8,397 research outputs found

    Data taking strategy for the phase study in ψ′→K+K−\psi^{\prime} \to K^+K^-

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    The study of the relative phase between strong and electromagnetic amplitudes is of great importance for understanding the dynamics of charmonium decays. The information of the phase can be obtained model-independently by fitting the scan data of some special decay channels, one of which is ψ′→K+K−\psi^{\prime} \to K^{+}K^{-}. To find out the optimal data taking strategy for a scan experiment in the measurement of the phase in ψ′→K+K−\psi^{\prime} \to K^{+} K^{-}, the minimization process is analyzed from a theoretical point of view. The result indicates that for one parameter fit, only one data taking point in the vicinity of a resonance peak is sufficient to acquire the optimal precision. Numerical results are obtained by fitting simulated scan data. Besides the results related to the relative phase between strong and electromagnetic amplitudes, the method is extended to analyze the fits of other resonant parameters, such as the mass and the total decay width of ψ′\psi^{\prime}.Comment: 13 pages, 7 figure

    Machine Learning and Portfolio Optimization

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    The portfolio optimization model has limited impact in practice due to estimation issues when applied with real data. To address this, we adapt two machine learning methods, regularization and cross-validation, for portfolio optimization. First, we introduce performance-based regularization (PBR), where the idea is to constrain the sample variances of the estimated portfolio risk and return, which steers the solution towards one associated with less estimation error in the performance. We consider PBR for both mean-variance and mean-CVaR problems. For the mean-variance problem, PBR introduces a quartic polynomial constraint, for which we make two convex approximations: one based on rank-1 approximation and another based on a convex quadratic approximation. The rank-1 approximation PBR adds a bias to the optimal allocation, and the convex quadratic approximation PBR shrinks the sample covariance matrix. For the mean-CVaR problem, the PBR model is a combinatorial optimization problem, but we prove its convex relaxation, a QCQP, is essentially tight. We show that the PBR models can be cast as robust optimization problems with novel uncertainty sets and establish asymptotic optimality of both Sample Average Approximation (SAA) and PBR solutions and the corresponding efficient frontiers. To calibrate the right hand sides of the PBR constraints, we develop new, performance-based k-fold cross-validation algorithms. Using these algorithms, we carry out an extensive empirical investigation of PBR against SAA, as well as L1 and L2 regularizations and the equally-weighted portfolio. We find that PBR dominates all other benchmarks for two out of three of Fama-French data sets

    Versatile Atomic Magnetometry Assisted by Bayesian Inference

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    Quantum sensors typically translate external fields into a periodic response whose frequency is then determined by analyses performed in Fourier space. This allows for a linear inference of the parameters that characterize external signals. In practice, however, quantum sensors are able to detect fields only in a narrow range of amplitudes and frequencies. A departure from this range, as well as the presence of significant noise sources and short detection times, lead to a loss of the linear relationship between the response of the sensor and the target field, thus limiting the working regime of the sensor. Here we address these challenges by means of a Bayesian inference approach that is tolerant to strong deviations from desired periodic responses of the sensor and is able to provide reliable estimates even with a very limited number of measurements. We demonstrate our method for an 171^{171}Yb+^{+} trapped-ion quantum sensor but stress the general applicability of this approach to different systems.Comment: 5+14 pages, 3+9 figures. Comments are welcome

    Information loss in local dissipation environments

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    The sensitivity of entanglement to the thermal and squeezed reservoirs' parameters is investigated regarding entanglement decay and what is called sudden-death of entanglement, ESD, for a system of two qubit pairs. The dynamics of information is investigated by means of the information disturbance and exchange information. We show that for squeezed reservoir, we can keep both of the entanglement and information survival for a long time. The sudden death of information is seen in the case of thermal reservoir

    Search for D to phi l nu and measurement of the branching fraction for D to phi pi

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    Using a data sample of integrated luminosity of about 33 pb−1^{-1} collected around 3.773 GeV with the BESII detector at the BEPC collider, the semileptonic decays D+→ϕe+νeD^+ \to \phi e ^+\nu_e, D+→ϕμ+νμD^+ \to \phi \mu^+\nu_\mu and the hadronic decay D+→ϕπ+D^+ \to \phi \pi^+ are studied. The upper limits of the branching fractions are set to be BF(D+→ϕe+νe)<BF(D^+ \to \phi e ^+\nu_e) < 2.01% and BF(D+→ϕμ+νμ)<BF(D^+ \to \phi \mu^+ \nu_\mu) < 2.04% at the 90% confidence level. The ratio of the branching fractions for D+→ϕπ+D^+ \to \phi \pi^+ relative to D+→K−π+π+D^+ \to K^-\pi^+\pi^+ is measured to be 0.057±0.011±0.0030.057 \pm 0.011 \pm 0.003. In addition, the branching fraction for D+→ϕπ+D^+ \to \phi \pi^+ is obtained to be (5.2±1.0±0.4)×10−3(5.2 \pm 1.0 \pm 0.4) \times 10^{-3}.Comment: 6 pages, 5 figures, to be published in Eur.Phys.J.

