TB78: Sequential Surveys of the Pine Leaf Chermid, Pineus pinifoliae

Sequential survey procedures are described for classifying damage to white pine produced by the pine leaf chermid. Damage classes are based on degree of needle stunting, and field procedures for making measurements are given. Survey procedures are also presented for classifying infestation levels of two stages of the insect. These allow prediction of damage levels before damage occurs.https://digitalcommons.library.umaine.edu/aes_techbulletin/1119/thumbnail.jp

B837: New Approaches to Chemical Control of White Pine Weevil Damage

There has been little recent research on control of damage by the white pine weevil, and available insecticides have been few and have become obsolete. Tests in Canada suggested that the insect growth-regulating chemical diflubenzeron was effective, and we have successfully repeated those tests in Maine using several formulations of Dimilin and several ground application systems. Aerial trials have not succeeded; the probable reasons for their failure are discussed. We present a general description of the weevil and its damage, approaches to control of damage, and specific recommendations for use of ground applications of Dimilin, which has recently become registered for this use.https://digitalcommons.library.umaine.edu/aes_bulletin/1010/thumbnail.jp

B658: Susceptibility and Vulnerability of Forests to the Pine Leaf Aphid Pineus Pinilolile (Fitch) (Adelgidae)

In the late 1950s, early 1960s, Maine and surrounding regions experienced an outbreak of the pine leaf aphid (or adelgid). The population progression began about 1955, as indicated by tree growth reductions (2), a peak was reached about 1961, after which populations gradually regressed through the late 1960s. As a result of the outbreak, there was considerable growth reduction of white pine in some regions and scattered tree mortality. Among the many observations on the insect made during the outbreak were (a) the aphid was abundant in only certain portions of Maine and remained uncommon in the remainder of the state, and (b) in those regions where the insect was abundant, some stands of pine suffered relatively severe damage while others were largely unaffected. This study sought to provide explanations for differences in the abundance of aphids throughout the state and the varying amount of damage between stands. Information gained in a study of this sort is useful in explaining the distribution and abundance of the insect and in suggesting silvicultural procedures designed to increase resistance of stands to insect damage.https://digitalcommons.library.umaine.edu/aes_bulletin/1015/thumbnail.jp

Patterns of Tree Mortality During an Uncontrolled Spruce Budworm Outbreak in Baxter State Park, 1983

A forest inventory of Baxter State Park, Maine, was done during the summer and fall of 1983 to study the patterns of mortality in balsam fir and in the red-black spruce complex during an uncontrolled spruce budworm outbreak. The volume of trees that died during the outbreak was estimated at 40% of the original quantity. Fir mortality was consistently greater than that of spruce. The percentage mortality of spruce was always greater in the higher elevation zone than in the lower elevation zone. Percentage mortality of fir and spruce showed inconsistent patterns in relation to the proportion of hardwood species basal area and to the proportion of fir basal area in a stand. However, those mortality patterns tended to be pronounced when the overall tree mortality was relatively high. Hypotheses on the mechanisms producing the tree mortality patterns during a budworm outbreak were summarized and observed tree mortality patterns were then interpreted according to those hypotheses. Most observed patterns could be explained by more than one hypothesis. It was suggested that complex processes were responsible for developing certain tree mortality patterns, and a deductive method based on the simple observations would not reveal the true mechanisms.https://digitalcommons.library.umaine.edu/aes_techbulletin/1081/thumbnail.jp

Patterns of Tree Mortality During an Uncontrolled Spruce Budworm Outbreak in Baxter State Park, 1983

A forest inventory of Baxter State Park, Maine, was done during the summer and fall of 1983 to study the patterns of mortality in balsam fir and in the red-black spruce complex during an uncontrolled spruce budworm outbreak. The volume of trees that died during the outbreak was estimated at 40% of the original quantity. Fir mortality was consistently greater than that of spruce. The percentage mortality of spruce was always greater in the higher elevation zone than in the lower elevation zone. Percentage mortality of fir and spruce showed inconsistent patterns in relation to the proportion of hardwood species basal area and to the proportion of fir basal area in a stand. However, those mortality patterns tended to be pronounced when the overall tree mortality was relatively high. Hypotheses on the mechanisms producing the tree mortality patterns during a budworm outbreak were summarized and observed tree mortality patterns were then interpreted according to those hypotheses. Most observed patterns could be explained by more than one hypothesis. It was suggested that complex processes were responsible for developing certain tree mortality patterns, and a deductive method based on the simple observations would not reveal the true mechanisms.https://digitalcommons.library.umaine.edu/aes_techbulletin/1081/thumbnail.jp

B819: The Spruce Budworm Outbreak in Maine in the 1970\u27s–Assessment and Directions for the Future

