347 research outputs found
Coupled non-equilibrium growth equations: Self-consistent mode coupling using vertex renormalization
We find that studying the simplest of the coupled non-equilibrium growth
equations of Barabasi by self-consistent mode coupling requires the use of
dressed vertices. Using the vertex renormalization, we find a roughness
exponent which already in the leading order is quite close to the numerical
value.Comment: 7 pages, 3 figure
Design, fabrication, and characterization of deep-etched waveguide gratings
One-dimensional (1-D) deep-etched gratings on a specially grown AlGaAs wafer were designed and fabricated. The gratings were fabricated using state-of-the-art electron beam lithography and high-aspect-ratio reactive ion etching (RIE) in order to achieve the required narrow deep air slots with good accuracy and reproducibility. Since remarkable etch depths (up to 1.5 /spl mu/m), which completely cut through the waveguide core layer, have been attained, gratings composed of only five periods (and, thus, shorter than 6 /spl mu/m) have a bandgap larger than 100 nm. A defect was introduced by increasing the width of the central semiconductor tooth to create microcavities that exhibit a narrow transmission peak (less than 7 nm) around the wavelength of 1530 nm. The transmission spectra between 1460 and 1580 nm have been systematically measured, and the losses have been estimated for a set of gratings, both with and without a defect, for different periods and air slot dimensions. Numerical results obtained via a bidirectional beam propagation code allowed the evaluation of transmissivity, reflectivity, and diffraction losses. By comparing experimental results with the authors' numerical findings, a clear picture of the role of the grating's geometric parameters in determining its spectral features and diffractive losses is illustrated
Frequency-dependent (ac) Conduction in Disordered Composites: a Percolative Study
In a recent paper [Phys. Rev. B{\bf57}, 3375 (1998)], we examined in detail
the nonlinear (electrical) dc response of a random resistor cum tunneling bond
network (, introduced by us elsewhere to explain nonlinear response of
metal-insulator type mixtures). In this work which is a sequel to that paper,
we consider the ac response of the -based correlated () model.
Numerical solutions of the Kirchoff's laws for the model give a power-law
exponent (= 0.7 near ) of the modulus of the complex ac conductance at
moderately low frequencies, in conformity with experiments on various types of
disordered systems. But, at very low frequencies, it gives a simple quadratic
or linear dependence on the frequency depending upon whether the system is
percolating or not. We do also discuss the effective medium approximation
() of our and the traditional random network model, and discuss
their comparative successes and shortcomings.Comment: Revised and reduced version with 17 LaTeX pages plus 8 JPEG figure
Scaling properties in off equilibrium dynamical processes
In the present paper, we analyze the consequences of scaling hypotheses on
dynamic functions, as two times correlations . We show, under general
conditions, that must obey the following scaling behavior , where the scaling variable is
and , two
undetermined functions. The presence of a non constant exponent
signals the appearance of multiscaling properties in the dynamics.Comment: 6 pages, no figure
Molecular dynamics simulations of oxide memristors: thermal effects
We have extended our recent molecular-dynamic simulations of memristors to
include the effect of thermal inhomogeneities on mobile ionic species appearing
during operation of the device. Simulations show a competition between an
attractive short-ranged interaction between oxygen vacancies and an enhanced
local temperature in creating/destroying the conducting oxygen channels. Such a
competition would strongly affect the performance of the memristive devices.Comment: submit/0169777; 6 pages, 4 figure
Transport on percolation clusters with power-law distributed bond strengths: when do blobs matter?
The simplest transport problem, namely maxflow, is investigated on critical
percolation clusters in two and three dimensions, using a combination of
extremal statistics arguments and exact numerical computations, for power-law
distributed bond strengths of the type .
Assuming that only cutting bonds determine the flow, the maxflow critical
exponent \ve is found to be \ve(\alpha)=(d-1) \nu + 1/(1-\alpha). This
prediction is confirmed with excellent accuracy using large-scale numerical
simulation in two and three dimensions. However, in the region of anomalous
bond capacity distributions () we demonstrate that, due to
cluster-structure fluctuations, it is not the cutting bonds but the blobs that
set the transport properties of the backbone. This ``blob-dominance'' avoids a
cross-over to a regime where structural details, the distribution of the number
of red or cutting bonds, would set the scaling. The restored scaling exponents
however still follow the simplistic red bond estimate. This is argued to be due
to the existence of a hierarchy of so-called minimum cut-configurations, for
which cutting bonds form the lowest level, and whose transport properties scale
all in the same way. We point out the relevance of our findings to other scalar
transport problems (i.e. conductivity).Comment: 9 pages + Postscript figures. Revtex4+psfig. Submitted to PR
Traveling length and minimal traveling time for flow through percolation networks with long-range spatial correlations
We study the distributions of traveling length l and minimal traveling time t
through two-dimensional percolation porous media characterized by long-range
spatial correlations. We model the dynamics of fluid displacement by the
convective movement of tracer particles driven by a pressure difference between
two fixed sites (''wells'') separated by Euclidean distance r. For strongly
correlated pore networks at criticality, we find that the probability
distribution functions P(l) and P(t) follow the same scaling Ansatz originally
proposed for the uncorrelated case, but with quite different scaling exponents.