    Measurements of branching fractions for inclusive K0~/K0 and K*(892)+- decays of neutral and charged D mesons

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    Using the data sample of about 33 pb-1 collected at and around 3.773 GeV with the BES-II detector at the BEPC collider, we have studied inclusive K0~/K0 and K*(892)+- decays of D0 and D+ mesons. The branching fractions for the inclusive K0~/K0 and K*(892)- decays are measured to be BF(D0 to K0~/K0 X)=(47.6+-4.8+-3.0)%, BF(D+ to K0~/K0 X)=(60.5+-5.5+-3.3)%, BF(D0 to K*- X)=(15.3+- 8.3+- 1.9)% and BF(D+ to K*- X)=(5.7+- 5.2+- 0.7)%. The upper limits of the branching fractions for the inclusive K*(892)+ decays are set to be BF(D0 to K*+ X)<3.6% and BF(D+ to K*+ X) <20.3% at 90% confidence level

    Perceptual integration for qualitatively different 3-D cues in the human brain.

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    The visual system's flexibility in estimating depth is remarkable: We readily perceive 3-D structure under diverse conditions from the seemingly random dots of a "magic eye" stereogram to the aesthetically beautiful, but obviously flat, canvasses of the Old Masters. Yet, 3-D perception is often enhanced when different cues specify the same depth. This perceptual process is understood as Bayesian inference that improves sensory estimates. Despite considerable behavioral support for this theory, insights into the cortical circuits involved are limited. Moreover, extant work tested quantitatively similar cues, reducing some of the challenges associated with integrating computationally and qualitatively different signals. Here we address this challenge by measuring fMRI responses to depth structures defined by shading, binocular disparity, and their combination. We quantified information about depth configurations (convex "bumps" vs. concave "dimples") in different visual cortical areas using pattern classification analysis. We found that fMRI responses in dorsal visual area V3B/KO were more discriminable when disparity and shading concurrently signaled depth, in line with the predictions of cue integration. Importantly, by relating fMRI and psychophysical tests of integration, we observed a close association between depth judgments and activity in this area. Finally, using a cross-cue transfer test, we found that fMRI responses evoked by one cue afford classification of responses evoked by the other. This reveals a generalized depth representation in dorsal visual cortex that combines qualitatively different information in line with 3-D perception

    Direct Measurements of the Branching Fractions for Inclusive K±K^\pm and Inclusive Semileptonic Decays of D+D^+ and D0D^0 Mesons

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    With singly-tagged Dˉ\bar D samples selected from the data collected at and around 3.773 GeV with the BESII detector at the BEPC collider, we have measured the branching fractions for the inclusive K±K^\pm decays of D+D^+ and D0D^0 mesons, which are BF(D+→K−X)=(24.7±1.3±1.2)BF(D^+\to K^-X) = (24.7 \pm 1.3 \pm 1.2)%, BF(D+→K+X)=(6.1±0.9±0.4)BF(D^+\to K^+X) = (6.1 \pm 0.9 \pm 0.4) %, BF(D0→K−X)=(57.8±1.6±3.2)BF(D^0\to K^-X) = (57.8 \pm 1.6 \pm 3.2) % and BF(D0→K+X)=(3.5±0.7±0.3)BF(D^0\to K^+X) = (3.5 \pm 0.7 \pm 0.3) %, respectively. We have also measured the branching fractions for the inclusive semileptonic decays of D+D^+ and D0D^0 mesons to be BF(D+→e+X)=(15.2±0.9±0.8)BF(D^+ \to e^+ X)=(15.2 \pm 0.9 \pm 0.8)% and BF(D0→e+X)=(6.3±0.7±0.4)BF(D^0 \to e^+ X) =(6.3 \pm 0.7 \pm 0.4) %. These yield the ratio of their partial widths to be Γ(D+→e+X)/Γ(D0→e+X)=0.95±0.12±0.07\Gamma(D^+ \to e^+X)/\Gamma(D^0 \to e^+X)=0.95 \pm 0.12 \pm 0.07.Comment: 6 pages, 5 figure

    Lack of association between estrogen receptor β dinucleotide repeat polymorphism and autoimmune thyroid diseases in Japanese patients

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    BACKGROUND: The autoimmune thyroid diseases (AITDs), such as Graves' disease (GD) and Hashimoto's thyroiditis (HT), appear to develop as a result of complex interactions between predisposing genes and environmental triggers. Susceptibility to AITDs is conferred by genes in the human leukocyte antigen (HLA) and genes unlinked to HLA, including the CTLA-4 gene. Recently, estrogen receptor (ER) β, located at human chromosome 14q23-24.1, was identifed. We analyzed a dinucleotide (CA)n repeat polymorphism located in the flanking region of ERβ gene in patients with AITDs and in normal subjects. High heterozygosity makes this polymorphism a useful marker in the genetic study of disorders affecting female endocrine systems. We also correlated a ERβ gene microsatellite polymorphism with bone mineral density (BMD) in the distal radius and biochemical markers of bone turnover in patients with GD in remission. RESULTS: Fourteen different alleles were found in 133 patients with GD, 114 patients with HT, and 179 controls subjects. The various alleles were designated as allele(*)1 through allele(*)14 according to the number of the repeats, from 18 to 30. There was no significant difference in the distributions of ERβ alleles between patient groups and controls. Although recent study demonstrated a significant relation between a allele(*)9 in the ERβ gene and BMD in postmenopausal Japanese women, there were no statistically significant interaction between this allele and BMD in the distal radius, nor biochemical markers in patients with GD in remission. CONCLUSIONS: The present results do not support an association between the ERβ microsatellite marker and AITD in the Japanese population. We also suggest that the ERβ microsatellite polymorphism has at most a minor pathogenic importance in predicting the risk of osteoporosis as a complication of GD
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