Hazard ratios for incident type 2 diabetes, by anthropometric measure: The ARIC study.

<p>Hazard ratios for incident type 2 diabetes, by anthropometric measure: The ARIC study.</p

Harrell’s C-Index for incident type 2 diabetes by anthropometric measure: The ARIC study.

<p>Harrell’s C-Index for incident type 2 diabetes by anthropometric measure: The ARIC study.</p

Forest plot of hazard ratios by race-gender groups: The ARIC study.

<p>Abbreviations: ARIC, Atherosclerosis Risk in Communities; a body shape index (ABSI), body adiposity index (BAI), body mass index (BMI), waist circumference (WC), waist to height ratio (WHtR), waist to hip ratio (WHR), and waist to hip to height ratio (WHHR). Individual models using repeated measures survival analysis were constructed with T2DM status (yes/no) as the response, with each anthropometric measure as the exposure variable, adjusted for age (5-year increments), over 4 visits (baseline: 1987–1989, visit 2: 1990–1992, visit 3: 1993–1995, visit 4: 1996–1998) using Atherosclerosis Risk in Communities study data.</p

Simple regression of outcome variables on dietary phytosterol intake.

<p>Results are R-squares of simple ordinary least square regression analysis (n = 94 observations), all <i>P</i> < 0.0001.</p><p>Simple regression of outcome variables on dietary phytosterol intake.</p

Correlations between plasma non-cholesterol sterol concentrations at each dietary phytosterol intake level.

<p>Values reflect Spearman’s rank-order correlation coefficients in the Supplement Study (A) and the Natural Study (B).</p

Observed dietary phytosterol intake (DPI) (A, Supplement Study; B, Natural Study).

<p>*significantly different from 50 mg/2000 Kcal, <i>P</i> < 0.01; **significantly different from 50 mg/2000 Kcal or 450 mg/2000 Kcal (A), or from 126 mg/2000 Kcal (B), <i>P</i> < 0.0001.</p

Participant characteristics at baseline.

<p>Values are means ± SD.</p><p>Participant characteristics at baseline.</p

Simple regression of outcome variables on dietary phytosterol intake.

<p>Results are R-squares of simple ordinary least square regression analysis (n = 94 observations), all <i>P</i> < 0.0001.</p><p>Simple regression of outcome variables on dietary phytosterol intake.</p

Observed dietary phytosterol intake (DPI) (A, Supplement Study; B, Natural Study).

<p>*significantly different from 50 mg/2000 Kcal, <i>P</i> < 0.01; **significantly different from 50 mg/2000 Kcal or 450 mg/2000 Kcal (A), or from 126 mg/2000 Kcal (B), <i>P</i> < 0.0001.</p

Regression of plasma phytosterol levels normalized by 5-α-cholestanol on dietary phytosterol intake (DPI).

<p>All values were transformed to natural log. Dotted lines represent 95% prediction limits of Ln(Campesterol/5-α-cholestanol) (A) and Ln(Total phytosterols/5-α-cholestanol) (B).</p