669 research outputs found
A system of difference equations with elliptic coefficients and Bethe vectors
An elliptic analogue of the deformed Knizhnik-Zamolodchikov equations is
introduced. A solution is given in the form of a Jackson-type integral of Bethe
vectors of the XYZ-type spin chains.Comment: 20 pages, AMS-LaTeX ver.1.1 (amssymb), 15 figures in LaTeX picture
environment
Construction of some missing eigenvectors of the XYZ spin chain at the discrete coupling constants and the exponentially large spectral degeneracy of the transfer matrix
We discuss an algebraic method for constructing eigenvectors of the transfer
matrix of the eight vertex model at the discrete coupling parameters. We
consider the algebraic Bethe ansatz of the elliptic quantum group for the case where the parameter satisfies for arbitrary integers , and . When or
is odd, the eigenvectors thus obtained have not been discussed previously.
Furthermore, we construct a family of degenerate eigenvectors of the XYZ spin
chain, some of which are shown to be related to the loop algebra
symmetry of the XXZ spin chain. We show that the dimension of some degenerate
eigenspace of the XYZ spin chain on sites is given by , if
is an even integer. The construction of eigenvectors of the transfer matrices
of some related IRF models is also discussed.Comment: 19 pages, no figure (revisd version with three appendices
Eigenvalues of Ruijsenaars-Schneider models associated with root system in Bethe ansatz formalism
Ruijsenaars-Schneider models associated with root system with a
discrete coupling constant are studied. The eigenvalues of the Hamiltonian are
givein in terms of the Bethe ansatz formulas. Taking the "non-relativistic"
limit, we obtain the spectrum of the corresponding Calogero-Moser systems in
the third formulas of Felder et al [20].Comment: Latex file, 25 page
-analogue of modified KP hierarchy and its quasi-classical limit
A -analogue of the tau function of the modified KP hierarchy is defined by
a change of independent variables. This tau function satisfies a system of
bilinear -difference equations. These bilinear equations are translated to
the language of wave functions, which turn out to satisfy a system of linear
-difference equations. These linear -difference equations are used to
formulate the Lax formalism and the description of quasi-classical limit. These
results can be generalized to a -analogue of the Toda hierarchy. The results
on the -analogue of the Toda hierarchy might have an application to the
random partition calculus in gauge theories and topological strings.Comment: latex2e, a4 paper 15 pages, no figure; (v2) a few references are
adde
Explorations of the Extended ncKP Hierarchy
A recently obtained extension (xncKP) of the Moyal-deformed KP hierarchy
(ncKP hierarchy) by a set of evolution equations in the Moyal-deformation
parameters is further explored. Formulae are derived to compute these equations
efficiently. Reductions of the xncKP hierarchy are treated, in particular to
the extended ncKdV and ncBoussinesq hierarchies. Furthermore, a good part of
the Sato formalism for the KP hierarchy is carried over to the generalized
framework. In particular, the well-known bilinear identity theorem for the KP
hierarchy, expressed in terms of the (formal) Baker-Akhiezer function, extends
to the xncKP hierarchy. Moreover, it is demonstrated that N-soliton solutions
of the ncKP equation are also solutions of the first few deformation equations.
This is shown to be related to the existence of certain families of algebraic
identities.Comment: 34 pages, correction of typos in (7.2) and (7.5
WNT signalling in prostate cancer
Genome sequencing and gene expression analyses of prostate tumours have highlighted the potential importance of genetic and epigenetic changes observed in WNT signalling pathway components in prostate tumours-particularly in the development of castration-resistant prostate cancer. WNT signalling is also important in the prostate tumour microenvironment, in which WNT proteins secreted by the tumour stroma promote resistance to therapy, and in prostate cancer stem or progenitor cells, in which WNT-β-catenin signals promote self-renewal or expansion. Preclinical studies have demonstrated the potential of inhibitors that target WNT receptor complexes at the cell membrane or that block the interaction of β-catenin with lymphoid enhancer-binding factor 1 and the androgen receptor, in preventing prostate cancer progression. Some WNT signalling inhibitors are in phase I trials, but they have yet to be tested in patients with prostate cancer
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