42,175 research outputs found
Recent Solar Measurements Results at the Parabolic Dish Test Site
After the Mexican volcanic eruptions of March 28, April 3 and 4, 1982, the question of its effect on insolation levels at the Parabolic Dish Test Site (PDTS) naturally arose. Clearly, the answer to the original question is that the Mexican volcanic explosion had a significant impact on energy and insolation levels at the PDTS and, furthermore, it has been quite long lasting. The first really significant decrease in energy and insolation levels occurred in June 1982 when the energy level decreased by 19.7% while the peak insolation levels went down by 4.0%. June of 1982 was also the first month (of 13 consecutive months) when peak insolation levels did not equal or exceed 1,000 W/sq m. Signs of a recovery from the effects of the volcanic explosion began to appear in May of 1983, when the energy level exceeded that of May 1981 as well as May 1982. It would appear that energy and insolation levels are improving at the PDTS, but have not quite reached normal or pre-volcanic levels. At this time the data would seem to suggest a return to normal energy and insolation levels will occur in the very near future
Mark 4-A DSCC (Magellan-era) telemetry system description
An update to the description of the Deep Space Communications Complex portion of the Mark 4-A Telemetry system is given. This system is currently implemented at all signal processing centers. The upgrade of the telemetry system was undertaken primarily in support of the Voyager-Neptune Encounter and the Magellan mission. The Mark 3 Telemetry System is the predecessor of the Mark 4-A system
Interacting Turing-Hopf Instabilities Drive Symmetry-Breaking Transitions in a Mean-Field Model of the Cortex: A Mechanism for the Slow Oscillation
Electrical recordings of brain activity during the transition from wake to anesthetic coma show temporal and spectral alterations that are correlated with gross changes in the underlying brain state. Entry into anesthetic unconsciousness is signposted by the emergence of large, slow oscillations of electrical activity (≲1 Hz) similar to the slow waves observed in natural sleep. Here we present a two-dimensional mean-field model of the cortex in which slow spatiotemporal oscillations arise spontaneously through a Turing (spatial) symmetry-breaking bifurcation that is modulated by a Hopf (temporal) instability. In our model, populations of neurons are densely interlinked by chemical synapses, and by interneuronal gap junctions represented as an inhibitory diffusive coupling. To demonstrate cortical behavior over a wide range of distinct brain states, we explore model dynamics in the vicinity of a general-anesthetic-induced transition from “wake” to “coma.” In this region, the system is poised at a codimension-2 point where competing Turing and Hopf instabilities coexist. We model anesthesia as a moderate reduction in inhibitory diffusion, paired with an increase in inhibitory postsynaptic response, producing a coma state that is characterized by emergent low-frequency oscillations whose dynamics is chaotic in time and space. The effect of long-range axonal white-matter connectivity is probed with the inclusion of a single idealized point-to-point connection. We find that the additional excitation from the long-range connection can provoke seizurelike bursts of cortical activity when inhibitory diffusion is weak, but has little impact on an active cortex. Our proposed dynamic mechanism for the origin of anesthetic slow waves complements—and contrasts with—conventional explanations that require cyclic modulation of ion-channel conductances. We postulate that a similar bifurcation mechanism might underpin the slow waves of natural sleep and comment on the possible consequences of chaotic dynamics for memory processing and learning
Modelling general anaesthesia as a first-order phase transition in the cortex
Since 1997 we have been developing a theoretical foundation for general anaesthesia. We have been able to demonstrate that the abrupt change in brain state broughton by anaesthetic drugs can be characterized as a first-order phase transition in the population-average membrane voltage of the cortical neurons. The theory predicts that, as the critical point of phase-change into unconsciousness is approached, the electrical fluctuations in cortical activity will grow strongly in amplitude while slowing in frequency, becoming more correlated both in time and in space. Thus the bio-electrical change of brain-state has deep similarities with thermodynamic phase changes of classical physics. The theory further predicts the existence of a second critical point, hysteretically separated from the first, corresponding to the return path from comatose unconsciousness back to normal responsiveness. There is a steadily accumulating body of clinical evidence in support of all of the phasetransition predictions: low-frequency power surge in EEG activity; increased correlation time and correlation length in EEG fluctuations; hysteresis separation, with respect to drug concentration, between the point of induction and the point of emergence
Optimal payload rate limit algorithm for zero-G manipulators
An algorithm for continuously computing safe maximum relative velocities for two bodies joined by a manipulator is discussed. The maximum velocities are such that if the brakes are applied at that instant, the ensuing travel between the bodies will be less than or equal to a predetermined amount. An improvement in the way this limit is computed for space manipulators is shown. The new method is explained, test cases are posed, and the results of these tests are displayed and discussed
The Behaviour of the Green Function for the BFKL Pomeron with Running Coupling
We analyse here in LO the physical properties of the Green function solution
for the BFKL equation. We show that the solution obeys the orthonormality
conditions in the physical region and fulfills the completeness requirements.
