Review of: Rationality, Allocation, and Reproduction by Vivian Walsh

This article is a review of the book Rationality, Allocation, and Reproduction by Vivian Walsh

Effort and catch estimates for northern and central California marine recreational fisheries, 1981-1986

Nearly 200 species of finfish are taken by the marine recreational fishery along the northern and central California coast. This data report provides estimates of total effort, total catch, and fishery demographics for the years 1981 through 1986 for that fishery. Catch estimate data are presented by number and weight of species, by disposition of the fish caught (e.g. kept or thrown back), by type of access and fishing gear used, and by geographic zone. (311pp.

Acción : diario de Teruel y su provincia: Año III Número 549 - (02/09/34)

In circumstances of limited water supply, the stability of grain yield in sorghum may be improved by manipulating the interaction between plant spacing and crop maturity. The plant density which maximises grain yield over environments may also be influenced by differences in compensatory capacity among hybrids of the same maturity category. Consequently, variation among 8 sorghum hybrids in the response of grain yield to plant density was examined at 5 locations.When panicle number was used as a covariate, the interaction between hybrid, density, and site was significant (

Spanish Colonial Documents Pertaining to Mission Santa Cruz de San Saba (41MN23), Menard County, Texas

The Texas Department of Transportation (TxDOT) is widening a 5-mile-long section of FM 2092 west of Menard in Menard County, Texas. The highway passes immediately south of the site of the Mission Santa Cruz de San Sabá (41MN23). Built in 1757 and destroyed in 1758, the mission is a time capsule of Spanish colonial archeology in the northern frontier of New Spain, along with the related Presidio de las Amarillas (41MN1, popularly known as Presidio San Sabá), which was occupied from 1757 to 1768. The presidio location has long been known, but researchers did not rediscover the mission site until 1993. Texas Tech University conducted intensive archeological investigations at the mission site from 1993 through 1997. In the spring of 2006, the Texas Department of Transportation (TxDOT) contracted Prewitt and Associates, Inc., to conduct an archeological survey of this portion of FM 2092. The work included archeological testing of the portion of the mission site that extended into the road right of way. Intact and significant mission archeological remains were found, and data recovery investigations were subsequently undertaken in 2006–2007. In conjunction with the FM 2092 survey effort, TxDOT sought to locate, transcribe, and translate previously unpublished or unknown Spanish colonial documents that might add to the story of the mission and presidio and the interpretation of the archeological remains of Mission San Sabá. Many Spanish documents pertaining to the mission and presidio have been translated and made available to researchers since the mid-twentieth century, but it is certain that additional documents exist and may offer further information on the lifeways and politics of those who occupied the mission. For this study, 10 previously unpublished Spanish documents—those considered to be most relevant to Mission San Sabá—were selected and then transcribed and translated in their entirety. Each of these documents is presented here in three forms: a facsimile of the original handwritten Spanish text, a complete Spanish transcription of the document, and a complete English translation of the document

Hydrocarbon-Water Solubilities At Elevated Temperatures Near the Three-Phase Equilibrium Pressure

The purpose of this work was to establish a reliable experimental apparatus and procedures for measuring liquid-liquid equilibrium data at ambient and elevated temperatures and pressures. A continuous flow apparatus has been designed and constructed to obtain mutual solubilites at temperatures from ambient to 623 K and pressures up to 13.8 MPa. The accuracy of the apparatus and methods used was determined by measurements on the well-documented benzene-water system. Mutual solubility data have also been measured for the aqueous binary systems involving decane and I-hexene at temperatures up to the three-phase critical end point

Adjusting storm-influenced wind observations for barotropic hurricane track prediction.

