Democratic Security: A Concept for the Swedish Total Defense Approach

Democracy and the rule of law are classified as “national interests” and ”critical assets” in Sweden’s National Security Strategy and Military Strategic Doctrine. These assets are targets of a range of challenges and attacks. For instance, foreign powers seek to create confusion and polarization through disinformation and influence operations. Domestic violent extremist groups promote conspiracy narratives in order to divide society and create mistrust of authorities. Meanwhile, social phenomena such as serious organized crime and honor-based violence undermine and counteract the basic principles and functions of both democracy and the rule of law and, additionally, are actively exploited by malicious actors. Importantly, these challenges frequently overlap and interact. This paper gives an overview of Democratic Security, a model created in order to answer to two primary questions: what does it mean to posit democracy and the rule of law as critical assets? And, how does one operationalize the defense of values and principles? It is an approach to understanding threats and challenges democracy and the rule of law, within which these principles and processes are seen not only as core critical assets but also as normative for effective and democratically sustainable counter-measures. This enables analysis of both vulnerabilities and threats that is highly granular

Anders Strindberg reflects on the impact of Jonathan Gaddy’s CHDS thesis, “An Ontology of Power: Perception and Reality in Conflict”

The article record may be found at https://www.hsaj.org/articles/17421Sponsored the U. S. Department of Homeland Security’s National Preparedness Directorate, FEMA, CHDS is part of the Naval Postgraduate School (NPS)

Anders Strindberg reviews Peter Berger and Thomas Luckmann, The Social Construction of Reality

The article record may be found at https://www.hsaj.org/articles/21407Sponsored the U. S. Department of Homeland Security’s National Preparedness Directorate, FEMA, CHDS is part of the Naval Postgraduate School (NPS)

Democratic Security: A Concept for the Swedish Total Defense Approach

Democracy and the rule of law are classified as “national interests” and ”critical assets” in Sweden’s National Security Strategy and Military Strategic Doctrine. These assets are targets of a range of challenges and attacks. For instance, foreign powers seek to create confusion and polarization through disinformation and influence operations. Domestic violent extremist groups promote conspiracy narratives in order to divide society and create mistrust of authorities. Meanwhile, social phenomena such as serious organized crime and honor-based violence undermine and counteract the basic principles and functions of both democracy and the rule of law and, additionally, are actively exploited by malicious actors. Importantly, these challenges frequently overlap and interact. This paper gives an overview of Democratic Security, a model created in order to answer to two primary questions: what does it mean to posit democracy and the rule of law as critical assets? And, how does one operationalize the defense of values and principles? It is an approach to understanding threats and challenges democracy and the rule of law, within which these principles and processes are seen not only as core critical assets but also as normative for effective and democratically sustainable counter-measures. This enables analysis of both vulnerabilities and threats that is highly granular

From the river to the sea? : honour, identity and politics in historical and contemporary Palestinian rejectionism

The present thesis seeks to understand and explain the rhetoric and behaviour of the rejectionist 'current' within the Palestinian national movement. It proceeds from the view that extant scholarship, primarily from within the fields of terrorism and security studies, has profoundly misunderstood rejectionist speech and behaviour by ignoring the explanatory capacity of Emic - the research subject's perception - as well as the influence of the sociocultural milieu within which rejectionism exists. The thesis proceeds to set up a 'socioculturally sensitive' analytical framework drawn from social identity theory, a heuristic, non-reductionist model for understanding group interaction and conflict. Emphasizing cultural norms and cues identified by anthropologists as salient in the eastern Mediterranean, the thesis suggests that the social value of honour, patron-client dynamics and a firmly entrenched group orientation must be significant elements of a model for understanding rejectionist behaviour. The main analytical narrative suggests that for reasons derived from ideology, patron-client relations and group dynamics, what has distinguished the rejectionists from the mainstream have been a qualitatively different set of preconditions for, and objectives of diplomatic negotiations. To the main rejectionist factions the goal of liberating Palestine has always been inextricably intertwined with the goal of restoring national honour; one without the other has been impossible and to claim otherwise would mean a depletion of factional and personal honour. To the rejectionists, there has never been any question of deviating from the fundamental goals - national recognition, repatriation, self-determination and independent statehood, not even for tactical reasons. This 'higher standard' likely derives from their structurally and politically subordinate position within the national movement, and the need to creatively enhance their own social status and appeal

lslamism: Religion, Radicalization, and Resistance (Publication Announcement)

A publication announcement created for an event at the Dudley Knox Library, Naval Postgraduate Schoo

The role of circulating galectin-1 in type 2 diabetes and chronic kidney disease : evidence from cross-sectional, longitudinal and Mendelian randomisation analyses

