not close to any obvious starforming region. Overall SN 2012hn resembles a group of faint H-poor SNe that have been discovered recently and for which a convincing and consistent physical explanation is still missing. They all appear to explode preferentially in remote locations offset from a massive host galaxy with deep limits on any dwarf host galaxies, favouring old progenitor systems. SN 2012hn adds heterogeneity to this sample of objects. We discuss potential explosion channels including He-shell detonations and double detonations of white dwarfs as well as peculiar core-collapse SNe.Comment: 14 pages, 14 figures, accepted to MNRAS on 14/10/201

Fate specification and tissue-specific cell cycle control of the <i>Caenorhabditis elegans</i> intestine

Coordination between cell fate specification and cell cycle control in multicellular organisms is essential to regulate cell numbers in tissues and organs during development, and its failure may lead to oncogenesis. In mammalian cells, as part of a general cell cycle checkpoint mechanism, the F-box protein β-transducin repeat-containing protein (β-TrCP) and the Skp1/Cul1/F-box complex control the periodic cell cycle fluctuations in abundance of the CDC25A and B phosphatases. Here, we find that the Caenorhabditis elegans β-TrCP orthologue LIN-23 regulates a progressive decline of CDC-25.1 abundance over several embryonic cell cycles and specifies cell number of one tissue, the embryonic intestine. The negative regulation of CDC-25.1 abundance by LIN-23 may be developmentally controlled because CDC-25.1 accumulates over time within the developing germline, where LIN-23 is also present. Concurrent with the destabilization of CDC-25.1, LIN-23 displays a spatially dynamic behavior in the embryo, periodically entering a nuclear compartment where CDC-25.1 is abundant