Plasma measurements conducted in the vincinity of Venus on the spacecraft VENERA-4

Plasma flux measurements in vicinity of Venus by charged particle traps on Venera-4 spacecraf

Comparison of Certain Results of Simultaneous Measurements of Solar Wind Characteristics on Spacecrafts ''Venera-3'' and ''Pioneer-6''

Ion concentration, ion velocity, and other solar wind characteristics measured simultaneously aboard spacecraf

Signs of crossing by the moon of the earth's magnetosphere tail according to data of charged particle traps on the first artificial satellite of the moon /Luna-10/

Space probe charged particle data evidence for moon crossing of Earth magnetospheric tai

Estrogen Receptors in the Medial Amygdala Inhibit the Expression of Male Prosocial Behavior

Studies using estrogen receptor alpha(ER) knock-out mice indicate that ER alpha masculinizes male behavior. Recent studies of ER alpha and male prosocial behavior have shown an inverse relationship between ER alpha expression in regions of the brain that regulate social behavior, including the medial amygdala (MeA), and the expression of male prosocial behavior. These studies have lead to the hypothesis that low levels of ER alpha are necessary to permit the expression of high levels of male prosocial behavior. To test this, viral vectors were used to enhance ER alpha in male prairie voles (Microtus ochrogaster), which display high levels of prosocial behavior and low levels of MeA ER alpha. Adult male prairie voles were transfected with ER alpha in the MeA (MeA-ER alpha) or the caudate-putamen (ER alpha control) or luciferase (MeAsitespecific control), and 3 weeks later tested for spontaneous alloparental behavior and partner preference. Enhancing ER alpha in the MeA altered/reduced male prosocial behavior. Only one-third of MeA-ER alpha males, compared with all control males, were alloparental. Me-A-ER alpha males also displayed a significant preference for a novel female. This is a critical finding because the manipulations of neuropeptides, oxytocin and vasopressin, can inhibit the formation of a partner preference, but do not lead to the formation of a preference for a novel female. The results support the hypothesis that low levels of ER alpha are necessary for high levels of male prosocial behavior, and provide the first direct evidence that site-specific ER alpha expression plays a critical role in the expression of male prosocial behavior

Estrogen Receptor-Alpha in the Bed Nucleus of the Stria Terminalis Regulates Social Affiliation in Male Prairie Voles (Microtus Ochrogaster)

Estrogen receptor alpha (ER alpha) typically masculinizes male behavior, while low levels of ER alpha in the medial amygdala (MeA) and the bed nucleus of the stria terminalis (BST) are associated with high levels of male prosocial behavior. In the males of the highly social prairie vole (Microtus ochrogaster), increasing ER alpha in the MeA inhibited the expression of spontaneous alloparental behavior and produced a preference for novel females. To test for the effects of increased ER alpha in the BST, a viral vector was used to enhance ER alpha expression in the BST of adult male prairie voles. Following treatment, adult males were tested for alloparental behavior with 1-3-day- old pups, and for heterosexual social preference and affiliation. Treatment did not affect alloparental behavior as 73% of ER alpha-BST males and 62.5% of control males were alloparental. Increasing ER alpha in the BST affected heterosexual affiliation, with ER alpha-BST males spending significantly less total time in side-by-side contact with females relative to time spent with control males. ER alpha-BST males did not show a preference for either the familiar or novel female. These findings differed significantly from those reported in ER alpha-MeA enhanced males, where ER alpha inhibited alloparental behavior and produced a preference for a novel female. The findings from this study suggest two things: first, that increased ER alpha in the BST decreases social affiliation and second, that altering ER alpha in different regions of the social neural circuit differentially impacts the expression of social behavior

Gale–Nikaido–Debreu and Milgrom–Shannon: Communal interactions with endogenous community structures

© 2016 Elsevier Inc.This paper examines Nash jurisdictional stability in a model with a continuum of agents whose characteristics are distributed over a unidimensional interval. Communal benefits and costs of each individual depend on her identity and the composition of the community which she belongs to. Since the framework is too general to yield an existence of Nash equilibrium, we introduce the essentiality of membership in one of the communities for all individuals. We highlight the Border Indifference Property (BIP), when all individuals located on a border between two adjacent jurisdictions are indifferent about joining either of them and show that BIP is a necessary condition for yielding a Nash equilibrium. We invoke the celebrated Gale–Nikaido–Debreu Lemma to guarantee the existence of a partition that satisfies BIP. We then proceed to demonstrate that BIP is not sufficient to yield a Nash equilibrium. The equilibrium existence under BIP is rescued when we use the Milgrom–Shannon monotone comparative statics conditions

