12,464 research outputs found
Implementing the three-particle quantization condition including higher partial waves
We present an implementation of the relativistic three-particle quantization
condition including both - and -wave two-particle channels. For this, we
develop a systematic expansion about threshold of the three-particle
divergence-free K matrix, , which is a
generalization of the effective range expansion of the two-particle K matrix,
. Relativistic invariance plays an important role in this
expansion. We find that -wave two-particle channels enter first at quadratic
order. We explain how to implement the resulting multichannel quantization
condition, and present several examples of its application. We derive the
leading dependence of the threshold three-particle state on the two-particle
-wave scattering amplitude, and use this to test our implementation. We show
how strong two-particle -wave interactions can lead to significant effects
on the finite-volume three-particle spectrum, including the possibility of a
generalized three-particle Efimov-like bound state. We also explore the
application to the system, which is accessible to lattice QCD
simulations, where we study the sensitivity of the spectrum to the components
of . Finally, we investigate the circumstances
under which the quantization condition has unphysical solutions.Comment: 57 pages, 12 figures, 3 tables (v2: Made minor clarifications,
updated a reference, fixed typos
Stanford telemetry monitoring experiment on Lunar Explorer 35 Final report
Explorer 35 data analysis including occultation study and antenna pattern interpretation along with electromagnetic property experiment
Progress report on the relativistic three-particle quantization condition
We describe recent work on the relativistic three-particle quantization
condition, generalizing and applying the original formalism of Hansen and
Sharpe, and of Brice\~no, Hansen and Sharpe. In particular, we sketch three
recent developments: the generalization of the formalism to include K-matrix
poles; the numerical implementation of the quantization condition in the
isotropic approximation; and ongoing work extending the description of the
three-particle divergence-free K matrix beyond the isotropic approximation.Comment: 7 pages, 1 figure, Proceedings of Lattice 201
Copper Oxide Nanoparticles Impact Several Toxicological Endpoints and Cause Neurodegeneration in \u3cem\u3eCaenorhabditis elegans\u3c/em\u3e
Engineered nanoparticles are becoming increasingly incorporated into technology and consumer products. In 2014, over 300 tons of copper oxide nanoparticles were manufactured in the United States. The increased production of nanoparticles raises concerns regarding the potential introduction into the environment or human exposure. Copper oxide nanoparticles commonly release copper ions into solutions, which contribute to their toxicity. We quantified the inhibitory effects of both copper oxide nanoparticles and copper sulfate on C. elegans toxicological endpoints to elucidate their biological effects. Several toxicological endpoints were analyzed in C. elegans, including nematode reproduction, feeding behavior, and average body length. We examined three wild C. elegans isolates together with the Bristol N2 laboratory strain to explore the influence of different genotypic backgrounds on the physiological response to copper challenge. All strains exhibited greater sensitivity to copper oxide nanoparticles compared to copper sulfate, as indicated by reduction of average body length and feeding behavior. Reproduction was significantly reduced only at the highest copper dose, though still more pronounced with copper oxide nanoparticles compared to copper sulfate treatment. Furthermore, we investigated the effects of copper oxide nanoparticles and copper sulfate on neurons, cells with known vulnerability to heavy metal toxicity. Degeneration of dopaminergic neurons was observed in up to 10% of the population after copper oxide nanoparticle exposure. Additionally, mutants in the divalent-metal transporters, smf-1 or smf-2, showed increased tolerance to copper exposure, implicating both transporters in copper-induced neurodegeneration. These results highlight the complex nature of CuO nanoparticle toxicity, in which a nanoparticle-specific effect was observed in some traits (average body length, feeding behavior) and a copper ion specific effect was observed for other traits (neurodegeneration, response to stress)
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