Autoignition characteristics of no. 2 diesel fuel

Parametric tests to map the ignition delay characteristics were conducted at pressures of 3, 4, and 5 atm, inlet air temperatures up to 1150 K and fuel air equivalence ratios ranging from 0.2 to 1.0. Ignition delay times in the range of 6 msec to 60 msec at freestream flow velocities ranging from 10 m/sec to 40 m/sec were obtained. The ignition delay times appeared to correlate with the inverse of pressure and the inverse exponent of temperature

Autoignition characteristics of aircraft-type fuels

The ignition delay characteristics of Jet A, JP 4, no. 2 diesel, cetane and an experimental referee broad specification (ERBS) fuel in air at inlet temperatures up to 1000 K, pressures of 10, 15, 20, 25 and 30 atm, and fuel air equivalence ratios of 0.3, 0.5, 0.7 and 1.0 were mapped. Ignition delay times in the range of 1 to 50 msec at freestream flow velocities ranging from 20 to 100 m/sec were obtained using a continuous flow test apparatus which permitted independent variation and evaluation of the effect of temperature, pressure, flow rate, and fuel/air ratio. The ignition delay times for all fuels tested appeared to correlate with the inverse of pressure and the inverse exponent of temperature. With the exception of pure cetane, which had the shortest ignition delay times, the differences between the fuels tested did not appear to be significant. The apparent global activation energies for the typical gas turbine fuels ranged from 38 to 40 kcal/mole, while the activation energy determined for cetane was 50 kcal/mole. In addition, the data indicate that for lean mixtures, ignition delay times decrease with increasing equivalence ratio. It was also noted that physical (apparatus dependent) phenomena, such as mixing (i.e., length and number of injection sites) and airstream cooling (due to fuel heating, vaporization and convective heat loss) can have an important effect on the ignition delay

Case Study: Double J Dairy

The case presents the history and current operations of the Double J Dairy, a large scale (milking about 4,300 cows) dairy farm in the Central Valley of California. The case discusses continuing issues with water, labor and environmental regulations

Are associations between the perceived home and neighbourhood environment and children′s physical activity and sedentary behaviour moderated by urban/rural location?

Abstract not availableJo Salmon, Jenny Veitch, Gavin Abbott, Mai ChinAPaw, Johannes J.Brug, Saskia J. teVelde, Verity Cleland, Clare Hume, David Crawford, Kylie Bal

Migraine in women: the role of hormones and their impact on vascular diseases

Migraine is a predominantly female disorder. Menarche, menstruation, pregnancy, and menopause, and also the use of hormonal contraceptives and hormone replacement treatment may influence migraine occurrence. Migraine usually starts after menarche, occurs more frequently in the days just before or during menstruation, and ameliorates during pregnancy and menopause. Those variations are mediated by fluctuation of estrogen levels through their influence on cellular excitability or cerebral vasculature. Moreover, administration of exogenous hormones may cause worsening of migraine as may expose migrainous women to an increased risk of vascular disease. In fact, migraine with aura represents a risk factor for stroke, cardiac disease, and vascular mortality. Studies have shown that administration of combined oral contraceptives to migraineurs may further increase the risk for ischemic stroke. Consequently, in women suffering from migraine with aura caution should be deserved when prescribing combined oral contraceptives

Zwolinske & Duke in Lab

We studied the in vivo effects of 4-amidinoindan-1-one 2'-amidinohydrazone (CGP 48664A), alpha-difluoromethylornithine (DFMO) and a combination of CGP 48654A-DFMO on tumor growth, cell-cycle phase distribution and polyamine contents. DBA-2, mice were inoculated i.p. with 10(5) L1210 cells on day 0, treated i.p. on days 1-4 and killed on day 5. As compared to controls, CGP 48664A, DFMO and the CGP 48664A-DFMO combination reduced L1210 cell numbers by 33, 43 and 85%, respectively. CGP 48664A did not affect cell-cycle phase distribution. DFMO and the CGP 48664A-DFMO combination caused a moderate and a heavy accumulation in G(0)/G(1)- and G(2)/M-phases, respectively. Compared with controls, the CGP 48664A-DFMO combination reduced putrescine, spermidine and total polyamines, but did not affect spermine. Compared with CGP 48664A, the CGP 48664A-DFMO combination caused lower putrescine and total polyamines, higher spermine, but no change in spermidine. Compared with DFMO, the CGP 48664A-DFMO combination caused higher putrescine and spermidine, lower spermine, but no change in total polyamine levels. We conclude that CGP 48664A potentiates the cystostatic effect of DFMO in vivo. The resulting growth inhibition is accompanied by an accumulation in G(0)/G(1)- and G(2)/M-phases and a reduction of putrescine and spermidine. The data suggest that perturbed polyamine composition rather than reduced spermidine or total polyamine pool size causes a profound growth inhibition. (C) 1995 Wiley-Liss, Inc