Complete 0 hbar omega calculations of Gamow-Teller strengths for nuclei in the iron region

Gamow-Teller strengths for selected nuclei in the iron region (A~56) have been investigated via shell-model Monte Carlo calculations with realistic interactions in the complete fp basis. Results for all cases show significant quenching relative to single-particle estimates, in quantitative agreement with (n,p) data. The J=1,T=0 residual interaction and the f_{7/2}-f_{5/2} spin-orbit splitting are shown to play major roles in the quenching mechanism. Calculated B(E2, 2^+_1 -> 0^+_1) values are in fair agreement with experiment using effective charges of e_p=1.1e and e_n=0.1e.Comment: 13 pages + 1 postscript file, Caltech preprint MAP-16

Gamow-Teller strength in 54Fe and 56Fe

Through a sequence of large scale shell model calculations, total Gamow-Teller strengths ($S_+$ and $S_-$) in $^{54}$Fe and $^{56}$Fe are obtained. They reproduce the experimental values once the $\sigma\tau$ operator is quenched by the standard factor of $0.77$. Comparisons are made with recent Shell Model Monte Carlo calculations. Results are shown to depend critically on the interaction. From an analysis of the GT+ and GT$-$ strength functions it is concluded that experimental evidence is consistent with the $3(N-Z)$ sum rule.Comment: 6 pages, RevTeX 3.0 using psfig, 7 Postscript figures included using uufile

Ground and excited states Gamow-Teller strength distributions of iron isotopes and associated capture rates for core-collapse simulations

This paper reports on the microscopic calculation of ground and excited states Gamow-Teller (GT) strength distributions, both in the electron capture and electron decay direction, for $^{54,55,56}$Fe. The associated electron and positron capture rates for these isotopes of iron are also calculated in stellar matter. These calculations were recently introduced and this paper is a follow-up which discusses in detail the GT strength distributions and stellar capture rates of key iron isotopes. The calculations are performed within the framework of the proton-neutron quasiparticle random phase approximation (pn-QRPA) theory. The pn-QRPA theory allows a microscopic \textit{state-by-state} calculation of GT strength functions and stellar capture rates which greatly increases the reliability of the results. For the first time experimental deformation of nuclei are taken into account. In the core of massive stars isotopes of iron, $^{54,55,56}$Fe, are considered to be key players in decreasing the electron-to-baryon ratio ($Y_{e}$) mainly via electron capture on these nuclide. The structure of the presupernova star is altered both by the changes in $Y_{e}$ and the entropy of the core material. Results are encouraging and are compared against measurements (where possible) and other calculations. The calculated electron capture rates are in overall good agreement with the shell model results. During the presupernova evolution of massive stars, from oxygen shell burning stages till around end of convective core silicon burning, the calculated electron capture rates on $^{54}$Fe are around three times bigger than the corresponding shell model rates. The calculated positron capture rates, however, are suppressed by two to five orders of magnitude.Comment: 18 pages, 12 figures, 10 table

The nucleon-nucleon interaction

We review the major progress of the past decade concerning our understanding of the nucleon-nucleon interaction. The focus is on the low-energy region (below pion production threshold), but a brief outlook towards higher energies is also given. The items discussed include charge-dependence, the precise value of the $\pi NN$ coupling constant, phase shift analysis and high-precision NN data and potentials. We also address the issue of a proper theory of nuclear forces. Finally, we summarize the essential open questions that future research should be devoted to.Comment: 42 pages, 12 figures, iopart.cls style; Topical Review prepared for J. Phys. G: Nucl. Part. Phy

Shell-model Monte Carlo studies of fp-shell nuclei

We study the gross properties of even-even and $N=Z$ nuclei with $A=48-64$ using shell-model Monte Carlo methods. Our calculations account for all $0 \hbar \omega$ configurations in the $fp$-shell and employ the modified Kuo-Brown interaction KB3. We find good agreement with data for masses and total $B(E2)$ strengths, the latter employing effective charges $e_p=1.35e$ and $e_n=0.35e$. The calculated total Gamow-Teller strengths agree consistently with the $B(GT_+)$-values deduced from $(n,p)$ data if the shell model results are renormalized by $0.64$, as has already been established for $sd$-shell nuclei. The present calculations therefore suggest that this renormalization (i.e., $g_A=1$ in the nuclear medium) is universal.Comment: 20 pages, 7 figures, Caltech Preprint

Growth inhibition of oral mutans streptococci and candida by commercial probiotic lactobacilli - an in vitro study

