Non Degenerate Dual Atomic Parametric Amplifier: Entangled Atomic Fields

In this paper, we investigate the dynamics of two coupled quantum degenerate atomic fields (BEC) interacting with two classical optical fields in the nonlinear atom optics regime. Two photon interaction produces entangled atom-atom pairs which exhibit nonclassical correlations. Since the system involves the creation of two correlated atom pairs, we call it the nondegenerate dual atomic parametric amplifier.Comment: 5 figure

Quantitative Constraints on the Transport Properties of Hot Partonic Matter from Semi-Inclusive Single High Transverse Momentum Pion Suppression in Au+Au collisions at sqrt(s_NN) = 200 GeV

The PHENIX experiment has measured the suppression of semi-inclusive single high transverse momentum pi^0's in Au+Au collisions at sqrt(s_NN) = 200 GeV. The present understanding of this suppression is in terms of energy-loss of the parent (fragmenting) parton in a dense color-charge medium. We have performed a quantitative comparison between various parton energy-loss models and our experimental data. The statistical point-to-point uncorrelated as well as correlated systematic uncertainties are taken into account in the comparison. We detail this methodology and the resulting constraint on the model parameters, such as the initial color-charge density dN^g/dy, the medium transport coefficient , or the initial energy-loss parameter epsilon_0. We find that high transverse momentum pi^0 suppression in Au+Au collisions has sufficient precision to constrain these model dependent parameters at the +/1 20%-25% (one standard deviation) level. These constraints include only the experimental uncertainties, and further studies are needed to compute the corresponding theoretical uncertainties.Comment: 422 authors, 13 pages text, RevTeX-4, 9 figures, 2 tables. This version is updated with changes made during the review process and is now the same as what was published in Physical Review C. Plain text data tables for the points plotted in figures for this and previous PHENIX publications are publicly available at http://www.phenix.bnl.gov/papers.htm

Effect of dietary honey on intestinal microflora and toxicity of mycotoxins in mice

BACKGROUND: Bee honey is a functional food which has a unique composition, antimicrobial properties and bifidogenic effect. In order to assess whether honey can inhibit the toxic effect of mycotoxins, the present study was undertaken. METHODS: Production of biomass and toxins by Aspergillus parasiticus and Aspergillus ochraceus were followed in media without and with honey. Although aflatoxins and ochratoxin A. were administrated to male Swiss albino mice up to 1 μg and 10 ng/kg body weight/day respectively. The experimental animals were fed diets without our with 10% honey for two months. The changes in colonic probiotic bacteria, determintal colon enzyme glucuronidases, and genotoxicity were followed. RESULTS: Addition of 32% in its media increased the biomass of A parasiticus, while the biomass of A. ochraceus decreased and Ochratoxin A. was not produced. When the honey was added at the ratio of 32 and 48% in the medium. No relationship was found between mycelium weight and production of mycotoxins. Oral administration of aflatoxins (mixture of B(1), B(2), G(1) and G(2)) and Ochratoxin A. induced structural and numerical chromosomal aberrations in bone marrow and germ cells of male mice, whereas, honey treatment reduced the genotoxicity of mycotoxins. Also both toxins induced histopathological changes in liver and kidney. Feeding on diet supplemented with honey improved the histopathological changes in case of aflatoxin group, but not in the case of ochratoxin A. group (except of kidney in two cases). No significant differences were found in the activity of colon β-glucuronidase between group fed diet with or without honey. On the other hand, the colon bifido bacteria and lactobacilli counts were increased markedly in group receiving diet supplemented with honey. CONCLUSION: Substituting sugars with honey in processed food can inhibit the harmful and genotoxic effects of mycotoxins, and improve the gut microflora