375 research outputs found

    Resonance Production at STAR

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    We present the first measurement of mid-rapidity Ļ•\phi vector meson production in Au+AuAu+Au collisions at RHIC (sNN=130\sqrt{s_{_{NN}}} =130 GeV) from the STAR detector. For the 11% most central collisions, the slope parameter from an exponential fit to the transverse mass distribution is T=T=~379Ā±\pm51(stat) Ā±\pm 45(syst)~MeV, the yield dN/dydN/dy =5.73Ā±=5.73\pm0.37(stat)Ā±\pm 0.57(syst) per event and the ratio NĻ•/Nhāˆ’N_{\phi}/N_{h^-} is found to be 0.021Ā±\pm0.001(stat)Ā±\pm 0.03(syst). We currently place the value of the NĻ•/NKāˆ’N_{\phi}/N_{K^-} ratio between 0.10 and 0.16. The measured ratios NĻ•/Nhāˆ’N_{\phi}/N_{h^-} and NĻ•/NKāˆ’N_{\phi}/N_{K^-}, as well as TT for the Ļ•\phi meson at mid-rapidity do not change for the selected centrality bins.Comment: 8 pages, 7 figures (in eps) talk given at XXXI International Symposium on Multiparticle Dynamics, Sep. 1-7, 2001, Datong China at http://ismd31.ccnu.edu.cn

    Behavioral-Independent Features of Complex Heartbeat Dynamics

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    We test whether the complexity of cardiac interbeat interval time series is simply a consequence of the wide range of scales characterizing human behavior, especially physical activity, by analyzing data taken from healthy adult subjects under three conditions with controls: (i) a ``constant routine'' protocol where physical activity and postural changes are kept to a minimum, (ii) sympathetic blockade, and (iii) parasympathetic blockade. We find that when fluctuations in physical activity and other behavioral modifiers are minimized, a remarkable level of complexity of heartbeat dynamics remains, while for neuroautonomic blockade the multifractal complexity decreases.Comment: 4 pages with 6 eps figures. Latex file. For more details or for downloading the PDF file of the published article see http://polymer.bu.edu/~amaral/Heart.html and http://polymer.bu.edu/~amaral/Multifractal.htm

    Decreased alveolar bone turnover is related to the occurrence of root resorption during experimental tooth movement in dogs

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    Objective:ā€‚ To investigate the relationship between root resorption (RR) and bone turnover in two different types of tooth movement in dogs. Materials and Methods:ā€‚ A total of 16 dogs in two different groups were used. Tooth movement of dog premolars resulted from approximately 200 g of force. Histomorphometric analysis of premolar roots was assessed after 4 and 12 weeks of tooth movement by comparing nonresorptive to resorptive surfaces. Results:ā€‚ Histomorphometric analysis indicated a significant decrease in the bone formation rate in the root resorptive areas, which resulted in decreased bone volume after 12 weeks. The threshold to detect RR in periapical radiographs was about 1.0 mm2. Conclusions:ā€‚ A sustained mechanical load, due to the prolonged stress and strain of continuous mechanics, induces elevated bone metabolic activity, such as the bone turnover (remodeling) and change in bone volume (modeling). Therefore, our data support the hypothesis that increased RR is related to decreased bone formation (turnover) in high stress areas exposed to prolonged orthodontic tooth movement

    Low-energy properties and magnetization plateaus in a 2-leg mixed spin ladder

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    Using the density matrix renormalization group technique we investigate the low-energy properties and the magnetization plateau behavior in a 2-leg mixed spin ladder consisting of a spin-1/2 chain coupled with a spin-1 chain. The calculated results show that the system is in the same universality class as the spin-3/2 chain when the interchain coupling is strongly ferromagnetic, but the similarity between the two systems is less clear under other coupling conditions. We have identified two types of magnetization plateau phases. The calculation of the magnetization distribution on the spin-1/2 and the spin-1 chains on the ladder shows that one plateau phase is related to the partially magnetized valence-bond-solid state, and the other plateau state contains strongly coupled S=1 and s=1/2 spins on the rung.Comment: 6 pages with 8 eps figure

    Decrease in p3-Alcb37 and p3-Alcb40, products of Alcadein b generated by g-secretase cleavages, in aged monkeys and patients with Alzheimerā€™s disease

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    Introduction Neuronal p3-AlcĪ² peptides are generated from the precursor protein Alcadein Ī² (AlcĪ²) through cleavage by Ī±- and Ī³-secretases of the amyloid Ī² (AĪ²) protein precursor (APP). To reveal whether p3-AlcĪ² is involved in Alzheimer\u27s disease (AD) contributes for the development of novel therapy and/or drug targets. Methods We developed new sandwich enzyme-linked immunosorbent assay (sELISA) systems to quantitate levels of p3-AlcĪ² in the cerebrospinal fluid (CSF). Results In monkeys, CSF p3-AlcĪ² decreases with age, and the aging is also accompanied by decreased brain expression of AlcĪ². In humans, CSF p3-AlcĪ² levels decrease to a greater extent in those with AD than in age-matched controls. Subjects carrying presenilin gene mutations show a significantly lower CSF p3-AlcĪ² level. A cell study with an inverse modulator of Ī³-secretase remarkably reduces the generation of p3-AlcĪ²37 while increasing the production of AĪ²42. Discussion Aging decreases the generation of p3-AlcĪ², and further significant decrease of p3-AlcĪ² caused by aberrant Ī³-secretase activity may accelerate pathogenesis in AD

    Decrease in p3-Alcb37 and p3-Alcb40, products of Alcadein b generated by g-secretase cleavages, in aged monkeys and patients with Alzheimerā€™s disease

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    Introduction Neuronal p3-AlcĪ² peptides are generated from the precursor protein Alcadein Ī² (AlcĪ²) through cleavage by Ī±- and Ī³-secretases of the amyloid Ī² (AĪ²) protein precursor (APP). To reveal whether p3-AlcĪ² is involved in Alzheimer\u27s disease (AD) contributes for the development of novel therapy and/or drug targets. Methods We developed new sandwich enzyme-linked immunosorbent assay (sELISA) systems to quantitate levels of p3-AlcĪ² in the cerebrospinal fluid (CSF). Results In monkeys, CSF p3-AlcĪ² decreases with age, and the aging is also accompanied by decreased brain expression of AlcĪ². In humans, CSF p3-AlcĪ² levels decrease to a greater extent in those with AD than in age-matched controls. Subjects carrying presenilin gene mutations show a significantly lower CSF p3-AlcĪ² level. A cell study with an inverse modulator of Ī³-secretase remarkably reduces the generation of p3-AlcĪ²37 while increasing the production of AĪ²42. Discussion Aging decreases the generation of p3-AlcĪ², and further significant decrease of p3-AlcĪ² caused by aberrant Ī³-secretase activity may accelerate pathogenesis in AD
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