Aims/hypothesis: Galectin-1 modulates inflammation and angiogenesis, and cross-sectional studies indicate that galectin-1 may be a uniting factor between obesity, type 2 diabetes and kidney function. We examined whether circulating galectin-1 can predict incidence of chronic kidney disease (CKD) and type 2 diabetes in a middle-aged population, and if Mendelian randomisation (MR) can provide evidence for causal direction of effects. Methods: Participants (n = 4022; 58.6% women) in the Malmö Diet and Cancer Study–Cardiovascular Cohort enrolled between 1991 and 1994 (mean age 57.6 years) were examined. eGFR was calculated at baseline and after a mean follow-up of 16.6 ± 1.5 years. Diabetes status was ascertained through registry linkage (mean follow-up of 18.4 ± 6.1 years). The associations of baseline galectin-1 with incident CKD and type 2 diabetes were assessed with Cox regression, adjusting for established risk factors. In addition, a genome-wide association study on galectin-1 was performed to identify genetic instruments for two-sample MR analyses utilising the genetic associations obtained from the Chronic Kidney Disease Genetics (CKDGen) Consortium (41,395 cases and 439,303 controls) and the DIAbetes Genetics Replication And Meta-analysis (DIAGRAM) consortium (74,124 cases and 824,006 controls). One genome-wide significant locus in the galectin-1 gene region was identified (sentinel SNP rs7285699; p = 2.4 × 10−11). The association between galectin-1 and eGFR was also examined in individuals with newly diagnosed diabetes from the All New Diabetics In Scania (ANDIS) cohort. Results: Galectin-1 was strongly associated with lower eGFR at baseline (p = 2.3 × 10−89) but not with incident CKD. However, galectin-1 was associated with increased risk of type 2 diabetes (per SD increase, HR 1.12; 95% CI 1.02, 1.24). Two-sample MR analyses could not ascertain a causal effect of galectin-1 on CKD (OR 0.92; 95% CI 0.82, 1.02) or type 2 diabetes (OR 1.05; 95% CI 0.98, 1.14) in a general population. However, in individuals with type 2 diabetes from ANDIS who belonged to the severe insulin-resistant diabetes subgroup and were at high risk of diabetic nephropathy, genetically elevated galectin-1 was significantly associated with higher eGFR (p = 5.7 × 10−3). Conclusions/interpretation: Galectin-1 is strongly associated with lower kidney function in cross-sectional analyses, and two-sample MR analyses suggest a causal protective effect on kidney function among individuals with type 2 diabetes at high risk of diabetic nephropathy. Future studies are needed to explore the mechanisms by which galectin-1 affects kidney function and whether it could be a useful target among individuals with type 2 diabetes for renal improvement. Graphical abstract: [Figure not available: see fulltext.

Metabolic profiling of galectin-1 and galectin-3 : a cross-sectional, multi-omics, association study

Objectives Experimental studies indicate a role for galectin-1 and galectin-3 in metabolic disease, but clinical evidence from larger populations is limited. Methods We measured circulating levels of galectin-1 and galectin-3 in the Prospective investigation of Obesity, Energy and Metabolism (POEM) study, participants (n = 502, all aged 50 years) and characterized the individual association profiles with metabolic markers, including clinical measures, metabolomics, adipose tissue distribution (Imiomics) and proteomics. Results Galectin-1 and galectin-3 were associated with fatty acids, lipoproteins and triglycerides including lipid measurements in the metabolomics analysis adjusted for body mass index (BMI). Galectin-1 was associated with several measurements of adiposity, insulin secretion and insulin sensitivity, while galectin-3 was associated with triglyceride-glucose index (TyG) and fasting insulin levels. Both galectins were associated with inflammatory pathways and fatty acid binding protein (FABP)4 and -5-regulated triglyceride metabolic pathways. Galectin-1 was also associated with several proteins related to adipose tissue differentiation. Conclusions The association profiles for galectin-1 and galectin-3 indicate overlapping metabolic effects in humans, while the distinctly different associations seen with fat mass, fat distribution, and adipose tissue differentiation markers may suggest a functional role of galectin-1 in obesity

Hyperinsulinemia and insulin resistance in the obese may develop as part of a homeostatic response to elevated free fatty acids : A mechanistic case-control and a population-based cohort study

Background: It is commonly accepted that in obesity free fatty acids (FFA) cause insulin resistance and hyperglycemia, which drives hyperinsulinemia. However, hyperinsulinemia is observed in subjects with normoglycaemia and thus the paradigm above should be reevaluated. Methods: We describe two studies: MD-Lipolysis, a case control study investigating the mechanisms of obesity-driven insulin resistance by a systemic metabolic analysis, measurements of adipose tissue lipolysis by microdialysis, and adipose tissue genomics; and POEM, a cohort study used for validating differences in circulating metabolites in relation to adiposity and insulin resistance observed in the MD-Lipolysis study. Findings: In insulin-resistant obese with normal glycaemia from the MD-Lipolysis study, hyperinsulinemia was associated with elevated FFA. Lipolysis, assessed by glycerol release per adipose tissue mass or adipocyte surface, was similar between obese and lean individuals. Adipose tissue from obese subjects showed reduced expression of genes mediating catecholamine-driven lipolysis, lipid storage, and increased expression of genes driving hyperplastic growth. In the POEM study, FFA levels were specifically elevated in obese-overweight subjects with normal fasting glucose and high fasting levels of insulin and C-peptide. Interpretation: In obese subjects with normal glycaemia elevated circulating levels of FFA at fasting are the major metabolic derangement candidate driving fasting hyperinsulinemia. Elevated FFA in obese with normal glycaemia were better explained by increased fat mass rather than by adipose tissue insulin resistance. These results support the idea that hyperinsulinemia and insulin resistance may develop as part of a homeostatic adaptive response to increased adiposity and FFA.De två första författarna delar förstaförfattarskapet.</p