We relate these changes in dynamical behavior to the main morphological
difference between correlated and uncorrelated clusters, namely, the
compactness of their backbones. Our simulations reveal that the dynamical
scaling exponents for correlated geometries take values intermediate between
the uncorrelated and homogeneous limiting cases
Initial-State Interactions in the Unpolarized Drell-Yan Process
We show that initial-state interactions contribute to the
distribution in unpolarized Drell-Yan lepton pair production and , without suppression. The asymmetry is expressed as a
product of chiral-odd distributions , where the quark-transversity function
is the transverse momentum dependent, light-cone
momentum distribution of transversely polarized quarks in an {\it unpolarized}
proton. We compute this (naive) -odd and chiral-odd distribution function
and the resulting asymmetry explicitly in a quark-scalar diquark
model for the proton with initial-state gluon interaction. In this model the
function equals the -odd (chiral-even) Sivers
effect function . This suggests that the
single-spin asymmetries in the SIDIS and the Drell-Yan process are closely
related to the asymmetry of the unpolarized Drell-Yan process,
since all can arise from the same underlying mechanism. This provides new
insight regarding the role of quark and gluon orbital angular momentum as well
as that of initial- and final-state gluon exchange interactions in hard QCD
processes.Comment: 22 pages, 6 figure
Concentration Dependence of Superconductivity and Order-Disorder Transition in the Hexagonal Rubidium Tungsten Bronze RbxWO3. Interfacial and bulk properties
We revisited the problem of the stability of the superconducting state in
RbxWO3 and identified the main causes of the contradictory data previously
published. We have shown that the ordering of the Rb vacancies in the
nonstoichiometric compounds have a major detrimental effect on the
superconducting temperature Tc.The order-disorder transition is first order
only near x = 0.25, where it cannot be quenched effectively and Tc is reduced
below 1K. We found that the high Tc's which were sometimes deduced from
resistivity measurements, and attributed to compounds with .25 < x < .30, are
to be ascribed to interfacial superconductivity which generates spectacular
non-linear effects. We also clarified the effect of acid etching and set more
precisely the low-rubidium-content boundary of the hexagonal phase.This work
makes clear that Tc would increase continuously (from 2 K to 5.5 K) as we
approach this boundary (x = 0.20), if no ordering would take place - as its is
approximately the case in CsxWO3. This behaviour is reminiscent of the
tetragonal tungsten bronze NaxWO3 and asks the same question : what mechanism
is responsible for this large increase of Tc despite the considerable
associated reduction of the electron density of state ? By reviewing the other
available data on these bronzes we conclude that the theoretical models which
are able to answer this question are probably those where the instability of
the lattice plays a major role and, particularly, the model which call upon
local structural excitations (LSE), associated with the missing alkali atoms.Comment: To be published in Physical Review
Do brain regions involved in threat processing mediate the association between developmental trauma and psychosis
Background: There is growing evidence that developmental trauma - psychologically traumatic events during childhood and/or adolescence – is causally associated with increased risk of psychosis in adulthood [1]. However, an understanding of the precise mechanisms underlying this is lacking. Consistent with biopsychosocial and computational theories of psychosis [2,3], multiple lines of evidence converge on the role of altered threat processing in the pathway linking developmental trauma and psychosis [4,5]. Here, in a well-characterised birth cohort, we investigate prospectively, the effect of developmental trauma on volumes of brain structures involved in threat processing, and examine their roles in the association between developmental trauma and psychotic experiences in adulthood.
Methods: We used data from the Avon Longitudinal Study of Parents and Children (ALSPAC) study, a large population-based cohort in the United Kingdom. Data from 418 participants were derived from parent- or self-reported assessments. Trauma variables represent trauma exposure (between 0-17 years), the number of types, and timing of trauma: childhood (0-10.9 years) or adolescence (11-17 years). Psychotic experiences were assessed using the psychosis-like symptoms semi-structured interview at 12 and 18 years. Magnetic resonance imaging was used to measure volumes of the whole brain, amygdala, vmPFC, and striatum at age 18. We used logistic and linear regression, and mediation analyses to examine associations. In addition, we explored whether these associations could be explained by genetic confounding or reverse causation, by repeating analyses (1) whilst adjusting for schizophrenia polygenic risk scores (PRS), and (2) in a subgroup of individuals who did not report psychotic experiences at age 12 (n=304).
Results: Exposure to developmental trauma was associated with an increased odds of psychotic experiences at age 18 (OR=1.80; 95% CI=1.17-2.81, p<.001), with evidence supporting dose-response effects for exposure to multiple trauma types (B=.18, p<.001, R2=.05), and at both age periods (B=.15, p<.001, R2=.03). Developmental trauma was associated with reduced left amygdalar volumes in adulthood (B=-.01, p<.01, R2=.02), with evidence supporting a dose-response association, whereby exposure to three or more types of trauma (B=-.004, p<.05, R2=.01), and exposure to trauma during both childhood and adolescence (B=-.003, p<.05, R2=.01), had a greater effect compared with exposure during childhood or adolescence only. Developmental trauma was not associated with alterations in vmPFC and striatal volumes. Reduced left amygdalar volumes mediated 16% (95% CI=2%-80%, p=.03) of the association between developmental trauma and psychotic experiences (mediation effect: 0.04, 95% CI=0.01-0.08, p=.015). These findings substantively remained the same in sensitivity analyses aimed to minimise the effects of reverse causation and genetic confounding.
Conclusions: In this study, we found evidence of a dose-response association between developmental trauma and reduced left amygdalar volumes in adulthood, and of a mediating role of left amygdalar volumes in the trauma-psychosis association. These findings were not explained by reverse causation or genetic confounding. These findings provide observational evidence for the hypothesis that a causal association between developmental trauma and altered threat processing underlies vulnerability to psychosis. Importantly, our identification of a neurobiological mediator of the trauma-psychosis relationship informs strategies for secondary and tertiary prevention of psychosis associated with developmental trauma
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