Supplemental Security Income: Calculating the Impact of Earnings on Benefits

The purpose of this guide is to educate New Yorkers with disabilities about the impact of earnings on Supplemental Security Income benefits

Medicaid & Work: Keeping your Medicaid While You Work

The purpose of this guide is to educate New Yorkers with disabilities who receive SSI about how they can go to work and keep their Medicaid

Ticket to Work: Choosing the Right Employment Network

The purpose of this guide is to educate New Yorkers with disabilities who have a Ticket to Work from the Social Security Administration on how to secure the services and supports to go to work by choosing an Employment Network

The “New” Ticket to Work and Self-Sufficiency Program: Enhancing Economic Self-Sufficiency of Beneficiaries through Work Opportunities and Public/Private Partnership

Discusses the history of the Social Security Administration’s Vocational Rehabilitation and describes the intents and functioning of the Ticket to Work and work Incentives Improvement Act of 1999. This publication is based on federal Social Security and Supplemental Security Income (SSI) laws, regulations and policy. Following Sections I and II pertaining to historical context and evolution of SSA and the Ticket, information presented regarding the operations and structure of the Ticket to Work and Self-Sufficiency Program is based exclusively on the new 2008 regulations

Discovery of a Galaxy Cluster in the Foreground of the Wide-Separation Quasar Pair UM425

We report the discovery of a cluster of galaxies in the field of UM425, a pair of quasars separated by 6.5arcsec. Based on this finding, we revisit the long-standing question of whether this quasar pair is a binary quasar or a wide-separation lens. Previous work has shown that both quasars are at z=1.465 and show broad absorption lines. No evidence for a lensing galaxy has been found between the quasars, but there were two hints of a foreground cluster: diffuse X-ray emission observed with Chandra, and an excess of faint galaxies observed with the Hubble Space Telescope. Here we show, via VLT spectroscopy, that there is a spike in the redshift histogram of galaxies at z=0.77. We estimate the chance of finding a random velocity structure of such significance to be about 5%, and thereby interpret the diffuse X-ray emission as originating from z=0.77, rather than the quasar redshift. The mass of the cluster, as estimated from either the velocity dispersion of the z=0.77 galaxies or the X-ray luminosity of the diffuse emission, would be consistent with the theoretical mass required for gravitational lensing. The positional offset between the X-ray centroid and the expected location of the mass centroid is about 40kpc, which is not too different from offsets observed in lower redshift clusters. However, UM425 would be an unusual gravitational lens, by virtue of the absence of a bright primary lensing galaxy. Unless the mass-to-light ratio of the galaxy is at least 80 times larger than usual, the lensing hypothesis requires that the galaxy group or cluster plays a uniquely important role in producing the observed deflections. Based on observations performed with the Very Large Telescope at the European Southern Observatory, Paranal, Chile.Comment: 12 pages, accepted by ApJ 2005, May 1

The CC chemokine ligand 3 regulates CD11c+CD11b+CD8alpha- dendritic cell maturation and activation following viral infection of the central nervous system: implications for a role in T cell activation.

The role of CC chemokine ligand 3 (CCL3) in activation of dendritic cells (DCs) following mouse hepatitis virus (MHV) infection of the central nervous system (CNS) was examined. The results indicate that CCL3 participates in an effective host response to MHV infection by contributing to CD11c+CD11b+CD8alpha- DC maturation, activation, and migration to cervical lymph nodes (CLN). Diminished CD8alpha- DC activation correlated with reduced IFN-gamma expression by virus-specific T cells accompanied by increased IL-10 production suggesting that CCL3 contributes to an effective host response to viral infection by enhancing the T cell activation potential of DC