2,004 research outputs found

    Patch Plate Materials Compatibility Assessment

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    Lunar dust proved to be a greater problem during the Apollo missions than was originally anticipated. The highly angular, charged dust particles stuck to seals, radiators, and visors; clogged mechanisms; and abraded space suits. As reported by Apollo 12 astronaut Pete Conrad "We must have had more than a hundred hours suited work with the same equipment, and the wear was not as bad on the training suits as it is on these flight suits in just the eight hours we were out.". Dust clinging to surfaces was also transport-ed into habitable spaces leading to lung and eye irritation of the astronauts. The Apollo astronauts were on the Lunar surface less than 24 hours and experienced many dust related problems. With the Artemis program, we are planning longer stays on the surface, with more activities that have the potential to put the astronauts and equipment in contact with greater quantities of Lunar dust. The success of these missions will depend on our understanding of material interactions with Lunar dust and the development of ways to mitigate dust effects in cases where exposure to dust will lead to failure of components, unacceptable loss of power or thermal control, unacceptable loss of visibility, or health issues. Through the Lunar Surface In-novation Initiative (LSII), we are initiating a Patch Plate Materials Compatibility Assessment project. The overall goal of the three year project is to develop passive approaches to mitigate Lunar dust adhesion to surfaces for technologies that are currently at TRL levels 2-3 to bring them to TRL level 5 through ground-based assessment, culminating in a demonstration flight experiment on a Commercial Lunar Payload Services (CLPS) lander in 2022-2023. This paper discusses the detailed technical objectives and approach for this project. References: Gaier, J.R. "The Effects of Lunar Dust on EVA Systems During the Apollo Missions," NASA/TM-2005-213610/REV1, (2005), Apollo 12 Technical Crew Debriefing, December 1, 1969, pp. 10-54

    Enhanced low-energy γ\gamma-decay strength of 70^{70}Ni and its robustness within the shell model

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    Neutron-capture reactions on very neutron-rich nuclei are essential for heavy-element nucleosynthesis through the rapid neutron-capture process, now shown to take place in neutron-star merger events. For these exotic nuclei, radiative neutron capture is extremely sensitive to their γ\gamma-emission probability at very low γ\gamma energies. In this work, we present measurements of the γ\gamma-decay strength of 70^{70}Ni over the wide range 1.3Eγ81.3 \leq E_{\gamma} \leq 8 MeV. A significant enhancement is found in the γ\gamma-decay strength for transitions with Eγ<3E_\gamma < 3 MeV. At present, this is the most neutron-rich nucleus displaying this feature, proving that this phenomenon is not restricted to stable nuclei. We have performed E1E1-strength calculations within the quasiparticle time-blocking approximation, which describe our data above Eγ5E_\gamma \simeq 5 MeV very well. Moreover, large-scale shell-model calculations indicate an M1M1 nature of the low-energy γ\gamma strength. This turns out to be remarkably robust with respect to the choice of interaction, truncation and model space, and we predict its presence in the whole isotopic chain, in particular the neutron-rich 72,74,76Ni^{72,74,76}\mathrm{Ni}.Comment: 9 pages, 9 figure

    Beta-delayed gamma decay of 26P: Possible evidence of a proton halo

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    Background: Measurements of β\beta decay provide important nuclear structure information that can be used to probe isospin asymmetries and inform nuclear astrophysics studies. Purpose: To measure the β\beta-delayed γ\gamma decay of 26^{26}P and compare the results with previous experimental results and shell-model calculations. Method: A 26^{26}P fast beam produced using nuclear fragmentation was implanted into a planar germanium detector. Its β\beta-delayed γ\gamma-ray emission was measured with an array of 16 high-purity germanium detectors. Positrons emitted in the decay were detected in coincidence to reduce the background. Results: The absolute intensities of 26^{26}P β\beta-delayed γ\gamma-rays were determined. A total of six new β\beta-decay branches and 15 new γ\gamma-ray lines have been observed for the first time in 26^{26}P β\beta-decay. A complete β\beta-decay scheme was built for the allowed transitions to bound excited states of 26^{26}Si. ftft values and Gamow-Teller strengths were also determined for these transitions and compared with shell model calculations and the mirror β\beta-decay of 26^{26}Na, revealing significant mirror asymmetries. Conclusions: A very good agreement with theoretical predictions based on the USDB shell model is observed. The significant mirror asymmetry observed for the transition to the first excited state (δ=51(10)%\delta=51(10)\%) may be evidence for a proton halo in 26^{26}P.Comment: 15 pages, 10 figures, 7 table

    Isobaric multiplet mass equation in the A=31A=31 T=3/2T = 3/2 quartets

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    The observed mass excesses of analog nuclear states with the same mass number AA and isospin TT can be used to test the isobaric multiplet mass equation (IMME), which has, in most cases, been validated to a high degree of precision. A recent measurement [Kankainen et al., Phys. Rev. C 93 041304(R) (2016)] of the ground-state mass of 31^{31}Cl led to a substantial breakdown of the IMME for the lowest A=31,T=3/2A = 31, T = 3/2 quartet. The second-lowest A=31,T=3/2A = 31, T = 3/2 quartet is not complete, due to uncertainties associated with the identity of the 31^{31}S member state. Using a fast 31^{31}Cl beam implanted into a plastic scintillator and a high-purity Ge γ\gamma-ray detection array, γ\gamma rays from the 31^{31}Cl(βγ)(\beta\gamma)31^{31}S sequence were measured. Shell-model calculations using USDB and the recently-developed USDE interactions were performed for comparison. Isospin mixing between the 31^{31}S isobaric analog state (IAS) at 6279.0(6) keV and a nearby state at 6390.2(7) keV was observed. The second T=3/2T = 3/2 state in 31^{31}S was observed at Ex=7050.0(8)E_x = 7050.0(8) keV. Isospin mixing in 31^{31}S does not by itself explain the IMME breakdown in the lowest quartet, but it likely points to similar isospin mixing in the mirror nucleus 31^{31}P, which would result in a perturbation of the 31^{31}P IAS energy. USDB and USDE calculations both predict candidate 31^{31}P states responsible for the mixing in the energy region slightly above Ex=6400E_x = 6400 keV. The second quartet has been completed thanks to the identification of the second 31^{31}S T=3/2T = 3/2 state, and the IMME is validated in this quartet

    Drosophila CLIP-190 and mammalian CLIP-170 display reduced microtubule plus end association in the nervous system

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    Axons act like cables, electrically wiring the nervous system. Polar bundles of microtubules (MTs) form their backbones and drive their growth. Plus end–tracking proteins (+TIPs) regulate MT growth dynamics and directionality at their plus ends. However, current knowledge about +TIP functions, mostly derived from work in vitro and in nonneuronal cells, may not necessarily apply to the very different context of axonal MTs. For example, the CLIP family of +TIPs are known MT polymerization promoters in nonneuronal cells. However, we show here that neither Drosophila CLIP-190 nor mammalian CLIP-170 is a prominent MT plus end tracker in neurons, which we propose is due to low plus end affinity of the CAP-Gly domain–containing N-terminus and intramolecular inhibition through the C-terminus. Instead, both CLIP-190 and CLIP-170 form F-actin–dependent patches in growth cones, mediated by binding of the coiled-coil domain to myosin-VI. Because our loss-of-function analyses in vivo and in culture failed to reveal axonal roles for CLIP-190, even in double-mutant combinations with four other +TIPs, we propose that CLIP-190 and -170 are not essential axon extension regulators. Our findings demonstrate that +TIP functions known from nonneuronal cells do not necessarily apply to the regulation of the very distinct MT networks in axons
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