Non-local anomaly of the axial-vector current for bound states

We demonstrate that the amplitude $<\rho\gamma|\partial_\nu (\bar q\gamma_\nu \gamma_5 q)|0>$ does not vanish in the limit of zero quark masses. This represents a new kind of violation of the classical equation of motion for the axial current and should be interpreted as the axial anomaly for bound states. The anomaly emerges in spite of the fact that the one loop integrals are ultraviolet-finite as guaranteed by the presence of the bound-state wave function. As a result, the amplitude behaves like $\sim 1/p^2$ in the limit of a large momentum $p$ of the current. This is to be compared with the amplitude $$ which remains finite in the limit $p^2\to\infty$. The observed effect leads to the modification of the classical equation of motion of the axial-vector current in terms of the non-local operator and can be formulated as a non-local axial anomaly for bound states.Comment: revtex, 4 pages, numerical value for $\kappa$ in Eq. (19) is corrected, Eqs. (22) and (23) are modified. New references added. Results remain unchange

Effects of Prepubertal or Adult Site-Specific Knockdown of Estrogen Receptor β in the Medial Preoptic Area and Medial Amygdala on Social Behaviors in Male Mice

Testosterone, after being converted to estradiol in the brain, acts on estrogen receptors (ERα and ERβ) and controls the expression of male-type social behavior. Previous studies in male mice have revealed that ERα expressed in the medial preoptic area (MPOA) and medial amygdala (MeA) are differently involved in the regulation of sexual and aggressive behaviors by testosterone action at the time of testing in adult and/or on brain masculinization process during pubertal period. However, a role played by ERβ in these brain regions still remains unclear. Here we examined the effects of site-specific knockdown of ERβ (βERKD) in the MPOA and MeA on male social behaviors with the use of adeno-associated viral mediated RNA interference methods in ICR/Jcl mice. Prepubertal βERKD in the MPOA revealed that continuous suppression of ERβ gene expression throughout the pubertal period and adulthood decreased aggressive but not sexual behavior tested as adults. Because βERKD in the MPOA only in adulthood did not affect either sexual or aggressive behaviors, it was concluded that pubertal ERβ in the MPOA might have an essential role for the full expression of aggressive behavior in adulthood. On the other hand, although neither prepubertal nor adult βERKD in the MeA had any effects on sexual and aggressive behavior, βERKD in adulthood disrupted sexual preference of receptive females over nonreceptive females. Collectively, these results suggest that ERβ in the MPOA and MeA are involved in the regulation of male sexual and aggressive behavior in a manner substantially different from that of ERα

Estrogen Receptor-α in the Bed Nucleus of the Stria Terminalis Regulates Social Affiliation in Male Prairie Voles (Microtus ochrogaster)

Estrogen receptor alpha (ERα) typically masculinizes male behavior, while low levels of ERα in the medial amygdala (MeA) and the bed nucleus of the stria terminalis (BST) are associated with high levels of male prosocial behavior. In the males of the highly social prairie vole (Microtus ochrogaster), increasing ERα in the MeA inhibited the expression of spontaneous alloparental behavior and produced a preference for novel females. To test for the effects of increased ERα in the BST, a viral vector was used to enhance ERα expression in the BST of adult male prairie voles. Following treatment, adult males were tested for alloparental behavior with 1–3-day-old pups, and for heterosexual social preference and affiliation. Treatment did not affect alloparental behavior as 73% of ERα-BST males and 62.5% of control males were alloparental. Increasing ERα in the BST affected heterosexual affiliation, with ERα-BST males spending significantly less total time in side-by-side contact with females relative to time spent with control males. ERα-BST males did not show a preference for either the familiar or novel female. These findings differed significantly from those reported in ERα-MeA enhanced males, where ERα inhibited alloparental behavior and produced a preference for a novel female. The findings from this study suggest two things: first, that increased ERα in the BST decreases social affiliation and second, that altering ERα in different regions of the social neural circuit differentially impacts the expression of social behavior