The unintegrated gluon density is shown to consists of a set of few poles with
parameters which could be determined by comparison with the DIS data of high
precision
Indirect Evidence for New Physics at the 10 TeV Scale
We show that the supersymmetric extension of the Standard Model modifies the
structure of the low lying BFKL discrete pomeron states (DPS) which give a
sizable contribution to the gluon structure function in the HERA x and Q2
region. The comparison of the gluon density, determined within DPS with N=1
SUSY, with data favours a supersymmetry scale of the order of 10 TeV. The DPS
method described here could open a new window to the physics beyond the
Standard Model.Comment: 14 pages, 6 figure
Phase transitions in single neurons and neural populations: Critical slowing, anesthesia, and sleep cycles
The firing of an action potential by a biological neuron represents a dramatic transition from small-scale linear stochastics (subthreshold voltage fluctuations) to gross-scale nonlinear dynamics (birth of a 1-ms voltage spike). In populations of neurons we see similar, but slower, switch-like there-and-back transitions between low-firing background states and high-firing activated states. These state transitions are controlled by varying levels of input current (single neuron), varying amounts of GABAergic drug (anesthesia), or varying concentrations of neuromodulators and neurotransmitters (natural sleep), and all occur within a milieu of unrelenting biological noise. By tracking the altering responsiveness of the excitable membrane to noisy stimulus, we can infer how close the neuronal system (single unit or entire population) is to switching threshold. We can quantify this “nearness to switching” in terms of the altering eigenvalue structure: the dominant eigenvalue approaches zero, leading to a growth in correlated, low-frequency power, with exaggerated responsiveness to small perturbations, the responses becoming larger and slower as the neural population approaches its critical point–-this is critical slowing. In this chapter we discuss phase-transition predictions for both single-neuron and neural-population models, comparing theory with laboratory and clinical measurement
Neonatal weight loss in breast and formula-fed infants
We have observed an increase in the number of breast fed babies presenting with dehydration and/or failure to thrive because of lactation failure and non-recognition of feeding problems. Recent reports1,2 support this experience and recommend monitoring of the weight of infants through the neonatal period. However, these reports acknowledge uncertainty as to what actually constitutes normal neonatal weight loss. Maisels and colleagues published two studies which have been quoted as giving guidance on normal loss. Both studies were designed primarily to study factors that influence breast milk jaundice. The first3 reported a mean weight loss of about 6% in 100 unselected well babies during the first 3 days. The subsequent study4 reported a mean weight loss of 6.86% in 186 infants. The timescale over which babies were weighed was not clearly indicated, although it may have only been 2-3 days. The sample was neither population based nor randomly selected, being largely preselected because of the presence of more pronounced jaundice. The distribution of data points for early neonatal weight loss are likely to be skewed, yet both studies reported the results as mean (SD). Owing to the design and method of data presentation, these studies cannot reliably inform the debate as to what constitutes the norm. Marchini and colleagues published reports also designed primarily to study other issues. One5 indicated a mean early weight loss of 5.7%. Measurements were recorded over a three day period, and no indication is given of the skewness of the data. Another study6 reported a median weight loss of about 6% recorded over a four day period. At least one baby lost > 15% of his/her birth weight during this time, but there is no clear information as to the frequency with which more extreme degrees of weight loss are observed
- …