Inverse Additive Problems for Minkowski Sumsets II

The Brunn-Minkowski Theorem asserts that $\mu_d(A+B)^{1/d}\geq \mu_d(A)^{1/d}+\mu_d(B)^{1/d}$ for convex bodies $A,\,B\subseteq \R^d$, where $\mu_d$ denotes the $d$-dimensional Lebesgue measure. It is well-known that equality holds if and only if $A$ and $B$ are homothetic, but few characterizations of equality in other related bounds are known. Let $H$ be a hyperplane. Bonnesen later strengthened this bound by showing $$\mu_d(A+B)\geq (M^{1/(d-1)}+N^{1/(d-1)})^{d-1}(\frac{\mu_d(A)}{M}+\frac{\mu_d(B)}{N}),$$ where $M=\sup\{\mu_{d-1}((\mathbf x+H)\cap A)\mid \mathbf x\in \R^d\}$ and $N=\sup\{\mu_{d-1}((\mathbf y+H)\cap B)\mid \mathbf y\in \R^d\}$. Standard compression arguments show that the above bound also holds when $M=\mu_{d-1}(\pi(A))$ and $N=\mu_{d-1}(\pi(B))$, where $\pi$ denotes a projection of $\mathbb R^d$ onto $H$, which gives an alternative generalization of the Brunn-Minkowski bound. In this paper, we characterize the cases of equality in this later bound, showing that equality holds if and only if $A$ and $B$ are obtained from a pair of homothetic convex bodies by `stretching' along the direction of the projection, which is made formal in the paper. When $d=2$, we characterize the case of equality in the former bound as well

On the critical pair theory in abelian groups : Beyond Chowla's Theorem

We obtain critical pair theorems for subsets S and T of an abelian group such that |S+T| < |S|+|T|+1. We generalize some results of Chowla, Vosper, Kemperman and a more recent result due to Rodseth and one of the authors.Comment: Submitted to Combinatorica, 23 pages, revised versio

Density of States for a Specified Correlation Function and the Energy Landscape

The degeneracy of two-phase disordered microstructures consistent with a specified correlation function is analyzed by mapping it to a ground-state degeneracy. We determine for the first time the associated density of states via a Monte Carlo algorithm. Our results are described in terms of the roughness of the energy landscape, defined on a hypercubic configuration space. The use of a Hamming distance in this space enables us to define a roughness metric, which is calculated from the correlation function alone and related quantitatively to the structural degeneracy. This relation is validated for a wide variety of disordered systems.Comment: Accepted for publication in Physical Review Letter

Effective Mass Dirac-Morse Problem with any kappa-value

The Dirac-Morse problem are investigated within the framework of an approximation to the term proportional to $1/r^2$ in the view of the position-dependent mass formalism. The energy eigenvalues and corresponding wave functions are obtained by using the parametric generalization of the Nikiforov-Uvarov method for any $\kappa$-value. It is also studied the approximate energy eigenvalues, and corresponding wave functions in the case of the constant-mass for pseudospin, and spin cases, respectively.Comment: 12 page

Symmetry breaking and the random-phase approximation in small quantum dots

The random-phase approximation has been used to compute the properties of parabolic two-dimensional quantum dots beyond the mean-field approximation. Special emphasis is put on the ground state correlation energy, the symmetry restoration and the role of the spurious modes within the random-phase approximation. A systematics with the Coulombic interaction strength is presented for the 2-electron dot, while for the 6- and 12-electron dots selected cases are discussed. The validity of the random-phase approximation is assessed by comparison with available exact results.Comment: 9 pages, 4 embedded + 6 gif Figs. Published versio

LRRK2 affects vesicle trafficking, neurotransmitter extracellular level and membrane receptor localization

The leucine-rich repeat kinase 2 (LRRK2) gene was found to play a role in the pathogenesis of both familial and sporadic Parkinson's disease (PD). LRRK2 encodes a large multi-domain protein that is expressed in different tissues. To date, the physiological and pathological functions of LRRK2 are not clearly defined. In this study we have explored the role of LRRK2 in controlling vesicle trafficking in different cellular or animal models and using various readouts. In neuronal cells, the presence of LRRK2(G2019S) pathological mutant determines increased extracellular dopamine levels either under basal conditions or upon nicotine stimulation. Moreover, mutant LRRK2 affects the levels of dopamine receptor D1 on the membrane surface in neuronal cells or animal models. Ultrastructural analysis of PC12-derived cells expressing mutant LRRK2(G2019S) shows an altered intracellular vesicle distribution. Taken together, our results point to the key role of LRRK2 to control vesicle trafficking in neuronal cells