Atmospheric effects on crater growth on Venus

Laboratory experiments allow examining the consequences of complex processes operating over a wide range of scales (both temporal and spatial) and frequently reveal effects that are obvious only in hindsight. Even though all processes may not scale directly, isolation of the controlling variables allows assessing first-order effects through analytical approximations. This approach can be illustrated by the systematic sequence of ballistic ejection, the response of an atmosphere to a strong energy source, the scaling of ejecta thickness, and the role of secondary cratering. Here it is proposed that the effects of atmospheric pressure and density on crater growth (hence, scaling) observed in laboratory experiments has particular relevance for craters on Venus

Effect of impact angle on central-peak/peak-ring formation and crater collapse on Venus

Although asymmetry in ejecta patterns and craters shape-in-plan are commonly cited as diagnostic features of impact angle, the early-time transfer of energy from impactor to target also creates distinctive asymmetries in crater profile with the greatest depth uprange. In order to simulate gravity-controlled crater-growth, laboratory experiments use loose particulate targets as analogs for low-strength material properties following passage of the shock. As a result, impact crater diameter D in laboratory experiments generally is many times greater than the impactor diameter 2r (factor of 40), and early-time asymmetries in energy transfer from oblique impacts are consumed by subsequent symmetrical crater growth, except at the lowest angles (less than 25 deg). Such asymmetry is evident for oblique (less than 60 deg from horizontal) impacts into aluminum where D/2r is only 2 to 4. Because cratering efficiency decreases with increasing crater size and decreasing impact angle, large scale planetary craters (4080 km) should have transient excavation diameters only 6-10 times larger than the impactor. At basin scales, D/2r is predicted to be only 3-5, i.e., approaching values for impacts into aluminum in laboratory experiments. As a result, evidence for early-time asymmetry in impactor energy transfer should become evident on planetary surfaces, yet craters generally retain a circular outline for all but the lowest impact angles

Timing of ancient extensional tectonic features on Mars

Although numerous studies have delineated the Tharsis and post-Tharsis volcanic/tectonic history on Mars, only a few attempts have examined the earlier epochs. This is not an easy task since unambiguous crater ages for pre-Tharsis and early Tharsis units are difficult to determine owing to a variety of active surface processes. Ancient tectonic features, however, have a sufficiently large superposed crater population that should permit relative dating. A technique for crater counting along linear features analagous to areal crater density is proposed. A modification of this approach has been tested and applied to a variety of ancient tectonic features

Impacts of free-floating objects: Unique Space Station experiments

The transfer of momentum and kinetic energy between planetary bodies forms the basis for wide-ranging problems in planetary science ranging from the collective long-term effects of minor perturbations to the catastrophic singular effect of a major collision. In the former case, the evolution of asteroid spin rates and orientations and planetary rotation rates are cited. In the latter case, the catastrophic angular momenta and the near-global disruption of partially molten planets are included. Although the collisional transfer of momentum and energy were discussed over the last two decades, major issues remain that largely reflect current limitations in earth-based experimental conditions and 3-D numerical codes. Two examples with potential applications in a Space Station laboratory are presented

Planetary impact experimentation

An understanding of impact processes in low- and microgravity environments would be advanced significantly by the construction and use of an impact facility on the Space Station. It is proposed that initial studies begin as soon as possible in ground-based impact laboratories, on the NASA KC-135 Reduced-Gravity Aircraft, and in existing drop towers. The resulting experience and information base could then be applied toward an experiment package designed for use on Shuttle orbiters to support pilot studies in orbital environments. These experiments, as well as the first efforts made on the IOC Space Station, should involve the impact of various free-floating targets; such studies would yield a substantial scientific return while providing valuable experience and engineering information for use in refining the design of the dedicated Space Station Impact Facility. The dedicated facility should be designed to support impact experimentation, including but not limited to cratering, asteroid and ring-particle dynamics, and accretional processes

Ca2+-controlled competitive diacylglycerol binding of protein kinase C isoenzymes in living cells

The cellular decoding of receptor-induced signaling is based in part on the spatiotemporal activation pattern of PKC isoforms. Because classical and novel PKC isoforms contain diacylglycerol (DAG)-binding C1 domains, they may compete for DAG binding. We reasoned that a Ca2+-induced membrane association of classical PKCs may accelerate the DAG binding and thereby prevent translocation of novel PKCs. Simultaneous imaging of fluorescent PKC fusion proteins revealed that during receptor stimulation, PKCα accumulated in the plasma membrane with a diffusion-limited kinetic, whereas translocation of PKCɛ was delayed and attenuated. In BAPTA-loaded cells, however, a selective translocation of PKCɛ, but not of coexpressed PKCα, was evident. A membrane-permeable DAG analogue displayed a higher binding affinity for PKCɛ than for PKCα. Subsequent photolysis of caged Ca2+ immediately recruited PKCα to the membrane, and DAG-bound PKCɛ was displaced. At low expression levels of PKCɛ, PKCα concentration dependently prevented the PKCɛ translocation with half-maximal effects at equimolar coexpression. Furthermore, translocation of endogenous PKCs in vascular smooth muscle cells corroborated the model that a competition between PKC isoforms for DAG binding occurs at native expression levels. We conclude that Ca2+-controlled competitive DAG binding contributes to the selective recruitment of PKC isoforms after receptor activation