<p>Abstract</p> <p>Background</p> <p>Probiotic bacteria are suggested to play a role in the maintenance of oral health. Such health promoting bacteria are added to different commercial probiotic products. The aim of the study was to investigate the ability of a selection of lactobacilli strains, used in commercially available probiotic products, to inhibit growth of oral mutans streptococci and <it>C. albicans in vitro</it>.</p> <p>Methods</p> <p>Eight probiotic lactobacilli strains were tested for growth inhibition on three reference strains and two clinical isolates of mutans streptococci as well as two reference strains and three clinical isolates of <it>Candida albicans </it>with an agar overlay method.</p> <p>Results</p> <p>At concentrations ranging from 10⁹to 10⁵CFU/ml, all lactobacilli strains inhibited the growth of the mutans streptococci completely with the exception of <it>L. acidophilus </it>La5 that executed only a slight inhibition of some strains at concentrations corresponding to 10⁷and 10⁵CFU/ml. At the lowest cell concentration (10³CFU/ml), only <it>L. plantarum </it>299v and <it>L. plantarum </it>931 displayed a total growth inhibition while a slight inhibition was seen for all five mutans streptococci strains by <it>L. rhamnosus </it>LB21, <it>L. paracasei </it>F19, <it>L. reuteri </it>PTA 5289 and <it>L. reuteri </it>ATCC 55730. All the tested lactobacilli strains reduced candida growth but the effect was generally weaker than for mutans streptococci. The two <it>L. plantarum </it>strains and <it>L. reuteri </it>ATCC 55730 displayed the strongest inhibition on <it>Candida albicans</it>. No significant differences were observed between the reference strains and the clinical isolates.</p> <p>Conclusion</p> <p>The selected probiotic strains showed a significant but somewhat varying ability to inhibit growth of oral mutans streptococci and <it>Candida albicans in vitro</it>.</p

Fine-Grid Calculations for Stellar Electron and Positron Capture Rates on Fe-Isotopes

The acquisition of precise and reliable nuclear data is a prerequisite to success for stellar evolution and nucleosynthesis studies. Core-collapse simulators find it challenging to generate an explosion from the collapse of the core of massive stars. It is believed that a better understanding of the microphysics of core-collapse can lead to successful results. The weak interaction processes are able to trigger the collapse and control the lepton-to-baryon ratio ($Y_{e}$) of the core material. It is suggested that the temporal variation of $Y_{e}$ within the core of a massive star has a pivotal role to play in the stellar evolution and a fine-tuning of this parameter at various stages of presupernova evolution is the key to generate an explosion. During the presupernova evolution of massive stars, isotopes of iron, mainly $^{54,55,56}$Fe, are considered to be key players in controlling $Y_{e}$ ratio via electron capture on these nuclide. Recently an improved microscopic calculation of weak interaction mediated rates for iron isotopes was introduced using the proton-neutron quasiparticle random phase approximation (pn-QRPA) theory. The pn-QRPA theory allows a microscopic \textit{state-by-state} calculation of stellar capture rates which greatly increases the reliability of calculated rates. The results were suggestive of some fine-tuning of the $Y_{e}$ ratio during various phases of stellar evolution. Here we present for the first time the fine-grid calculation of the electron and positron capture rates on $^{54,55,56}$Fe. Core-collapse simulators may find this calculation suitable for interpolation purposes and for necessary incorporation in the stellar evolution codes.Comment: 21 pages, 6 ps figures and 2 table

Dopamine acting at D1-like, D2-like and α1-adrenergic receptors differentially modulates theta and gamma oscillatory activity in primary motor cortex

The loss of dopamine (DA) in Parkinson’s is accompanied by the emergence of exaggerated theta and beta frequency neuronal oscillatory activity in the primary motor cortex (M1) and basal ganglia. DA replacement therapy or deep brain stimulation reduces the power of these oscillations and this is coincident with an improvement in motor performance implying a causal relationship. Here we provide in vitro evidence for the differential modulation of theta and gamma activity in M1 by DA acting at receptors exhibiting conventional and non-conventional DA pharmacology. Recording local field potentials in deep layer V of rat M1, co-application of carbachol (CCh, 5 μM) and kainic acid (KA, 150 nM) elicited simultaneous oscillations at a frequency of 6.49 ± 0.18 Hz (theta, n = 84) and 34.97 ± 0.39 Hz (gamma, n = 84). Bath application of DA resulted in a decrease in gamma power with no change in theta power. However, application of either the D1-like receptor agonist SKF38393 or the D2-like agonist quinpirole increased the power of both theta and gamma suggesting that the DA-mediated inhibition of oscillatory power is by action at other sites other than classical DA receptors. Application of amphetamine, which promotes endogenous amine neurotransmitter release, or the adrenergic α1-selective agonist phenylephrine mimicked the action of DA and reduced gamma power, a result unaffected by prior co-application of D1 and D2 receptor antagonists SCH23390 and sulpiride. Finally, application of the α1-adrenergic receptor antagonist prazosin blocked the action of DA on gamma power suggestive of interaction between α1 and DA receptors. These results show that DA mediates complex actions acting at dopamine D1-like and D2-like receptors, α1 adrenergic receptors and possibly DA/α1 heteromultimeric receptors to differentially modulate theta and gamma activity in M1