This report was initiated by the Maine Forest Service (MFS) in response to concerns that a serious effort was needed to capture the experiences and lessons learned during the 1970-85 spruce budworm outbreak in Maine. The report synthesizes the observations and experiences of land managers, as well as the principal results of recent scientific research on spruce budworm in Maine. This report briefly reviews budworm population dynamics and interactions with the forest, then describes the budworm\u27s impacts in detail. It then reviews the three principal responses: survey and detection; spraying; and silviculture and salvage. It then offers an overview of the outbreak\u27s effects and provides a summary of conclusions and recommendations for the future.https://digitalcommons.library.umaine.edu/aes_bulletin/1044/thumbnail.jp

Sociological and Human Developmental Explanations of Crime: Conflict or Consensus

This paper examines multidisciplinary correlates of delinquency in an attempt to integrate sociological and environmental theories of crime with human developmental and biological explanations of crime. Structural equation models are applied to assess links among biological, psychological, and environmental variables collected prospectively from birth through age 17 on a sample of 800 black children at high risk for learning and behavioral disorders. Results show that for both males and females, aggression and disciplinary problems in school during adolescence are the strongest predictors of repeat offense behavior. Whereas school achievement and family income and stability are also significant predictors of delinquency for males, early physical development is the next strongest predictor for females. Results indicate that some effects on delinquency also vary during different ages. It is suggested that behavioral and learning disorders have both sociological and developmental correlates and that adequate educational resources are necessary to ensure channels of legitimate opportunities for high-risk youths

The Emergence of Emotions

Emotion is conscious experience. It is the affective aspect of consciousness. Emotion arises from sensory stimulation and is typically accompanied by physiological and behavioral changes in the body. Hence an emotion is a complex reaction pattern consisting of three components: a physiological component, a behavioral component, and an experiential (conscious) component. The reactions making up an emotion determine what the emotion will be recognized as. Three processes are involved in generating an emotion: (1) identification of the emotional significance of a sensory stimulus, (2) production of an affective state (emotion), and (3) regulation of the affective state. Two opposing systems in the brain (the reward and punishment systems) establish an affective value or valence (stimulus-reinforcement association) for sensory stimulation. This is process (1), the first step in the generation of an emotion. Development of stimulus-reinforcement associations (affective valence) serves as the basis for emotion expression (process 2), conditioned emotion learning acquisition and expression, memory consolidation, reinforcement-expectations, decision-making, coping responses, and social behavior. The amygdala is critical for the representation of stimulus-reinforcement associations (both reward and punishment-based) for these functions. Three distinct and separate architectural and functional areas of the prefrontal cortex (dorsolateral prefrontal cortex, orbitofrontal cortex, anterior cingulate cortex) are involved in the regulation of emotion (process 3). The regulation of emotion by the prefrontal cortex consists of a positive feedback interaction between the prefrontal cortex and the inferior parietal cortex resulting in the nonlinear emergence of emotion. This positive feedback and nonlinear emergence represents a type of working memory (focal attention) by which perception is reorganized and rerepresented, becoming explicit, functional, and conscious. The explicit emotion states arising may be involved in the production of voluntary new or novel intentional (adaptive) behavior, especially social behavior

Child and family experiences with inborn errors of metabolism: a qualitative interview study with representatives of patient groups

© 2015, The Author(s). Background: Patient-centered health care for children with inborn errors of metabolism (IEM) and their families is important and requires an understanding of patient experiences, needs, and priorities. IEM-specific patient groups have emerged as important voices within these rare disease communities and are uniquely positioned to contribute to this understanding. We conducted qualitative interviews with IEM patient group representatives to increase understanding of patient and family experiences, needs, and priorities and inform patient-centered research and care. Methods: We developed a sampling frame of patient groups representing IEM disease communities from Canada, the United States, and United Kingdom. With consent, we interviewed participants to explore their views on experiences, needs, and outcomes that are most important to children with IEM and their families. We analyzed the data using a qualitative descriptive approach to identify key themes and sub-themes. Results: We interviewed 18 organizational representatives between February 28 and September 17, 2014, representing 16 IEMs and/or disease categories. Twelve participants voluntarily self-identified as parents and/or were themselves patients. Three key themes emerged from the coded data: managing the uncertainty associated with raising and caring for a child with a rare disease; challenges associated with the affected child’s life transitions, and; the collective struggle for improved outcomes and interventions that rare disease communities navigate. Conclusion: Health care providers can support children with IEM and their families by acknowledging and reducing uncertainty, supporting families through children’s life transitions, and contributing to rare disease communities’ progress toward improved interventions, experiences, and outcomes.The study was partially funded by the Rare Disease Foundation (RDF). In-kind support was provided by the Canadian Inherited Metabolic Diseases Research Network (CIMDRN) which is funded by the Canadian Institutes of Health Research (CIHR, grant TR3-119197) and administered by the University of Ottawa