Thesis. 1978. M.S.--Massachusetts Institute of Technology. Dept. of Meteorology.Microfiche copy available in Archives and Science.Bibliography : leaves 64-65.M.S

Neuromyelitis optica pathogenesis and aquaporin 4

Neuromyelitis optica (NMO) is a severe, debilitating human disease that predominantly features immunopathology in the optic nerves and the spinal cord. An IgG1 autoantibody (NMO-IgG) that binds aquaporin 4 (AQP4) has been identified in the sera of a significant number of NMO patients, as well as in patients with two related neurologic conditions, bilateral optic neuritis (ON), and longitudinal extensive transverse myelitis (LETM), that are generally considered to lie within the NMO spectrum of diseases. NMO-IgG is not the only autoantibody found in NMO patient sera, but the correlation of pathology in central nervous system (CNS) with tissues that normally express high levels of AQP4 suggests NMO-IgG might be pathogenic. If this is the case, it is important to identify and understand the mechanism(s) whereby an immune response is induced against AQP4. This review focuses on open questions about the "events" that need to be understood to determine if AQP4 and NMO-IgG are involved in the pathogenesis of NMO. These questions include: 1) How might AQP4-specific T and B cells be primed by either CNS AQP4 or peripheral pools of AQP4? 2) Do the different AQP4-expressing tissues and perhaps the membrane structural organization of AQP4 influence NMO-IgG binding efficacy and thus pathogenesis? 3) Does prior infection, genetic predisposition, or underlying immune dysregulation contribute to a confluence of events which lead to NMO in select individuals? A small animal model of NMO is essential to demonstrate whether AQP4 is indeed the incipient autoantigen capable of inducing NMO-IgG formation and NMO. If the NMO model is consistent with the human disease, it can be used to examine how changes in AQP4 expression and blood-brain barrier (BBB) integrity, both of which can be regulated by CNS inflammation, contribute to inductive events for anti-AQP4-specific immune response. In this review, we identify reagents and experimental questions that need to be developed and addressed to enhance our understanding of the pathogenesis of NMO. Finally, dysregulation of tolerance associated with autoimmune disease appears to have a role in NMO. Animal models would allow manipulation of hormone levels, B cell growth factors, and other elements known to increase the penetrance of autoimmune disease. Thus an AQP4 animal model would provide a means to manipulate events which are now associated with NMO and thus demonstrate what set of events or multiplicity of events can push the anti-AQP4 response to be pathogenic

Neuromyelitis Optica Pathogenesis and Aquaporin 4

Neuromyelitis optica (NMO) is a severe, debilitating human disease that predominantly features immunopathology in the optic nerves and the spinal cord. An IgG1 autoantibody (NMO-IgG) that binds aquaporin 4 (AQP4) has been identified in the sera of a significant number of NMO patients, as well as in patients with two related neurologic conditions, bilateral optic neuritis (ON), and longitudinal extensive transverse myelitis (LETM), that are generally considered to lie within the NMO spectrum of diseases. NMO-IgG is not the only autoantibody found in NMO patient sera, but the correlation of pathology in central nervous system (CNS) with tissues that normally express high levels of AQP4 suggests NMO-IgG might be pathogenic. If this is the case, it is important to identify and understand the mechanism(s) whereby an immune response is induced against AQP4. This review focuses on open questions about the events that need to be understood to determine if AQP4 and NMO-IgG are involved in the pathogenesis of NMO. These questions include: 1) How might AQP4-specific T and B cells be primed by either CNS AQP4 or peripheral pools of AQP4? 2) Do the different AQP4-expressing tissues and perhaps the membrane structural organization of AQP4 influence NMO-IgG binding efficacy and thus pathogenesis? 3) Does prior infection, genetic predisposition, or underlying immune dysregulation contribute to a confluence of events which lead to NMO in select individuals? A small animal model of NMO is essential to demonstrate whether AQP4 is indeed the incipient autoantigen capable of inducing NMO-IgG formation and NMO. If the NMO model is consistent with the human disease, it can be used to examine how changes in AQP4 expression and blood-brain barrier (BBB) integrity, both of which can be regulated by CNS inflammation, contribute to inductive events for anti-AQP4-specific immune response. In this review, we identify reagents and experimental questions that need to be developed and addressed to enhance our understanding of the pathogenesis of NMO. Finally, dysregulation of tolerance associated with autoimmune disease appears to have a role in NMO. Animal models would allow manipulation of hormone levels, B cell growth factors, and other elements known to increase the penetrance of autoimmune disease. Thus an AQP4 animal model would provide a means to manipulate events which are now associated with NMO and thus demonstrate what set of events or multiplicity of events can push the anti-AQP4 response to be pathogenic