Exploration of Lorentz violation in neutral-kaon decay

The KLOE collaboration recently reported bounds on the directional dependence of the lifetime of the short-lived neutral kaon $K^0_S$ with respect to the dipole anisotropy of the cosmic microwave background. We interpret their results in an effective field theory framework developed to probe the violation of Lorentz invariance in the weak interaction and previously applied to semileptonic processes, in particular $\beta$ decay. In this approach a general Lorentz-violating tensor $\chi^{\mu\nu}$ is added to the standard propagator of the $W$ boson. We perform an exploratory study of the prospects to search for Lorentz violation in nonleptonic decays. For the kaon, we find that the sensitivity to Lorentz violation is limited by the velocity of the kaons and by the extent to which hadronic effects can be calculated. In a simple model we derive the $K^0_S$ decay rate and calculate the asymmetry for the lifetime. Using the KLOE data, limits on the values of $\chi^{\mu\nu}$ are determined.Comment: accepted for publication in Physics Letters

The orbits of subdwarf-B + main-sequence binaries. II. Three eccentric systems; BD+29 3070, BD +34 1543 and Feige 87

The predicted orbital-period distribution of the subdwarf-B (sdB) population is bi-modal with a peak at short ( 250 days) periods. Observationally, many short-period sdB systems are known, but the predicted long period peak is missing as orbits have only been determined for a few long-period systems. As these predictions are based on poorly understood binary-interaction processes, it is of prime importance to confront the predictions with reliable observational data. We therefore initiated a monitoring program to find and characterize long-period sdB stars. In this paper we aim to determine the orbital parameters of the three long-period sdB+MS binaries BD+29 3070, BD+34 1543 and Feige 87, to constrain their absolute dimensions and the physical parameters of the components. High-resolution spectroscopic time series were obtained with HERMES at the Mercator telescope on La Palma, and analyzed to determine the radial velocities of both the sdB and MS components. Photometry from the literature was used to construct the spectral-energy distribution (SED) of the binaries. Atmosphere models were used to fit these SEDs and to determine the surface gravities and temperatures of both components of all systems. Spectral analysis was used to check the results of the SEDs. An orbital period of 1283 +- 63 d, a mass ratio of q = 0.39 +- 0.04 and a significant non-zero eccentricity of e = 0.15 +- 0.01 were found for BD+29 3070. For BD+34 1543 we determined P = 972 +- 2 d, q = 0.57 +- 0.01 and again a clear non-zero eccentricity of e = 0.16 +- 0.01. Last, for Feige 87 we found P = 936 +- 2 d, q = 0.55 +- 0.01 and e = 0.11 +- 0.01. BD+29 3070, BD+34 1543 and Feige 87 are long period sdB + MS binaries on clearly eccentric orbits. These results are in conflict with the predictions of stable Roche-lobe overflow models.Comment: 15 pages, 6 figures, Accepted by A&

Scattering Lens Resolves sub-100 nm Structures with Visible Light

The smallest structures that conventional lenses are able to optically resolve are of the order of 200 nm. We introduce a new type of lens that exploits multiple scattering of light to generate a scanning nano-sized optical focus. With an experimental realization of this lens in gallium phosphide we have succeeded to image gold nanoparticles at 97 nm optical resolution. Our work is the first lens that provides a resolution in the nanometer regime at visible wavelengths.Comment: 4 pages, 3 figure

Protein Homeostasis in Amyotrophic Lateral Sclerosis: Therapeutic Opportunities?

Protein homeostasis (proteostasis), the correct balance between production and degradation of proteins, is essential for the health and survival of cells. Proteostasis requires an intricate network of protein quality control pathways (the proteostasis network) that work to prevent protein aggregation and maintain proteome health throughout the lifespan of the cell. Collapse of proteostasis has been implicated in the etiology of a number of neurodegenerative diseases, including amyotrophic lateral sclerosis (ALS), the most common adult onset motor neuron disorder. Here, we review the evidence linking dysfunctional proteostasis to the etiology of ALS and discuss how ALS-associated insults affect the proteostasis network. Finally, we discuss the potential therapeutic benefit of proteostasis network modulation in ALS