Inducción rizogénica en estacas semileñosas de Berberidopsis corallina

- Latsague, M. ; Sáez, P.; Cifuentes, P.; Yáñez ,Y. Laboratorio de Ecofisiología Vegetal y Citogenética, Facultad de Recursos Naturales, Universidad Católica de Temuco, Chile.Berberidopsis corallina (michay rojo) es una especie endémica, catalogada como especie “En Peligro de Extinción”. Su persistente disminución está asociada a tala y competencia con Eucalyptus spp. y P. radiata. A raíz de continuas campañas realizadas por organizaciones ambientales, se han enfocado esfuerzos en intentar conservar los bosques nativos remanentes. Entre las herramientas de gestión a esta problemática se cuenta la propagación vegetativa, como una forma de aumentar el numero de individuos y acortar el período vegetativo, sin embargo, existe escasa información respecto a la propagación de michay rojo, por lo que, el objetivo del estudio fue evaluar la respuesta al tratamiento de enraizamiento de estacas semileñosas, como un aporte a la conservación de la especie. Las estacas se colectaron en marzo del 2007 en el sector Villa las Araucarias, al Sur de la Cordillera de Nahuelbuta, región de la Araucanía. Las estacas se trataron con Ácido Indol Butírico (AIB) en distintas concentraciones (0, 500,1000 y 1500 ppm). Luego de cinco meses en cama caliente, se obtuvo un 87% de enraizamiento total. La concentración 1000 ppm de AIB mostró los mejores resultados respecto al proceso de rizogénesis con un 90% de enraizamiento, encontrando la mayor longitud de raíces en este mismo tratamiento con un promedio de 13,64 cm. El mayor número de raíces se obtuvo en el tratamiento 1500 ppm de AIB con un valor promedio de 38,11 raíces por estaca. Se concluye que michay rojo puede ser reproducido vegetativamente a través de la rizogénesis de estacas tratadas con AIB

On KLJN-based secure key distribution in vehicular communication networks

In a former paper [Fluct. Noise Lett., 13 (2014) 1450020] we introduced a vehicular communication system with unconditionally secure key exchange based on the Kirchhoff-Law-Johnson-Noise (KLJN) key distribution scheme. In this paper, we address the secure KLJN key donation to vehicles. This KLJN key donation solution is performed lane-by-lane by using roadside key provider equipment embedded in the pavement. A method to compute the lifetime of the KLJN key is also given. This key lifetime depends on the car density and gives an upper limit of the lifetime of the KLJN key for vehicular communication networks.Comment: Accepted for publicatio

Seguimiento del curriculum profesional médico, habilidades básicas y específicas para su docencia, basado en microteachen y diagnóstico por observación total

Tesis Univ. Complutense.Fac. de MedicinaTRUEProQuestpu

Seguimiento del curriculum profesional médico, habilidades básicas y específicas para su docencia, basado en microteachen y diagnóstico por observación total

Tesis Univ. Complutense.Fac. de MedicinaTRUEProQuestpu

Asymmetry in functional connectivity of the human habenula revealed by high-resolution cardiac-gated resting state imaging

The habenula is a hub for cognitive and emotional signals that are relayed to the aminergic centers in the midbrain and, thus, plays an important role in goal-oriented behaviors. Although it is well described in rodents and non-human primates, the habenula functional network remains relatively uncharacterized in humans, partly because of the methodological challenges associated with the functional magnetic resonance imaging of small structures in the brain. Using high-resolution cardiac-gated resting state imaging in healthy humans and precisely identifying each participants' habenula, we show that the habenula is functionally coupled with the insula, parahippocampus, thalamus, periaqueductal grey, pons, striatum and substantia nigra/ventral tegmental area complex. Furthermore, by separately examining and comparing the functional maps from the left and right habenula, we provide the first evidence of an asymmetry in the functional connectivity of the habenula in humans. Hum Brain Mapp 37:2602-2615, 2016. © 2016 The Authors Human Brain Mapping Published by Wiley Periodicals, Inc

Elogio conque la nacion española felicita a su monarca ... Carlos IV, en su glorisisima exaltacion al trono

Fecha : "1789" tomada del Palau, T. III nº 43398.Pie de imp. tomado de colofón.Sign. : [] 2, *

ADGO 2.0: interpreting microarray data and list of genes using composite annotations

ADGO 2.0 is a web-based tool that provides composite interpretations for microarray data comparing two sample groups as well as lists of genes from diverse sources of biological information. Some other tools also incorporate composite annotations solely for interpreting lists of genes but usually provide highly redundant information. This new version has the following additional features: first, it provides multiple gene set analysis methods for microarray inputs as well as enrichment analyses for lists of genes. Second, it screens redundant composite annotations when generating and prioritizing them. Third, it incorporates union and subtracted sets as well as intersection sets. Lastly, users can upload their own gene sets (e.g. predicted miRNA targets) to generate and analyze new composite sets. The first two features are unique to ADGO 2.0. Using our tool, we demonstrate analyses of a microarray dataset and a list of genes for T-cell differentiation. The new ADGO is available at http://www.btool.org/ADGO2

Brain ApoA-I, ApoJ and ApoE Immunodetection in Cerebral Amyloid Angiopathy

ApoA-I; ApoE; Cerebral amyloid angiopathyApoA-I; ApoE; Angiopatía amiloide cerebralApoA-I; ApoE; Angiopatia amiloide cerebralCerebral amyloid angiopathy (CAA) is a common cause of lobar intracerebral hemorrhage (ICH) in elderly individuals and it is the result of the cerebrovascular deposition of beta-amyloid (Aβ) protein. CAA is frequently found in patients with Alzheimer's disease (AD), although it has an independent contribution to the cognitive deterioration associated with age. Specific apolipoproteins (Apo) have been associated with Aβ fibrillization and clearance from the brain. In this regard, in the present study, we analyzed the brain levels of ApoE, ApoA-I, and ApoJ/clusterin in autopsy brains from 20 post-mortem cases with CAA type I, CAA type II, with parenchymal Aβ deposits or without Aβ deposits. Our objective was to find a possible differential pattern of apolipoproteins distribution in the brain depending on the CAA pathological presentation. The protein expression levels were adjusted by the APOE genotype of the patients included in the study. We found that ApoE and ApoJ were abundantly present in meningeal, cortical, and capillary vessels of the brains with vascular Aβ accumulation. ApoE and ApoJ also deposited extracellularly in the parenchyma, especially in cases presenting Aβ diffuse and neuritic parenchymal deposits. In contrast, ApoA-I staining was only relevant in capillary walls in CAA type I cases. On the other hand, ICH was the principal cause of death among CAA patients in our cohort. We found that CAA patients with ICH more commonly had APOEε2 compared with CAA patients without ICH. In addition, patients who suffered an ICH presented higher vascular ApoE levels in brain. However, higher ApoE presence in cortical arteries was the only independent predictor of suffering an ICH in our cohort after adjusting by age and APOE genotype. In conclusion, while ApoE and ApoJ appear to be involved in both vascular and parenchymal Aβ pathology, ApoA-I seems to be mainly associated with CAA, especially in CAA type I pathology. We consider that our study helps to molecularly characterize the distribution subtypes of Aβ deposition within the brain